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{"target":"https://pubannotation.org/docs/sourcedb/PMC/sourceid/539061","sourcedb":"PMC","sourceid":"539061","source_url":"http://www.ncbi.nlm.nih.gov/pmc/539061","text":"Our results are particularly interesting in light of the recent implication that GSK-3β controls Snail's stability and subcellular localization [50]. Since Wnts are known to deactivate GSK-3β, Wnt and TGF-β2 signaling may contribute to Snail's transient induction and accumulation. Moreover, since inhibition of GSK-3β results in Snail upregulation and E-cadherin downregulation, Snail and GSK-3β may function at a crossroads in controlling hair bud development.","tracks":[]}