PMC:3585731 / 48348-48722 JSONTXT

Annnotations TAB JSON ListView MergeView

    testone

    {"project":"testone","denotations":[{"id":"T20802","span":{"begin":344,"end":353},"obj":"Gene_expression"},{"id":"T20801","span":{"begin":325,"end":335},"obj":"Positive_regulation"},{"id":"T20755","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T20754","span":{"begin":339,"end":343},"obj":"Protein"}],"relations":[{"id":"R13922","pred":"themeOf","subj":"T20754","obj":"T20802"},{"id":"R13933","pred":"themeOf","subj":"T20802","obj":"T20801"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    pmc-enju-pas

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results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    bionlp-st-ge-2016-test-proteins

    {"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T21082","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T21081","span":{"begin":339,"end":343},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    bionlp-st-ge-2016-uniprot

    {"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T22944","span":{"begin":339,"end":343},"obj":"http://www.uniprot.org/uniprot/P01375"},{"id":"T22930","span":{"begin":357,"end":361},"obj":"http://www.uniprot.org/uniprot/Q13546"},{"id":"T22917","span":{"begin":362,"end":365},"obj":"http://www.uniprot.org/uniprot/Q9Y243"},{"id":"T22883","span":{"begin":362,"end":365},"obj":"http://www.uniprot.org/uniprot/P31751"},{"id":"T22849","span":{"begin":362,"end":365},"obj":"http://www.uniprot.org/uniprot/P31749"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    GO-BP

    {"project":"GO-BP","denotations":[{"id":"T21259","span":{"begin":119,"end":127},"obj":"http://purl.obolibrary.org/obo/GO_0007349"},{"id":"T21256","span":{"begin":47,"end":59},"obj":"http://purl.obolibrary.org/obo/GO_0006954"},{"id":"T21253","span":{"begin":212,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0097300"},{"id":"T21239","span":{"begin":212,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0010939"},{"id":"T21226","span":{"begin":212,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0070265"},{"id":"T21193","span":{"begin":226,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0016265"},{"id":"T21176","span":{"begin":221,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0008219"},{"id":"T21166","span":{"begin":344,"end":353},"obj":"http://purl.obolibrary.org/obo/GO_0009058"},{"id":"T21153","span":{"begin":293,"end":304},"obj":"http://purl.obolibrary.org/obo/GO_0097528"},{"id":"T21152","span":{"begin":91,"end":102},"obj":"http://purl.obolibrary.org/obo/GO_0097528"},{"id":"T21134","span":{"begin":212,"end":231},"obj":"http://purl.obolibrary.org/obo/GO_0070266"},{"id":"T21129","span":{"begin":293,"end":304},"obj":"http://purl.obolibrary.org/obo/GO_0070266"},{"id":"T21128","span":{"begin":91,"end":102},"obj":"http://purl.obolibrary.org/obo/GO_0070266"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    GO-CC

    {"project":"GO-CC","denotations":[{"id":"T21292","span":{"begin":174,"end":182},"obj":"http://purl.obolibrary.org/obo/GO_0016020"},{"id":"T21289","span":{"begin":167,"end":182},"obj":"http://purl.obolibrary.org/obo/GO_0005886"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    sentences

    {"project":"sentences","denotations":[{"id":"T20976","span":{"begin":0,"end":374},"obj":"Sentence"},{"id":"T301","span":{"begin":0,"end":374},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    simple1

    {"project":"simple1","denotations":[{"id":"T21334","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T21333","span":{"begin":339,"end":343},"obj":"Protein"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    BioNLP16_DUT

    {"project":"BioNLP16_DUT","denotations":[{"id":"T22809","span":{"begin":344,"end":353},"obj":"Gene_expression"},{"id":"T22808","span":{"begin":325,"end":335},"obj":"Positive_regulation"},{"id":"T22781","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T22780","span":{"begin":339,"end":343},"obj":"Protein"}],"relations":[{"id":"R15392","pred":"themeOf","subj":"T22780","obj":"T22809"},{"id":"R15404","pred":"themeOf","subj":"T22809","obj":"T22808"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    BioNLP16_Messiy

    {"project":"BioNLP16_Messiy","denotations":[{"id":"T23112","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T23111","span":{"begin":339,"end":343},"obj":"Protein"},{"id":"T23140","span":{"begin":325,"end":335},"obj":"Positive_regulation"},{"id":"T23139","span":{"begin":344,"end":353},"obj":"Gene_expression"}],"relations":[{"id":"R15469","pred":"themeOf","subj":"T23111","obj":"T23139"},{"id":"R15479","pred":"themeOf","subj":"T23139","obj":"T23140"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    DLUT931

    {"project":"DLUT931","denotations":[{"id":"T23019","span":{"begin":325,"end":335},"obj":"Positive_regulation"},{"id":"T23018","span":{"begin":344,"end":353},"obj":"Gene_expression"},{"id":"T22982","span":{"begin":357,"end":361},"obj":"Protein"},{"id":"T22981","span":{"begin":339,"end":343},"obj":"Protein"}],"relations":[{"id":"R15441","pred":"themeOf","subj":"T23018","obj":"T23019"},{"id":"R15426","pred":"themeOf","subj":"T22981","obj":"T23018"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    bionlp-st-ge-2016-test-ihmc

    {"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T23574","span":{"begin":354,"end":373},"obj":"Protein"},{"id":"T23570","span":{"begin":174,"end":182},"obj":"Entity"},{"id":"T23554","span":{"begin":336,"end":353},"obj":"Protein"},{"id":"T23484","span":{"begin":357,"end":365},"obj":"Protein"},{"id":"T23622","span":{"begin":357,"end":365},"obj":"Protein"},{"id":"T23610","span":{"begin":164,"end":231},"obj":"Entity"},{"id":"T23607","span":{"begin":362,"end":365},"obj":"Protein"}],"text":"Our results highlight an important notion that inflammation not only passively accompanies necroptosis in a variety of cellular systems by the virtue of rapid loss of plasma membrane integrity characteristic for necrotic cell death, but also that it is an intrinsic and regulated component of necroptosis due to the specific activation of TNFα synthesis by RIP1/Akt kinases."}

    bionlp-st-ge-2016-spacy-parsed

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