PMC:3320587 / 779-994
Annnotations
pmc-enju-pas
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bionlp-st-ge-2016-uniprot
{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T414","span":{"begin":29,"end":34},"obj":"http://www.uniprot.org/uniprot/O94916"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":") ligand, and that an intact NFAT5 binding site in the viral promoter of R5-tropic HIV-1 subtype B and subtype C molecular clones is required for efficent induction of HIV-1 replication by MTb. Furthermore, silencin"}
GO-MF
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BioNLP16_Messiy
{"project":"BioNLP16_Messiy","denotations":[{"id":"T456","span":{"begin":133,"end":141},"obj":"Positive_regulation"},{"id":"T457","span":{"begin":155,"end":164},"obj":"Positive_regulation"}],"relations":[{"id":"R519","pred":"themeOf","subj":"T457","obj":"T456"}],"text":") ligand, and that an intact NFAT5 binding site in the viral promoter of R5-tropic HIV-1 subtype B and subtype C molecular clones is required for efficent induction of HIV-1 replication by MTb. Furthermore, silencin"}
bionlp-st-ge-2016-test-ihmc
{"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T823","span":{"begin":19,"end":47},"obj":"Binding"},{"id":"T782","span":{"begin":51,"end":96},"obj":"Entity"},{"id":"T787","span":{"begin":186,"end":192},"obj":"Protein"},{"id":"T788","span":{"begin":29,"end":34},"obj":"Protein"},{"id":"T790","span":{"begin":19,"end":47},"obj":"Entity"}],"relations":[{"id":"R600","pred":"themeOf","subj":"T790","obj":"T823"}],"text":") ligand, and that an intact NFAT5 binding site in the viral promoter of R5-tropic HIV-1 subtype B and subtype C molecular clones is required for efficent induction of HIV-1 replication by MTb. Furthermore, silencin"}
bionlp-st-ge-2016-spacy-parsed
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bionlp-st-ge-2016-test-tees
{"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T132","span":{"begin":29,"end":47},"obj":"Protein"}],"text":") ligand, and that an intact NFAT5 binding site in the viral promoter of R5-tropic HIV-1 subtype B and subtype C molecular clones is required for efficent induction of HIV-1 replication by MTb. Furthermore, silencin"}