PMC:3320587 / 2501-3574 JSON TXT

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2_test

{"project":"2_test","denotations":[{"id":"22496647-17359235-98010766","span":{"begin":399,"end":400},"obj":"17359235"},{"id":"22496647-1906500-98010767","span":{"begin":649,"end":650},"obj":"1906500"},{"id":"22496647-11854362-98010768","span":{"begin":653,"end":654},"obj":"11854362"},{"id":"22496647-17263638-98010769","span":{"begin":825,"end":826},"obj":"17263638"},{"id":"22496647-21852682-98010770","span":{"begin":846,"end":848},"obj":"21852682"},{"id":"22496647-1345800-98010771","span":{"begin":1045,"end":1047},"obj":"1345800"},{"id":"22496647-20299961-98010772","span":{"begin":1050,"end":1052},"obj":"20299961"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

pmc-enju-pas

bionlp-st-ge-2016-test-proteins

{"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T980","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T979","span":{"begin":552,"end":555},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

bionlp-st-ge-2016-uniprot

{"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T1773","span":{"begin":783,"end":786},"obj":"http://www.uniprot.org/uniprot/P01730"},{"id":"T1772","span":{"begin":552,"end":555},"obj":"http://www.uniprot.org/uniprot/P01730"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

GO-BP

{"project":"GO-BP","denotations":[{"id":"T1012","span":{"begin":511,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_1904515"},{"id":"T1011","span":{"begin":477,"end":493},"obj":"http://purl.obolibrary.org/obo/GO_0075136"},{"id":"T1007","span":{"begin":349,"end":362},"obj":"http://purl.obolibrary.org/obo/GO_0006351"},{"id":"T1006","span":{"begin":520,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_0007165"},{"id":"T1005","span":{"begin":309,"end":327},"obj":"http://purl.obolibrary.org/obo/GO_0007165"},{"id":"T1003","span":{"begin":309,"end":318},"obj":"http://purl.obolibrary.org/obo/GO_0023052"},{"id":"T1002","span":{"begin":511,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_1903572"},{"id":"T1001","span":{"begin":299,"end":327},"obj":"http://purl.obolibrary.org/obo/GO_1903572"},{"id":"T1000","span":{"begin":511,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_1902533"},{"id":"T999","span":{"begin":299,"end":327},"obj":"http://purl.obolibrary.org/obo/GO_1902533"},{"id":"T998","span":{"begin":121,"end":126},"obj":"http://purl.obolibrary.org/obo/GO_0016265"},{"id":"T1016","span":{"begin":511,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_0038007"},{"id":"T1015","span":{"begin":511,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_1902849"},{"id":"T1014","span":{"begin":511,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_0009967"},{"id":"T1013","span":{"begin":511,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_1904263"},{"id":"T1021","span":{"begin":527,"end":539},"obj":"http://purl.obolibrary.org/obo/GO_0009293"},{"id":"T1020","span":{"begin":520,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_0035556"},{"id":"T1019","span":{"begin":520,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_0036499"},{"id":"T1018","span":{"begin":520,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_0036500"},{"id":"T1017","span":{"begin":520,"end":548},"obj":"http://purl.obolibrary.org/obo/GO_0036498"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

GO-CC

{"project":"GO-CC","denotations":[{"id":"T1049","span":{"begin":578,"end":583},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T1048","span":{"begin":558,"end":563},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T1047","span":{"begin":477,"end":481},"obj":"http://purl.obolibrary.org/obo/GO_0018995"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

sentences

{"project":"sentences","denotations":[{"id":"T929","span":{"begin":852,"end":1072},"obj":"Sentence"},{"id":"T928","span":{"begin":657,"end":851},"obj":"Sentence"},{"id":"T927","span":{"begin":404,"end":656},"obj":"Sentence"},{"id":"T926","span":{"begin":207,"end":403},"obj":"Sentence"},{"id":"T925","span":{"begin":0,"end":206},"obj":"Sentence"},{"id":"T17","span":{"begin":0,"end":206},"obj":"Sentence"},{"id":"T18","span":{"begin":207,"end":403},"obj":"Sentence"},{"id":"T19","span":{"begin":404,"end":656},"obj":"Sentence"},{"id":"T20","span":{"begin":657,"end":851},"obj":"Sentence"},{"id":"T21","span":{"begin":852,"end":1072},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

ICD10

{"project":"ICD10","denotations":[{"id":"T1035","span":{"begin":14,"end":26},"obj":"http://purl.bioontology.org/ontology/ICD10/A15-A19.9"},{"id":"T1037","span":{"begin":159,"end":175},"obj":"http://purl.bioontology.org/ontology/ICD10/D84.9"},{"id":"T1036","span":{"begin":57,"end":69},"obj":"http://purl.bioontology.org/ontology/ICD10/A15-A19.9"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

simple1

{"project":"simple1","denotations":[{"id":"T1074","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T1073","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

BioNLP16_DUT

{"project":"BioNLP16_DUT","denotations":[{"id":"T2668","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T2667","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

BioNLP16_Messiy

{"project":"BioNLP16_Messiy","denotations":[{"id":"T1847","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T1846","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

DLUT931

{"project":"DLUT931","denotations":[{"id":"T1796","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T1795","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

bionlp-st-ge-2016-test-ihmc

{"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T2786","span":{"begin":497,"end":510},"obj":"Regulation"},{"id":"T2781","span":{"begin":955,"end":982},"obj":"Positive_regulation"},{"id":"T2768","span":{"begin":260,"end":264},"obj":"Protein"},{"id":"T2765","span":{"begin":430,"end":439},"obj":"Protein"},{"id":"T2762","span":{"begin":237,"end":265},"obj":"Protein"},{"id":"T2761","span":{"begin":965,"end":968},"obj":"Protein"},{"id":"T2759","span":{"begin":497,"end":500},"obj":"Protein"},{"id":"T2758","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T2754","span":{"begin":552,"end":555},"obj":"Protein"},{"id":"T2748","span":{"begin":552,"end":563},"obj":"Entity"},{"id":"T2746","span":{"begin":276,"end":298},"obj":"Entity"},{"id":"T2727","span":{"begin":269,"end":272},"obj":"Protein"},{"id":"T2718","span":{"begin":783,"end":793},"obj":"Entity"},{"id":"T2716","span":{"begin":578,"end":583},"obj":"Entity"},{"id":"T2713","span":{"begin":28,"end":31},"obj":"Protein"},{"id":"T2709","span":{"begin":153,"end":196},"obj":"Protein"},{"id":"T2705","span":{"begin":444,"end":463},"obj":"Protein"},{"id":"T2698","span":{"begin":699,"end":710},"obj":"Protein"}],"relations":[{"id":"R2152","pred":"themeOf","subj":"T2759","obj":"T2786"},{"id":"R2154","pred":"themeOf","subj":"T2761","obj":"T2781"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

bionlp-st-ge-2016-spacy-parsed

bionlp-st-ge-2016-test-tees

{"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T1882","span":{"begin":783,"end":788},"obj":"Protein"},{"id":"T1881","span":{"begin":552,"end":557},"obj":"Protein"},{"id":"T1880","span":{"begin":214,"end":224},"obj":"Binding"},{"id":"T1879","span":{"begin":214,"end":224},"obj":"Binding"},{"id":"T1878","span":{"begin":260,"end":264},"obj":"Protein"},{"id":"T1877","span":{"begin":228,"end":258},"obj":"Protein"}],"relations":[{"id":"R1391","pred":"themeOf","subj":"T1877","obj":"T1879"},{"id":"R1392","pred":"themeOf","subj":"T1878","obj":"T1880"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

testone

{"project":"testone","denotations":[{"id":"T855","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T854","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

test3

{"project":"test3","denotations":[{"id":"T900","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T899","span":{"begin":552,"end":555},"obj":"Protein"},{"id":"T881","span":{"begin":783,"end":786},"obj":"Protein"},{"id":"T880","span":{"begin":552,"end":555},"obj":"Protein"}],"text":"Mycobacterium tuberculosis (MTb), the causative agent of tuberculosis (TB), is the most common co-infection and cause of death in patients infected with human immunodeficiency virus type 1 (HIV-1) [1], [2]. Direct engagement of pathogen recognition receptors (PRRs) by MTb on mononuclear phagocytes activates signaling cascades that directly induce transcription from the proviral LTR (reviewed in [3]). Furthermore, inflammatory cytokines and chemokines produced by the human host in response to MTb infection activate signal transduction pathways in CD4 T cells and monocytic cells that also result in transcriptional activation of the HIV-1 LTR [4]–[6]. Activation of HIV-1 replication via these MTb-induced pathways ultimately leads to higher viral loads and, in turn, expedited CD4 T cell loss and progression to AIDS ([7], reviewed in [8]–[10]). Furthermore, the progressive immune compromise associated with HIV-1 infection itself is a major cause of latent MTb reactivation, as well as increased susceptibility to primary TB infection ([11]–[15], reviewed in [8]).\n"}

PMC:3320587 / 2501-3574 JSONTXT

Annnotations TAB JSON ListView MergeView

PMC:3320587 / 2501-3574 JSON TXT