PMC:3317373 / 0-356
Annnotations
pmc-enju-pas
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Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}
UBERON-AE
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GO-BP
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sentences
{"project":"sentences","denotations":[{"id":"T24","span":{"begin":0,"end":119},"obj":"Sentence"},{"id":"T25","span":{"begin":130,"end":341},"obj":"Sentence"},{"id":"T1","span":{"begin":0,"end":119},"obj":"Sentence"},{"id":"T2","span":{"begin":121,"end":129},"obj":"Sentence"},{"id":"T3","span":{"begin":130,"end":341},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"The Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}
ICD10
{"project":"ICD10","denotations":[{"id":"T255","span":{"begin":76,"end":82},"obj":"http://purl.bioontology.org/ontology/ICD10/T14.9"}],"text":"The Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}
bionlp-st-ge-2016-test-ihmc
{"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T593","span":{"begin":207,"end":250},"obj":"Positive_regulation"},{"id":"T591","span":{"begin":311,"end":340},"obj":"Protein"},{"id":"T590","span":{"begin":320,"end":330},"obj":"Entity"},{"id":"T567","span":{"begin":12,"end":19},"obj":"Protein"},{"id":"T574","span":{"begin":86,"end":119},"obj":"Entity"},{"id":"T579","span":{"begin":302,"end":306},"obj":"Protein"},{"id":"T580","span":{"begin":218,"end":226},"obj":"Protein"}],"relations":[{"id":"R449","pred":"themeOf","subj":"T580","obj":"T593"}],"text":"The Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}
bionlp-st-ge-2016-spacy-parsed
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Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}
bionlp-st-ge-2016-test-tees
{"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T287","span":{"begin":15,"end":34},"obj":"Protein"},{"id":"T293","span":{"begin":169,"end":177},"obj":"Positive_regulation"},{"id":"T292","span":{"begin":207,"end":217},"obj":"Positive_regulation"},{"id":"T291","span":{"begin":311,"end":316},"obj":"Protein"},{"id":"T290","span":{"begin":302,"end":306},"obj":"Protein"},{"id":"T289","span":{"begin":221,"end":226},"obj":"Protein"},{"id":"T288","span":{"begin":154,"end":158},"obj":"Protein"}],"relations":[{"id":"R207","pred":"themeOf","subj":"T289","obj":"T292"},{"id":"R208","pred":"themeOf","subj":"T292","obj":"T293"}],"text":"The Absence of Nrf2 Enhances NF-κB-Dependent Inflammation following Scratch Injury in Mouse Primary Cultured Astrocytes\n\nAbstract\nIt has been proved that Nrf2 depletion enhances inflammatory process through activation of NF-κB in the brain after TBI, but little is known about the relationship between Nrf2 and NF-κB in astrocytes after TBI. Hence, we used"}