PMC:1359074 / 21553-21803 JSONTXT

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    pmc-enju-pas

    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data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    bionlp-st-ge-2016-test-proteins

    {"project":"bionlp-st-ge-2016-test-proteins","denotations":[{"id":"T9085","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9084","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9083","span":{"begin":58,"end":60},"obj":"Protein"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    bionlp-st-ge-2016-uniprot

    {"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T9326","span":{"begin":235,"end":239},"obj":"http://www.uniprot.org/uniprot/P35712"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    UBERON-AE

    {"project":"UBERON-AE","denotations":[{"id":"T9023","span":{"begin":150,"end":155},"obj":"http://purl.obolibrary.org/obo/UBERON_0002107"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    GO-CC

    {"project":"GO-CC","denotations":[{"id":"T9093","span":{"begin":119,"end":124},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    sentences

    {"project":"sentences","denotations":[{"id":"T9030","span":{"begin":0,"end":250},"obj":"Sentence"},{"id":"T157","span":{"begin":0,"end":250},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    simple1

    {"project":"simple1","denotations":[{"id":"T9128","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9127","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9126","span":{"begin":58,"end":60},"obj":"Protein"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    BioNLP16_DUT

    {"project":"BioNLP16_DUT","denotations":[{"id":"T9712","span":{"begin":195,"end":201},"obj":"Negative_regulation"},{"id":"T9711","span":{"begin":80,"end":90},"obj":"Gene_expression"},{"id":"T9710","span":{"begin":240,"end":244},"obj":"Negative_regulation"},{"id":"T9699","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9698","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9697","span":{"begin":58,"end":60},"obj":"Protein"}],"relations":[{"id":"R6755","pred":"themeOf","subj":"T9697","obj":"T9711"},{"id":"R6756","pred":"themeOf","subj":"T9698","obj":"T9712"},{"id":"R6757","pred":"themeOf","subj":"T9699","obj":"T9710"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    BioNLP16_Messiy

    {"project":"BioNLP16_Messiy","denotations":[{"id":"T9352","span":{"begin":80,"end":90},"obj":"Gene_expression"},{"id":"T9351","span":{"begin":195,"end":201},"obj":"Negative_regulation"},{"id":"T9350","span":{"begin":240,"end":244},"obj":"Negative_regulation"},{"id":"T9340","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9339","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9338","span":{"begin":58,"end":60},"obj":"Protein"}],"relations":[{"id":"R6481","pred":"themeOf","subj":"T9338","obj":"T9352"},{"id":"R6482","pred":"themeOf","subj":"T9339","obj":"T9351"},{"id":"R6483","pred":"themeOf","subj":"T9340","obj":"T9350"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    DLUT931

    {"project":"DLUT931","denotations":[{"id":"T9401","span":{"begin":65,"end":68},"obj":"Positive_regulation"},{"id":"T9400","span":{"begin":240,"end":244},"obj":"Negative_regulation"},{"id":"T9399","span":{"begin":212,"end":221},"obj":"Negative_regulation"},{"id":"T9398","span":{"begin":195,"end":201},"obj":"Negative_regulation"},{"id":"T9397","span":{"begin":80,"end":90},"obj":"Gene_expression"},{"id":"T9380","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9379","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9378","span":{"begin":58,"end":60},"obj":"Protein"}],"relations":[{"id":"R6498","pred":"themeOf","subj":"T9378","obj":"T9397"},{"id":"R6499","pred":"themeOf","subj":"T9379","obj":"T9398"},{"id":"R6500","pred":"themeOf","subj":"T9379","obj":"T9399"},{"id":"R6501","pred":"themeOf","subj":"T9380","obj":"T9400"},{"id":"R6509","pred":"themeOf","subj":"T9397","obj":"T9401"},{"id":"R6510","pred":"themeOf","subj":"T9399","obj":"T9398"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    bionlp-st-ge-2016-test-ihmc

    {"project":"bionlp-st-ge-2016-test-ihmc","denotations":[{"id":"T9671","span":{"begin":58,"end":60},"obj":"Protein"},{"id":"T9668","span":{"begin":235,"end":249},"obj":"Protein"},{"id":"T9659","span":{"begin":94,"end":155},"obj":"Entity"},{"id":"T9651","span":{"begin":209,"end":211},"obj":"Protein"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    bionlp-st-ge-2016-spacy-parsed

    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"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    bionlp-st-ge-2016-test-tees

    {"project":"bionlp-st-ge-2016-test-tees","denotations":[{"id":"T9114","span":{"begin":235,"end":239},"obj":"Protein"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    testone

    {"project":"testone","denotations":[{"id":"T8982","span":{"begin":195,"end":201},"obj":"Negative_regulation"},{"id":"T8974","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T8973","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T8972","span":{"begin":58,"end":60},"obj":"Protein"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}

    test3

    {"project":"test3","denotations":[{"id":"T9019","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T9018","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T9017","span":{"begin":58,"end":60},"obj":"Protein"},{"id":"T8996","span":{"begin":235,"end":239},"obj":"Protein"},{"id":"T8995","span":{"begin":209,"end":211},"obj":"Protein"},{"id":"T8994","span":{"begin":58,"end":60},"obj":"Protein"}],"text":"These data demonstrate that the persistent high levels of ɛy are due to ectopic expression in the definitive erythroid cells that mature in the fetal liver, suggesting that there is an intrinsic defect of the ɛy silencing mechanism in Sox6-null mice."}