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is currently the leading cause of cancer-related death in the United States, causing 28% of all cancer deaths [1]. Although cigarette smoking is the primary risk factor, only 10–15% of heavy smokers (greater than 20 pack years) develop BC [1-3]. Antioxidant and DNA repair enzymes that provide protection from the effects of cigarette smoke are expressed in the progenitor cells for BC, normal bronchial epithelial cells (NBEC) [1]. Inherited inter-individual variation in the function of these genes plays a role in determining risk for BC [4-6]. Antioxidant enzymes protect NBEC from reactive oxygen species produced by interaction with and metabolism of xenobiotics such as pollution and cigarette smoke [4-7] as well as those produced by normal cellular metabolism. Reactive oxygen species cause many damaging reactions including denaturation of proteins, cross-linking of lipids and proteins and modification of nucleic acid bases, which can lead to cancer [7]. DNA repair enzymes repair the frequent damage to DNA caused by oxidant stress as well as other stresses, including bulky adducts derived from carcinogens in cigarette smoke [8]."}

{"project":"2_test","denotations":[{"id":"16255782-14705493-8759910","span":{"begin":243,"end":244},"obj":"14705493"},{"id":"16255782-16088748-8759910","span":{"begin":243,"end":244},"obj":"16088748"},{"id":"16255782-8242618-8759911","span":{"begin":545,"end":546},"obj":"8242618"},{"id":"16255782-1339300-8759911","span":{"begin":545,"end":546},"obj":"1339300"},{"id":"16255782-1486868-8759911","span":{"begin":545,"end":546},"obj":"1486868"},{"id":"16255782-8242618-8759912","span":{"begin":711,"end":712},"obj":"8242618"},{"id":"16255782-1339300-8759912","span":{"begin":711,"end":712},"obj":"1339300"},{"id":"16255782-1486868-8759912","span":{"begin":711,"end":712},"obj":"1486868"},{"id":"16255782-6290784-8759912","span":{"begin":711,"end":712},"obj":"6290784"},{"id":"16255782-6290784-8759913","span":{"begin":966,"end":967},"obj":"6290784"},{"id":"16255782-8533150-8759914","span":{"begin":1144,"end":1145},"obj":"8533150"}],"text":"BC is currently the leading cause of cancer-related death in the United States, causing 28% of all cancer deaths [1]. Although cigarette smoking is the primary risk factor, only 10–15% of heavy smokers (greater than 20 pack years) develop BC [1-3]. Antioxidant and DNA repair enzymes that provide protection from the effects of cigarette smoke are expressed in the progenitor cells for BC, normal bronchial epithelial cells (NBEC) [1]. Inherited inter-individual variation in the function of these genes plays a role in determining risk for BC [4-6]. Antioxidant enzymes protect NBEC from reactive oxygen species produced by interaction with and metabolism of xenobiotics such as pollution and cigarette smoke [4-7] as well as those produced by normal cellular metabolism. Reactive oxygen species cause many damaging reactions including denaturation of proteins, cross-linking of lipids and proteins and modification of nucleic acid bases, which can lead to cancer [7]. DNA repair enzymes repair the frequent damage to DNA caused by oxidant stress as well as other stresses, including bulky adducts derived from carcinogens in cigarette smoke [8]."}

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