CORD-19:f1fd5f33dcbe0aae1ce2efb7b7d22eafb4247f02 / 11440-11747
Annnotations
CORD-19_Custom_license_subset
{"project":"CORD-19_Custom_license_subset","denotations":[{"id":"T63","span":{"begin":0,"end":307},"obj":"Sentence"}],"text":"RV and RSV primarily infect airway epithelial cells, activating inflammatory pathways and increasing proinflammatory cytokine and chemokine production, such as tumor necrosis factor (TNF), IL-6, CXCL8 (IL-8), CCL5 (RANTES), granulocyte macrophage colony-stimulating factor (GM-CSF), and CCL3 (MIP-1a) (31) ."}
CORD-19-Sentences
{"project":"CORD-19-Sentences","denotations":[{"id":"TextSentencer_T63","span":{"begin":0,"end":307},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"RV and RSV primarily infect airway epithelial cells, activating inflammatory pathways and increasing proinflammatory cytokine and chemokine production, such as tumor necrosis factor (TNF), IL-6, CXCL8 (IL-8), CCL5 (RANTES), granulocyte macrophage colony-stimulating factor (GM-CSF), and CCL3 (MIP-1a) (31) ."}
CORD-19-PD-HP
{"project":"CORD-19-PD-HP","denotations":[{"id":"T88","span":{"begin":160,"end":165},"obj":"Phenotype"}],"attributes":[{"id":"A88","pred":"hp_id","subj":"T88","obj":"http://purl.obolibrary.org/obo/HP_0002664"}],"text":"RV and RSV primarily infect airway epithelial cells, activating inflammatory pathways and increasing proinflammatory cytokine and chemokine production, such as tumor necrosis factor (TNF), IL-6, CXCL8 (IL-8), CCL5 (RANTES), granulocyte macrophage colony-stimulating factor (GM-CSF), and CCL3 (MIP-1a) (31) ."}
CORD-19-PD-MONDO
{"project":"CORD-19-PD-MONDO","denotations":[{"id":"T128","span":{"begin":160,"end":165},"obj":"Disease"},{"id":"T23198","span":{"begin":160,"end":165},"obj":"Disease"}],"attributes":[{"id":"A128","pred":"mondo_id","subj":"T128","obj":"http://purl.obolibrary.org/obo/MONDO_0005070"},{"id":"A51758","pred":"mondo_id","subj":"T23198","obj":"http://purl.obolibrary.org/obo/MONDO_0005070"}],"text":"RV and RSV primarily infect airway epithelial cells, activating inflammatory pathways and increasing proinflammatory cytokine and chemokine production, such as tumor necrosis factor (TNF), IL-6, CXCL8 (IL-8), CCL5 (RANTES), granulocyte macrophage colony-stimulating factor (GM-CSF), and CCL3 (MIP-1a) (31) ."}