| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-16 |
Sentence |
denotes |
Hartnup disease. |
| TextSentencer_T2 |
17-70 |
Sentence |
denotes |
Clinical, pathological, and biochemical observations. |
| TextSentencer_T3 |
71-203 |
Sentence |
denotes |
Hartnup disease is a rare genetic disorder of amino acid transport associated with variable and intermittent clinical abnormalities. |
| TextSentencer_T4 |
204-376 |
Sentence |
denotes |
A family is described in which three siblings had an intermittently progressive neurological disease and two of the affected siblings had the Hartnup-pattern aminoaciduria. |
| TextSentencer_T5 |
377-532 |
Sentence |
denotes |
Neuropathological examination of one case showed severe diffuse atrophy, generalized neuronal loss in the cortex, and Purkinje cell loss in the cerebellum. |
| TextSentencer_T6 |
533-858 |
Sentence |
denotes |
In vivo and in vitro studies of intestinal amino acid transport in the surviving sibling indicated a partial defect in the transport of several neutral amino acids (tryptophan, alanine, serine, and methionine) with normal transport of other neutral amino acids (threonine, phenylalanine, histidine, tyrosine, and isoleucine). |
| TextSentencer_T7 |
859-948 |
Sentence |
denotes |
Transport of glycine, proline, hydroxyproline, and the basic amino acids appeared normal. |
| T1 |
0-16 |
Sentence |
denotes |
Hartnup disease. |
| T2 |
17-70 |
Sentence |
denotes |
Clinical, pathological, and biochemical observations. |
| T3 |
71-203 |
Sentence |
denotes |
Hartnup disease is a rare genetic disorder of amino acid transport associated with variable and intermittent clinical abnormalities. |
| T4 |
204-376 |
Sentence |
denotes |
A family is described in which three siblings had an intermittently progressive neurological disease and two of the affected siblings had the Hartnup-pattern aminoaciduria. |
| T5 |
377-532 |
Sentence |
denotes |
Neuropathological examination of one case showed severe diffuse atrophy, generalized neuronal loss in the cortex, and Purkinje cell loss in the cerebellum. |
| T6 |
533-858 |
Sentence |
denotes |
In vivo and in vitro studies of intestinal amino acid transport in the surviving sibling indicated a partial defect in the transport of several neutral amino acids (tryptophan, alanine, serine, and methionine) with normal transport of other neutral amino acids (threonine, phenylalanine, histidine, tyrosine, and isoleucine). |
| T7 |
859-948 |
Sentence |
denotes |
Transport of glycine, proline, hydroxyproline, and the basic amino acids appeared normal. |