| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-134 |
Sentence |
denotes |
Systemic mastocytosis associated with acute myeloid leukaemia: report of two cases and detection of the c-kit mutation Asp-816 to Val. |
| TextSentencer_T2 |
135-226 |
Sentence |
denotes |
A subset of patients with systemic mastocytosis (SM) develop acute myeloid leukaemia (AML). |
| TextSentencer_T3 |
227-342 |
Sentence |
denotes |
However, little is known about the biology of such leukaemias and their relationship to the mast cell (MC) lineage. |
| TextSentencer_T4 |
343-405 |
Sentence |
denotes |
We report on two female patients who suffered from SM and AML. |
| TextSentencer_T5 |
406-509 |
Sentence |
denotes |
According to FAB criteria, the leukaemias were classified as AML-M4 (patient 1) and AML-MO (patient 2). |
| TextSentencer_T6 |
510-673 |
Sentence |
denotes |
The coexistence of the two distinct neoplasms (AML and SM) was demonstrable by immunostaining of serial bone marrow (BM) sections with monoclonal antibodies (mAb). |
| TextSentencer_T7 |
674-835 |
Sentence |
denotes |
In particular, the MC infiltrates were found to react with mAb against MC-tryptase and MC growth factor receptor c-kit (CD117), but not with mAb to CD15 or CD34. |
| TextSentencer_T8 |
836-955 |
Sentence |
denotes |
In contrast, the AML blasts were immunoreactive for CD15 (patient 1) or CD34 (patient 2), but did not express tryptase. |
| TextSentencer_T9 |
956-1137 |
Sentence |
denotes |
The c-kit point mutation Asp-->Val at codon 816, considered to play a role in the transformation of MC progenitors, was detected in patient 1 in a BM cell fraction containing 4% MC. |
| TextSentencer_T10 |
1138-1203 |
Sentence |
denotes |
However, no c-kit mutation was found in pure AML blasts (<1% MC). |
| TextSentencer_T11 |
1204-1310 |
Sentence |
denotes |
These findings argue against an evolution of the AML clone from neoplastic MC or MC-committed progenitors. |
| T1 |
0-134 |
Sentence |
denotes |
Systemic mastocytosis associated with acute myeloid leukaemia: report of two cases and detection of the c-kit mutation Asp-816 to Val. |
| T2 |
135-226 |
Sentence |
denotes |
A subset of patients with systemic mastocytosis (SM) develop acute myeloid leukaemia (AML). |
| T3 |
227-342 |
Sentence |
denotes |
However, little is known about the biology of such leukaemias and their relationship to the mast cell (MC) lineage. |
| T4 |
343-405 |
Sentence |
denotes |
We report on two female patients who suffered from SM and AML. |
| T5 |
406-509 |
Sentence |
denotes |
According to FAB criteria, the leukaemias were classified as AML-M4 (patient 1) and AML-MO (patient 2). |
| T6 |
510-673 |
Sentence |
denotes |
The coexistence of the two distinct neoplasms (AML and SM) was demonstrable by immunostaining of serial bone marrow (BM) sections with monoclonal antibodies (mAb). |
| T7 |
674-835 |
Sentence |
denotes |
In particular, the MC infiltrates were found to react with mAb against MC-tryptase and MC growth factor receptor c-kit (CD117), but not with mAb to CD15 or CD34. |
| T8 |
836-955 |
Sentence |
denotes |
In contrast, the AML blasts were immunoreactive for CD15 (patient 1) or CD34 (patient 2), but did not express tryptase. |
| T9 |
956-1137 |
Sentence |
denotes |
The c-kit point mutation Asp-->Val at codon 816, considered to play a role in the transformation of MC progenitors, was detected in patient 1 in a BM cell fraction containing 4% MC. |
| T10 |
1138-1203 |
Sentence |
denotes |
However, no c-kit mutation was found in pure AML blasts (<1% MC). |
| T11 |
1204-1310 |
Sentence |
denotes |
These findings argue against an evolution of the AML clone from neoplastic MC or MC-committed progenitors. |