| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-197 |
Sentence |
denotes |
Prospective study of coronary heart disease incidence in relation to fasting total homocysteine, related genetic polymorphisms, and B vitamins: the Atherosclerosis Risk in Communities (ARIC) study. |
| TextSentencer_T2 |
198-209 |
Sentence |
denotes |
BACKGROUND: |
| TextSentencer_T3 |
210-375 |
Sentence |
denotes |
Elevated plasma total homocysteine (tHcy), low B-vitamin intake, and genetic polymorphisms related to tHcy metabolism may play roles in coronary heart disease (CHD). |
| TextSentencer_T4 |
376-412 |
Sentence |
denotes |
More prospective studies are needed. |
| TextSentencer_T5 |
413-433 |
Sentence |
denotes |
METHODS AND RESULTS: |
| TextSentencer_T6 |
434-646 |
Sentence |
denotes |
We used a prospective case-cohort design to determine whether tHcy-related factors are associated with incidence of CHD over an average of 3.3 years of follow-up in a biracial sample of middle-aged men and women. |
| TextSentencer_T7 |
647-928 |
Sentence |
denotes |
Age-, race-, and field center-adjusted CHD incidence was associated positively (P<0.05) with tHcy in women but not men, and CHD was associated negatively (P<0.05) with plasma folate (women only), plasma pyridoxal 5'-phosphate (both sexes), and vitamin supplementation (women only). |
| TextSentencer_T8 |
929-1161 |
Sentence |
denotes |
However, after accounting for other risk factors, only plasma pyridoxal 5'-phosphate was associated with CHD incidence; the relative risk for the highest versus lowest quintile of pyridoxal 5'-phosphate was 0.28 (95% CI=0.1 to 0.7). |
| TextSentencer_T9 |
1162-1326 |
Sentence |
denotes |
There was no association of CHD with the C677T mutation of the methylenetetrahydrofolate reductase gene or with 3 mutations of the cystathionine beta-synthase gene. |
| TextSentencer_T10 |
1327-1339 |
Sentence |
denotes |
CONCLUSIONS: |
| TextSentencer_T11 |
1340-1505 |
Sentence |
denotes |
Our prospective findings add uncertainty to conclusions derived mostly from cross-sectional studies that tHcy is a major, independent, causative risk factor for CHD. |
| TextSentencer_T12 |
1506-1604 |
Sentence |
denotes |
Our findings point more strongly to the possibility that vitamin B6 offers independent protection. |
| TextSentencer_T13 |
1605-1753 |
Sentence |
denotes |
Randomized trials, some of which are under way, are needed to better clarify the interrelationships of tHcy, B vitamins, and cardiovascular disease. |
| T1 |
0-197 |
Sentence |
denotes |
Prospective study of coronary heart disease incidence in relation to fasting total homocysteine, related genetic polymorphisms, and B vitamins: the Atherosclerosis Risk in Communities (ARIC) study. |
| T2 |
198-209 |
Sentence |
denotes |
BACKGROUND: |
| T3 |
210-375 |
Sentence |
denotes |
Elevated plasma total homocysteine (tHcy), low B-vitamin intake, and genetic polymorphisms related to tHcy metabolism may play roles in coronary heart disease (CHD). |
| T4 |
376-412 |
Sentence |
denotes |
More prospective studies are needed. |
| T5 |
413-433 |
Sentence |
denotes |
METHODS AND RESULTS: |
| T6 |
434-646 |
Sentence |
denotes |
We used a prospective case-cohort design to determine whether tHcy-related factors are associated with incidence of CHD over an average of 3.3 years of follow-up in a biracial sample of middle-aged men and women. |
| T7 |
647-928 |
Sentence |
denotes |
Age-, race-, and field center-adjusted CHD incidence was associated positively (P<0.05) with tHcy in women but not men, and CHD was associated negatively (P<0.05) with plasma folate (women only), plasma pyridoxal 5'-phosphate (both sexes), and vitamin supplementation (women only). |
| T8 |
929-1161 |
Sentence |
denotes |
However, after accounting for other risk factors, only plasma pyridoxal 5'-phosphate was associated with CHD incidence; the relative risk for the highest versus lowest quintile of pyridoxal 5'-phosphate was 0.28 (95% CI=0.1 to 0.7). |
| T9 |
1162-1326 |
Sentence |
denotes |
There was no association of CHD with the C677T mutation of the methylenetetrahydrofolate reductase gene or with 3 mutations of the cystathionine beta-synthase gene. |
| T10 |
1327-1339 |
Sentence |
denotes |
CONCLUSIONS: |
| T11 |
1340-1505 |
Sentence |
denotes |
Our prospective findings add uncertainty to conclusions derived mostly from cross-sectional studies that tHcy is a major, independent, causative risk factor for CHD. |
| T12 |
1506-1604 |
Sentence |
denotes |
Our findings point more strongly to the possibility that vitamin B6 offers independent protection. |
| T13 |
1605-1753 |
Sentence |
denotes |
Randomized trials, some of which are under way, are needed to better clarify the interrelationships of tHcy, B vitamins, and cardiovascular disease. |