PubMed:9108409
Annnotations
Inflammaging
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 0-124 | Sentence | denotes | Two distinct pathways of interleukin-5 synthesis in allergen-specific human T-cell clones are suppressed by glucocorticoids. |
| T2 | 125-307 | Sentence | denotes | Glucocorticoids (GC) have long been used as the most effective agents for the treatment of allergic diseases accompanied by eosinophilia such as chronic asthma and atopic dermatitis. |
| T3 | 308-468 | Sentence | denotes | The development of chronic eosinophilic inflammation is dependent on interleukin-5 (IL-5), a selective eosinophil-activating factor, produced by helper T cells. |
| T4 | 469-609 | Sentence | denotes | To delineate the regulatory mechanisms of human IL-5 synthesis, we established allergen-specific CD4+ T-cell clones from asthmatic patients. |
| T5 | 610-736 | Sentence | denotes | GC efficiently suppressed IL-5 synthesis of T-cell clones activated via either T-cell receptor (TCR) or IL-2 receptor (IL-2R). |
| T6 | 737-830 | Sentence | denotes | Induction of IL-5 mRNA upon TCR and IL-2R stimulation was totally inhibited by dexamethasone. |
| T7 | 831-1205 | Sentence | denotes | Human IL-5 promoter/enhancer-luciferase gene construct transfected to T-cell clones was transcribed on either TCR or IL-2R stimulation and was clearly downregulated by dexamethasone, indicating that the approximately 500-bp human IL-5 gene segment located 5' upstream of the coding region contains activation-inducible enhancer elements responsible for the regulation by GC. |
| T8 | 1206-1357 | Sentence | denotes | Electrophoretic mobility shift assay analysis suggested that AP-1 and NF-kappaB are among the possible targets of GC actions on TCR-stimulated T cells. |
| T9 | 1358-1429 | Sentence | denotes | NF-AT and NF-kappaB were not significantly induced by IL-2 stimulation. |
| T10 | 1430-1678 | Sentence | denotes | Our results showing that GC suppressed IL-5 production by human CD4+ T cells activated by two distinct stimuli, TCR and IL-2R stimulation, underscore the efficacy of GC in the treatment of allergic diseases via suppression of T-cell IL-5 synthesis. |
| T1 | 0-124 | Sentence | denotes | Two distinct pathways of interleukin-5 synthesis in allergen-specific human T-cell clones are suppressed by glucocorticoids. |
| T2 | 125-307 | Sentence | denotes | Glucocorticoids (GC) have long been used as the most effective agents for the treatment of allergic diseases accompanied by eosinophilia such as chronic asthma and atopic dermatitis. |
| T3 | 308-468 | Sentence | denotes | The development of chronic eosinophilic inflammation is dependent on interleukin-5 (IL-5), a selective eosinophil-activating factor, produced by helper T cells. |
| T4 | 469-609 | Sentence | denotes | To delineate the regulatory mechanisms of human IL-5 synthesis, we established allergen-specific CD4+ T-cell clones from asthmatic patients. |
| T5 | 610-736 | Sentence | denotes | GC efficiently suppressed IL-5 synthesis of T-cell clones activated via either T-cell receptor (TCR) or IL-2 receptor (IL-2R). |
| T6 | 737-830 | Sentence | denotes | Induction of IL-5 mRNA upon TCR and IL-2R stimulation was totally inhibited by dexamethasone. |
| T7 | 831-1205 | Sentence | denotes | Human IL-5 promoter/enhancer-luciferase gene construct transfected to T-cell clones was transcribed on either TCR or IL-2R stimulation and was clearly downregulated by dexamethasone, indicating that the approximately 500-bp human IL-5 gene segment located 5' upstream of the coding region contains activation-inducible enhancer elements responsible for the regulation by GC. |
| T8 | 1206-1357 | Sentence | denotes | Electrophoretic mobility shift assay analysis suggested that AP-1 and NF-kappaB are among the possible targets of GC actions on TCR-stimulated T cells. |
| T9 | 1358-1429 | Sentence | denotes | NF-AT and NF-kappaB were not significantly induced by IL-2 stimulation. |
| T10 | 1430-1678 | Sentence | denotes | Our results showing that GC suppressed IL-5 production by human CD4+ T cells activated by two distinct stimuli, TCR and IL-2R stimulation, underscore the efficacy of GC in the treatment of allergic diseases via suppression of T-cell IL-5 synthesis. |
jnlpba-st-training
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 25-38 | protein | denotes | interleukin-5 |
| T2 | 52-89 | cell_line | denotes | allergen-specific human T-cell clones |
| T3 | 377-390 | protein | denotes | interleukin-5 |
| T4 | 392-396 | protein | denotes | IL-5 |
| T5 | 401-439 | protein | denotes | selective eosinophil-activating factor |
| T6 | 453-467 | cell_type | denotes | helper T cells |
| T7 | 517-521 | protein | denotes | IL-5 |
| T8 | 548-584 | cell_line | denotes | allergen-specific CD4+ T-cell clones |
| T9 | 636-640 | protein | denotes | IL-5 |
| T10 | 689-704 | protein | denotes | T-cell receptor |
| T11 | 706-709 | protein | denotes | TCR |
| T12 | 714-727 | protein | denotes | IL-2 receptor |
| T13 | 729-734 | protein | denotes | IL-2R |
| T14 | 750-759 | RNA | denotes | IL-5 mRNA |
| T15 | 765-768 | protein | denotes | TCR |
| T16 | 773-778 | protein | denotes | IL-2R |
| T17 | 837-841 | protein | denotes | IL-5 |
| T18 | 901-914 | cell_line | denotes | T-cell clones |
| T19 | 941-944 | protein | denotes | TCR |
| T20 | 948-953 | protein | denotes | IL-2R |
| T21 | 1048-1078 | DNA | denotes | 500-bp human IL-5 gene segment |
| T22 | 1087-1098 | DNA | denotes | 5' upstream |
| T23 | 1129-1167 | DNA | denotes | activation-inducible enhancer elements |
| T24 | 1267-1271 | protein | denotes | AP-1 |
| T25 | 1276-1285 | protein | denotes | NF-kappaB |
| T26 | 1334-1356 | cell_type | denotes | TCR-stimulated T cells |
| T27 | 1358-1363 | protein | denotes | NF-AT |
| T28 | 1368-1377 | protein | denotes | NF-kappaB |
| T29 | 1412-1416 | protein | denotes | IL-2 |
| T30 | 1469-1473 | protein | denotes | IL-5 |
| T31 | 1488-1506 | cell_type | denotes | human CD4+ T cells |
| T32 | 1542-1545 | protein | denotes | TCR |
| T33 | 1550-1555 | protein | denotes | IL-2R |
| T34 | 1663-1667 | protein | denotes | IL-5 |
genia-medco-coref
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| C1 | 108-123 | NP | denotes | glucocorticoids |
| C2 | 125-145 | NP | denotes | Glucocorticoids (GC) |
| C3 | 377-397 | NP | denotes | interleukin-5 (IL-5) |
| C4 | 399-439 | NP | denotes | a selective eosinophil-activating factor |
| C5 | 548-608 | NP | denotes | allergen-specific CD4+ T-cell clones from asthmatic patients |
| C6 | 610-612 | NP | denotes | GC |
| C7 | 654-667 | NP | denotes | T-cell clones |
| C8 | 765-790 | NP | denotes | TCR and IL-2R stimulation |
| C9 | 816-829 | NP | denotes | dexamethasone |
| C10 | 901-914 | NP | denotes | T-cell clones |
| C11 | 934-965 | NP | denotes | either TCR or IL-2R stimulation |
| C12 | 999-1012 | NP | denotes | dexamethasone |
| C13 | 1202-1204 | NP | denotes | GC |
| C14 | 1276-1285 | NP | denotes | NF-kappaB |
| C15 | 1368-1377 | NP | denotes | NF-kappaB |
| C16 | 1455-1457 | NP | denotes | GC |
| C17 | 1520-1540 | NP | denotes | two distinct stimuli |
| C18 | 1542-1567 | NP | denotes | TCR and IL-2R stimulation |
| C19 | 1596-1598 | NP | denotes | GC |
| R1 | C2 | C1 | coref-ident | Glucocorticoids (GC),glucocorticoids |
| R2 | C4 | C3 | coref-appos | a selective eosinophil-activating factor,interleukin-5 (IL-5) |
| R3 | C6 | C2 | coref-ident | GC,Glucocorticoids (GC) |
| R4 | C7 | C5 | coref-ident | T-cell clones,allergen-specific CD4+ T-cell clones from asthmatic patients |
| R5 | C10 | C7 | coref-ident | T-cell clones,T-cell clones |
| R6 | C11 | C8 | coref-ident | either TCR or IL-2R stimulation,TCR and IL-2R stimulation |
| R7 | C12 | C9 | coref-ident | dexamethasone,dexamethasone |
| R8 | C13 | C6 | coref-ident | GC,GC |
| R9 | C15 | C14 | coref-ident | NF-kappaB,NF-kappaB |
| R10 | C16 | C13 | coref-ident | GC,GC |
| R11 | C17 | C8 | coref-ident | two distinct stimuli,TCR and IL-2R stimulation |
| R12 | C18 | C17 | coref-appos | TCR and IL-2R stimulation,two distinct stimuli |
| R13 | C19 | C16 | coref-ident | GC,GC |
pubmed-sentences-benchmark
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| S1 | 0-124 | Sentence | denotes | Two distinct pathways of interleukin-5 synthesis in allergen-specific human T-cell clones are suppressed by glucocorticoids. |
| S2 | 125-307 | Sentence | denotes | Glucocorticoids (GC) have long been used as the most effective agents for the treatment of allergic diseases accompanied by eosinophilia such as chronic asthma and atopic dermatitis. |
| S3 | 308-468 | Sentence | denotes | The development of chronic eosinophilic inflammation is dependent on interleukin-5 (IL-5), a selective eosinophil-activating factor, produced by helper T cells. |
| S4 | 469-609 | Sentence | denotes | To delineate the regulatory mechanisms of human IL-5 synthesis, we established allergen-specific CD4+ T-cell clones from asthmatic patients. |
| S5 | 610-736 | Sentence | denotes | GC efficiently suppressed IL-5 synthesis of T-cell clones activated via either T-cell receptor (TCR) or IL-2 receptor (IL-2R). |
| S6 | 737-830 | Sentence | denotes | Induction of IL-5 mRNA upon TCR and IL-2R stimulation was totally inhibited by dexamethasone. |
| S7 | 831-1205 | Sentence | denotes | Human IL-5 promoter/enhancer-luciferase gene construct transfected to T-cell clones was transcribed on either TCR or IL-2R stimulation and was clearly downregulated by dexamethasone, indicating that the approximately 500-bp human IL-5 gene segment located 5' upstream of the coding region contains activation-inducible enhancer elements responsible for the regulation by GC. |
| S8 | 1206-1357 | Sentence | denotes | Electrophoretic mobility shift assay analysis suggested that AP-1 and NF-kappaB are among the possible targets of GC actions on TCR-stimulated T cells. |
| S9 | 1358-1429 | Sentence | denotes | NF-AT and NF-kappaB were not significantly induced by IL-2 stimulation. |
| S10 | 1430-1678 | Sentence | denotes | Our results showing that GC suppressed IL-5 production by human CD4+ T cells activated by two distinct stimuli, TCR and IL-2R stimulation, underscore the efficacy of GC in the treatment of allergic diseases via suppression of T-cell IL-5 synthesis. |
GENIAcorpus
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 25-38 | protein_molecule | denotes | interleukin-5 |
| T2 | 52-89 | cell_line | denotes | allergen-specific human T-cell clones |
| T3 | 108-123 | lipid | denotes | glucocorticoids |
| T4 | 125-140 | lipid | denotes | Glucocorticoids |
| T5 | 142-144 | lipid | denotes | GC |
| T6 | 216-233 | other_name | denotes | allergic diseases |
| T7 | 249-261 | other_name | denotes | eosinophilia |
| T8 | 270-284 | other_name | denotes | chronic asthma |
| T9 | 289-306 | other_name | denotes | atopic dermatitis |
| T10 | 327-360 | other_name | denotes | chronic eosinophilic inflammation |
| T11 | 377-390 | protein_molecule | denotes | interleukin-5 |
| T12 | 392-396 | protein_molecule | denotes | IL-5 |
| T13 | 401-439 | protein_family_or_group | denotes | selective eosinophil-activating factor |
| T14 | 453-467 | cell_type | denotes | helper T cells |
| T15 | 511-516 | other_name | denotes | human |
| T16 | 517-521 | protein_molecule | denotes | IL-5 |
| T17 | 548-584 | cell_line | denotes | allergen-specific CD4+ T-cell clones |
| T18 | 590-608 | multi_cell | denotes | asthmatic patients |
| T19 | 610-612 | lipid | denotes | GC |
| T20 | 636-640 | protein_molecule | denotes | IL-5 |
| T21 | 689-704 | protein_family_or_group | denotes | T-cell receptor |
| T22 | 706-709 | protein_family_or_group | denotes | TCR |
| T23 | 714-727 | protein_molecule | denotes | IL-2 receptor |
| T24 | 729-734 | protein_molecule | denotes | IL-2R |
| T25 | 750-754 | protein_molecule | denotes | IL-5 |
| T26 | 765-768 | protein_family_or_group | denotes | TCR |
| T27 | 773-778 | protein_molecule | denotes | IL-2R |
| T28 | 816-829 | lipid | denotes | dexamethasone |
| T29 | 837-841 | protein_molecule | denotes | IL-5 |
| T30 | 901-914 | cell_line | denotes | T-cell clones |
| T31 | 941-944 | protein_family_or_group | denotes | TCR |
| T32 | 948-953 | protein_molecule | denotes | IL-2R |
| T33 | 999-1012 | lipid | denotes | dexamethasone |
| T34 | 1048-1060 | DNA_domain_or_region | denotes | 500-bp human |
| T35 | 1061-1065 | protein_molecule | denotes | IL-5 |
| T36 | 1087-1098 | DNA_domain_or_region | denotes | 5' upstream |
| T37 | 1129-1167 | DNA_family_or_group | denotes | activation-inducible enhancer elements |
| T38 | 1202-1204 | lipid | denotes | GC |
| T39 | 1206-1251 | other_name | denotes | Electrophoretic mobility shift assay analysis |
| T40 | 1267-1271 | protein_molecule | denotes | AP-1 |
| T41 | 1276-1285 | protein_complex | denotes | NF-kappaB |
| T42 | 1320-1322 | lipid | denotes | GC |
| T43 | 1334-1337 | protein_family_or_group | denotes | TCR |
| T44 | 1358-1363 | protein_molecule | denotes | NF-AT |
| T45 | 1368-1377 | protein_complex | denotes | NF-kappaB |
| T46 | 1412-1416 | protein_molecule | denotes | IL-2 |
| T47 | 1455-1457 | lipid | denotes | GC |
| T48 | 1469-1473 | protein_molecule | denotes | IL-5 |
| T49 | 1488-1506 | cell_type | denotes | human CD4+ T cells |
| T50 | 1542-1545 | protein_family_or_group | denotes | TCR |
| T51 | 1550-1555 | protein_molecule | denotes | IL-2R |
| T52 | 1596-1598 | lipid | denotes | GC |
| T53 | 1619-1636 | other_name | denotes | allergic diseases |
| T54 | 1656-1662 | other_name | denotes | T-cell |
| T55 | 1663-1667 | protein_molecule | denotes | IL-5 |