| Id |
Subject |
Object |
Predicate |
Lexical cue |
| S1 |
0-60 |
Sentence |
denotes |
[Molecular mechanisms of age-related lymphocyte dysfunction] |
| S2 |
62-143 |
Sentence |
denotes |
Aging is classically accompanied by a dysregulation of the immunologic machinery. |
| S3 |
144-308 |
Sentence |
denotes |
As a consequence, the immune response developed in senescent organisms is usually inappropriate, often inefficient, sometimes aberrant, and potentially detrimental. |
| S4 |
309-510 |
Sentence |
denotes |
The age-associated immune dysfunction may be implicated to some degree in the extreme susceptibility of the elderly to infection and neoplasia and may even participate in various aspects of senescence. |
| S5 |
511-614 |
Sentence |
denotes |
The current understanding of the molecular mechanisms underlying immunosenescence is still fragmentary. |
| S6 |
615-742 |
Sentence |
denotes |
The most extensively studied phenomenon is the progressive decline in the proliferative capacities of T lymphocytes with aging. |
| S7 |
743-863 |
Sentence |
denotes |
The loss of proliferative potential in response to antigenic challenge is a characteristic feature of immune senescence. |
| S8 |
864-944 |
Sentence |
denotes |
It is directly implicated in the emergence of the age-related immune deficiency. |
| S9 |
945-1159 |
Sentence |
denotes |
The purpose of this review is to show how the accumulation of various biochemical lesions with advancing age leads to the failure of a critical cell function, namely the activation-induced lymphocyte proliferation. |
| S10 |
1160-1309 |
Sentence |
denotes |
The biochemical modifications responsible for the defect in transduction and execution of the proliferative signal are analyzed as a function of age. |
| S11 |
1310-1550 |
Sentence |
denotes |
The multiple alterations observed on the various biochemical pathways may appear as a consequence of a unique deleterious mechanism more fundamentally related to the process of senescence such as the inability to cope with oxidative stress. |