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PubMed:8486665 / 1638-1862 JSONTXT

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sentences

Id Subject Object Predicate Lexical cue
T12 0-224 Sentence denotes These studies suggest that, as in Marfan's syndrome where the primary defect concerns the fibrillin gene, reduced decorin expression contributes to the formation of an abnormal matrix and the pathogenesis of these disorders.
T12 0-224 Sentence denotes These studies suggest that, as in Marfan's syndrome where the primary defect concerns the fibrillin gene, reduced decorin expression contributes to the formation of an abnormal matrix and the pathogenesis of these disorders.

DisGeNET5_gene_disease

Id Subject Object Predicate Lexical cue
8486665-11#114#121#gene1634 114-121 gene1634 denotes decorin
8486665-11#34#51#diseaseC0024796 34-51 diseaseC0024796 denotes Marfan's syndrome
114#121#gene163434#51#diseaseC0024796 8486665-11#114#121#gene1634 8486665-11#34#51#diseaseC0024796 associated_with decorin,Marfan's syndrome

mondo_disease

Id Subject Object Predicate Lexical cue mondo_id
T2 34-51 Disease denotes Marfan's syndrome http://purl.obolibrary.org/obo/MONDO_0007947

GlyCosmos15-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T2 34-51 Disease denotes Marfan's syndrome http://purl.obolibrary.org/obo/MONDO_0007947

sentences

Id Subject Object Predicate Lexical cue
T12 0-224 Sentence denotes These studies suggest that, as in Marfan's syndrome where the primary defect concerns the fibrillin gene, reduced decorin expression contributes to the formation of an abnormal matrix and the pathogenesis of these disorders.
T12 0-224 Sentence denotes These studies suggest that, as in Marfan's syndrome where the primary defect concerns the fibrillin gene, reduced decorin expression contributes to the formation of an abnormal matrix and the pathogenesis of these disorders.

GlyCosmos15-Sentences

Id Subject Object Predicate Lexical cue
T12 0-224 Sentence denotes These studies suggest that, as in Marfan's syndrome where the primary defect concerns the fibrillin gene, reduced decorin expression contributes to the formation of an abnormal matrix and the pathogenesis of these disorders.