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2015-BEL-Sample

Id Subject Object Predicate Lexical cue
T1 878-1128 a(CHEBI:glucocorticoid) increases p(MGI:Resp18) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar

2015-BEL-Sample-2

Id Subject Object Predicate Lexical cue
BEL:20000014 878-1128 a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar
BEL:20000016 878-1128 a(CHEBI:glucocorticoid) increases p(MGI:Resp18) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar
BEL:20024020 878-1262 a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar. Surprisingly, RESP18 was not processed to smaller peptides to any significant extent, nor was RESP18 or any smaller peptide secreted
BEL:20024022 878-1262 a(CHEBI:glucocorticoid) increases p(MGI:Resp18) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar. Surprisingly, RESP18 was not processed to smaller peptides to any significant extent, nor was RESP18 or any smaller peptide secreted
BEL:20000014 878-1128 a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar
BEL:20000016 878-1128 a(CHEBI:glucocorticoid) increases p(MGI:Resp18) denotes Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar