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PubMed:5676517 JSONTXT

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Inflammaging

Id Subject Object Predicate Lexical cue
T1 0-63 Sentence denotes Hemodynamic effects of isoproterenol in canine endotoxin shock.
T2 64-187 Sentence denotes Myocardial function and peripheral hemodynamic alterations were measured through the late stages of canine endotoxin shock.
T3 188-338 Sentence denotes 60 min postendotoxin paired animals were given infusions of either 5 ml/kg per hr of 5% dextrose or dextrose plus isoproterenol (0.25 mug/kg per min).
T4 339-467 Sentence denotes Comparable blood lactic and pyruvic acid levels were determined, the excess lactic acid calculated, and pH values were obtained.
T5 468-610 Sentence denotes During the initial stages the classic pattern of hemodynamic alterations was observed; an excess of lactic acid appeared and the pH decreased.
T6 611-907 Sentence denotes Outstanding was evidence of markedly reduced myocardial function in the late stages of shock with progressive rise in left ventricular end diastolic pressure (LVEDP), low cardiac index, rise of central venous pressure, increased central blood volume, tachycardia, and declining arterial pressure.
T7 908-987 Sentence denotes Analyses of left ventricular function curves also indicated myocardial failure.
T8 988-1141 Sentence denotes Infusion of dextrose alone failed to decrease mortality rate (10 of 18 dying), whereas the rate was significantly decreased with isoproterenol (2 of 18).
T9 1142-1196 Sentence denotes Dextrose infusion did not benefit myocardial function.
T10 1197-1360 Sentence denotes Isoproterenol resulted in a marked improvement in myocardial action with a significant increase in heart work associated with, yet very minor, increments of LVEDP.
T11 1361-1463 Sentence denotes In addition, tachycardia subsided, peripheral resistance decreased, and the blood pressure stabilized.
T12 1464-1588 Sentence denotes The prognostic value of excess lactic acid was doubtful but a progressive fall in later stages was associated with survival.
T1 0-63 Sentence denotes Hemodynamic effects of isoproterenol in canine endotoxin shock.
T2 64-187 Sentence denotes Myocardial function and peripheral hemodynamic alterations were measured through the late stages of canine endotoxin shock.
T3 188-338 Sentence denotes 60 min postendotoxin paired animals were given infusions of either 5 ml/kg per hr of 5% dextrose or dextrose plus isoproterenol (0.25 mug/kg per min).
T4 339-467 Sentence denotes Comparable blood lactic and pyruvic acid levels were determined, the excess lactic acid calculated, and pH values were obtained.
T5 468-610 Sentence denotes During the initial stages the classic pattern of hemodynamic alterations was observed; an excess of lactic acid appeared and the pH decreased.
T6 611-907 Sentence denotes Outstanding was evidence of markedly reduced myocardial function in the late stages of shock with progressive rise in left ventricular end diastolic pressure (LVEDP), low cardiac index, rise of central venous pressure, increased central blood volume, tachycardia, and declining arterial pressure.
T7 908-987 Sentence denotes Analyses of left ventricular function curves also indicated myocardial failure.
T8 988-1141 Sentence denotes Infusion of dextrose alone failed to decrease mortality rate (10 of 18 dying), whereas the rate was significantly decreased with isoproterenol (2 of 18).
T9 1142-1196 Sentence denotes Dextrose infusion did not benefit myocardial function.
T10 1197-1360 Sentence denotes Isoproterenol resulted in a marked improvement in myocardial action with a significant increase in heart work associated with, yet very minor, increments of LVEDP.
T11 1361-1463 Sentence denotes In addition, tachycardia subsided, peripheral resistance decreased, and the blood pressure stabilized.
T12 1464-1588 Sentence denotes The prognostic value of excess lactic acid was doubtful but a progressive fall in later stages was associated with survival.