Id |
Subject |
Object |
Predicate |
Lexical cue |
T1 |
0-201 |
Sentence |
denotes |
Ascorbic Acid 2-Glucoside Stably Promotes the Primitiveness of Embryonic and Mesenchymal Stem Cells Through Ten-Eleven Translocation- and cAMP-Responsive Element-Binding Protein-1-Dependent Mechanisms. |
T2 |
202-207 |
Sentence |
denotes |
Aims: |
T3 |
208-334 |
Sentence |
denotes |
The naive or primitive states of stem cells (SCs) residing in specific niches are unstable and difficult to preserve in vitro. |
T4 |
335-462 |
Sentence |
denotes |
Vitamin C (VitC), in addition to suppressing oxygen radicals, exerts pleiotropic effects to preserve the core functions of SCs. |
T5 |
463-605 |
Sentence |
denotes |
However, this compound is labile and readily oxidized, resulting in cellular toxicity and preventing its reliable application in this context. |
T6 |
606-826 |
Sentence |
denotes |
We found that a VitC derivative, ascorbic acid 2-glucoside (AA2G), stably maintains the naive pluripotency of murine embryonic SCs (mESCs) and the primitiveness of human mesenchymal SCs (hMSCs) without cellular toxicity. |
T7 |
827-835 |
Sentence |
denotes |
Results: |
T8 |
836-968 |
Sentence |
denotes |
The beneficial effects of AA2G and related molecular mechanisms were evaluated in mESCs, induced pluripotent-SCs (iPSCs), and hMSCs. |
T9 |
969-1071 |
Sentence |
denotes |
AA2G was stable in aqueous solution and barely induced cellular toxicity in cultured SCs, unlike VitC. |
T10 |
1072-1281 |
Sentence |
denotes |
AA2G supplementation recapitulated the well-known effects of VitC, including induction of ten-eleven translocation-dependent DNA demethylation in mESCs and suppression of p53 during generation of murine iPSCs. |
T11 |
1282-1464 |
Sentence |
denotes |
Furthermore, supplementation of hMSCs with AA2G improved therapeutic outcomes in an asthma mouse model by promoting their self-renewal, engraftment, and anti-inflammatory properties. |
T12 |
1465-1657 |
Sentence |
denotes |
Particularly, activation of the cAMP-responsive element-binding protein-1 (CREB1) pathway contributed to the ability of AA2G to maintain naive pluripotency of mESCs and functionality of hMSCs. |
T13 |
1658-1684 |
Sentence |
denotes |
Innovation and Conclusion: |
T14 |
1685-1912 |
Sentence |
denotes |
Given its long-lasting effects and low cellular toxicity, AA2G supplementation is useful to support the naive pluripotency of mESCs and the primitiveness of hMSCs, affecting their developmental potency and therapeutic efficacy. |
T15 |
1913-2014 |
Sentence |
denotes |
Furthermore, we demonstrate the significance of the CREB1 pathway in the mechanism of action of AA2G. |
T1 |
0-201 |
Sentence |
denotes |
Ascorbic Acid 2-Glucoside Stably Promotes the Primitiveness of Embryonic and Mesenchymal Stem Cells Through Ten-Eleven Translocation- and cAMP-Responsive Element-Binding Protein-1-Dependent Mechanisms. |
T2 |
202-207 |
Sentence |
denotes |
Aims: |
T3 |
208-334 |
Sentence |
denotes |
The naive or primitive states of stem cells (SCs) residing in specific niches are unstable and difficult to preserve in vitro. |
T4 |
335-462 |
Sentence |
denotes |
Vitamin C (VitC), in addition to suppressing oxygen radicals, exerts pleiotropic effects to preserve the core functions of SCs. |
T5 |
463-605 |
Sentence |
denotes |
However, this compound is labile and readily oxidized, resulting in cellular toxicity and preventing its reliable application in this context. |
T6 |
606-826 |
Sentence |
denotes |
We found that a VitC derivative, ascorbic acid 2-glucoside (AA2G), stably maintains the naive pluripotency of murine embryonic SCs (mESCs) and the primitiveness of human mesenchymal SCs (hMSCs) without cellular toxicity. |
T7 |
827-835 |
Sentence |
denotes |
Results: |
T8 |
836-968 |
Sentence |
denotes |
The beneficial effects of AA2G and related molecular mechanisms were evaluated in mESCs, induced pluripotent-SCs (iPSCs), and hMSCs. |
T9 |
969-1071 |
Sentence |
denotes |
AA2G was stable in aqueous solution and barely induced cellular toxicity in cultured SCs, unlike VitC. |
T10 |
1072-1281 |
Sentence |
denotes |
AA2G supplementation recapitulated the well-known effects of VitC, including induction of ten-eleven translocation-dependent DNA demethylation in mESCs and suppression of p53 during generation of murine iPSCs. |
T11 |
1282-1464 |
Sentence |
denotes |
Furthermore, supplementation of hMSCs with AA2G improved therapeutic outcomes in an asthma mouse model by promoting their self-renewal, engraftment, and anti-inflammatory properties. |
T12 |
1465-1657 |
Sentence |
denotes |
Particularly, activation of the cAMP-responsive element-binding protein-1 (CREB1) pathway contributed to the ability of AA2G to maintain naive pluripotency of mESCs and functionality of hMSCs. |
T13 |
1658-1684 |
Sentence |
denotes |
Innovation and Conclusion: |
T14 |
1685-1912 |
Sentence |
denotes |
Given its long-lasting effects and low cellular toxicity, AA2G supplementation is useful to support the naive pluripotency of mESCs and the primitiveness of hMSCs, affecting their developmental potency and therapeutic efficacy. |
T15 |
1913-2014 |
Sentence |
denotes |
Furthermore, we demonstrate the significance of the CREB1 pathway in the mechanism of action of AA2G. |