PubMed:30445050 / 272-596 JSONTXT

{"project":"GoldHamster","denotations":[{"id":"T802","span":{"begin":28,"end":33},"obj":"C017082"},{"id":"T803","span":{"begin":28,"end":33},"obj":"C017082"},{"id":"T804","span":{"begin":28,"end":40},"obj":"D001156"},{"id":"T805","span":{"begin":34,"end":40},"obj":"892592"},{"id":"T807","span":{"begin":63,"end":77},"obj":"669"},{"id":"T808","span":{"begin":63,"end":77},"obj":"C000639619"},{"id":"T813","span":{"begin":140,"end":156},"obj":"267366"},{"id":"T810","span":{"begin":140,"end":156},"obj":"D001205"},{"id":"T811","span":{"begin":140,"end":156},"obj":"CHEBI:113451"},{"id":"T812","span":{"begin":140,"end":156},"obj":"D001205"},{"id":"T816","span":{"begin":165,"end":170},"obj":"PR:O23702"},{"id":"T819","span":{"begin":228,"end":245},"obj":"5499"},{"id":"T820","span":{"begin":228,"end":245},"obj":"D006861"},{"id":"T821","span":{"begin":228,"end":245},"obj":"CHEBI:16240"},{"id":"T822","span":{"begin":228,"end":245},"obj":"D006861"},{"id":"T824","span":{"begin":282,"end":287},"obj":"C017082"},{"id":"T825","span":{"begin":282,"end":287},"obj":"C017082"},{"id":"T826","span":{"begin":282,"end":294},"obj":"D001156"},{"id":"T827","span":{"begin":288,"end":294},"obj":"892592"},{"id":"T828","span":{"begin":314,"end":323},"obj":"D007239"},{"id":"T829","span":{"begin":314,"end":323},"obj":"D007239"}],"text":"In this study, using axenic brine shrimp and pathogenic strain Vibrio harveyi as the host-pathogen model, we confirmed that pretreatment of sodium ascorbate (NaAs), at an optimum concentration, was a prooxidant by generation of hydrogen peroxide, inducing protective effects in the brine shrimp against V. harveyi infection."}

{"project":"PubMed_ArguminSci","denotations":[{"id":"T2","span":{"begin":0,"end":324},"obj":"DRI_Background"}],"text":"In this study, using axenic brine shrimp and pathogenic strain Vibrio harveyi as the host-pathogen model, we confirmed that pretreatment of sodium ascorbate (NaAs), at an optimum concentration, was a prooxidant by generation of hydrogen peroxide, inducing protective effects in the brine shrimp against V. harveyi infection."}

{"project":"Goldhamster2_Cellosaurus","denotations":[{"id":"T7","span":{"begin":106,"end":108},"obj":"CVCL_5M23|Cancer_cell_line|Mesocricetus auratus"},{"id":"T8","span":{"begin":168,"end":170},"obj":"CVCL_8754|Cancer_cell_line|Homo sapiens"},{"id":"T9","span":{"begin":168,"end":170},"obj":"CVCL_H241|Cancer_cell_line|Homo sapiens"},{"id":"T10","span":{"begin":198,"end":199},"obj":"CVCL_6479|Finite_cell_line|Mus musculus"}],"text":"In this study, using axenic brine shrimp and pathogenic strain Vibrio harveyi as the host-pathogen model, we confirmed that pretreatment of sodium ascorbate (NaAs), at an optimum concentration, was a prooxidant by generation of hydrogen peroxide, inducing protective effects in the brine shrimp against V. harveyi infection."}