| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-99 |
Sentence |
denotes |
Loss of the homologous recombination gene rad51 leads to Fanconi anemia-like symptoms in zebrafish. |
| T2 |
100-208 |
Sentence |
denotes |
RAD51 is an indispensable homologous recombination protein, necessary for strand invasion and crossing over. |
| T3 |
209-349 |
Sentence |
denotes |
It has recently been designated as a Fanconi anemia (FA) gene, following the discovery of two patients carrying dominant-negative mutations. |
| T4 |
350-499 |
Sentence |
denotes |
FA is a hereditary DNA-repair disorder characterized by various congenital abnormalities, progressive bone marrow failure, and cancer predisposition. |
| T5 |
500-568 |
Sentence |
denotes |
In this report, we describe a viable vertebrate model of RAD51 loss. |
| T6 |
569-734 |
Sentence |
denotes |
Zebrafish rad51 loss-of-function mutants developed key features of FA, including hypocellular kidney marrow, sensitivity to cross-linking agents, and decreased size. |
| T7 |
735-887 |
Sentence |
denotes |
We show that some of these symptoms stem from both decreased proliferation and increased apoptosis of embryonic hematopoietic stem and progenitor cells. |
| T8 |
888-1008 |
Sentence |
denotes |
Comutation of p53 was able to rescue the hematopoietic defects seen in the single mutants, but led to tumor development. |
| T9 |
1009-1176 |
Sentence |
denotes |
We further demonstrate that prolonged inflammatory stress can exacerbate the hematological impairment, leading to an additional decrease in kidney marrow cell numbers. |
| T10 |
1177-1395 |
Sentence |
denotes |
These findings strengthen the assignment of RAD51 as a Fanconi gene and provide more evidence for the notion that aberrant p53 signaling during embryogenesis leads to the hematological defects seen later in life in FA. |
| T11 |
1396-1559 |
Sentence |
denotes |
Further research on this zebrafish FA model will lead to a deeper understanding of the molecular basis of bone marrow failure in FA and the cellular role of RAD51. |
| T1 |
0-99 |
Sentence |
denotes |
Loss of the homologous recombination gene rad51 leads to Fanconi anemia-like symptoms in zebrafish. |
| T2 |
100-208 |
Sentence |
denotes |
RAD51 is an indispensable homologous recombination protein, necessary for strand invasion and crossing over. |
| T3 |
209-349 |
Sentence |
denotes |
It has recently been designated as a Fanconi anemia (FA) gene, following the discovery of two patients carrying dominant-negative mutations. |
| T4 |
350-499 |
Sentence |
denotes |
FA is a hereditary DNA-repair disorder characterized by various congenital abnormalities, progressive bone marrow failure, and cancer predisposition. |
| T5 |
500-568 |
Sentence |
denotes |
In this report, we describe a viable vertebrate model of RAD51 loss. |
| T6 |
569-734 |
Sentence |
denotes |
Zebrafish rad51 loss-of-function mutants developed key features of FA, including hypocellular kidney marrow, sensitivity to cross-linking agents, and decreased size. |
| T7 |
735-887 |
Sentence |
denotes |
We show that some of these symptoms stem from both decreased proliferation and increased apoptosis of embryonic hematopoietic stem and progenitor cells. |
| T8 |
888-1008 |
Sentence |
denotes |
Comutation of p53 was able to rescue the hematopoietic defects seen in the single mutants, but led to tumor development. |
| T9 |
1009-1176 |
Sentence |
denotes |
We further demonstrate that prolonged inflammatory stress can exacerbate the hematological impairment, leading to an additional decrease in kidney marrow cell numbers. |
| T10 |
1177-1395 |
Sentence |
denotes |
These findings strengthen the assignment of RAD51 as a Fanconi gene and provide more evidence for the notion that aberrant p53 signaling during embryogenesis leads to the hematological defects seen later in life in FA. |
| T11 |
1396-1559 |
Sentence |
denotes |
Further research on this zebrafish FA model will lead to a deeper understanding of the molecular basis of bone marrow failure in FA and the cellular role of RAD51. |