PubMed:28098423 / 0-255 JSONTXT

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    PubMed_ArguminSci

    {"project":"PubMed_ArguminSci","denotations":[{"id":"T1","span":{"begin":128,"end":225},"obj":"DRI_Background"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}

    TEST-DiseaseOrPhenotypicFeature

    {"project":"TEST-DiseaseOrPhenotypicFeature","denotations":[{"id":"T1","span":{"begin":65,"end":84},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T2","span":{"begin":210,"end":224},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A1","pred":"#label","subj":"T1","obj":"DISEASE"},{"id":"A2","pred":"#label","subj":"T2","obj":"D066126"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}

    TEST-ChemicalEntity

    {"project":"TEST-ChemicalEntity","denotations":[{"id":"T1","span":{"begin":45,"end":56},"obj":"ChemicalEntity"},{"id":"T4","span":{"begin":156,"end":167},"obj":"ChemicalEntity"},{"id":"T7","span":{"begin":169,"end":172},"obj":"ChemicalEntity"}],"attributes":[{"id":"A1","pred":"ID:","subj":"T1","obj":"D004317"},{"id":"A2","pred":"ID:","subj":"T1","obj":"http://purl.obolibrary.org/obo/CHEBI_64816"},{"id":"A3","pred":"ID:","subj":"T1","obj":"http://purl.obolibrary.org/obo/CHEBI_28748"},{"id":"A4","pred":"ID:","subj":"T4","obj":"D004317"},{"id":"A5","pred":"ID:","subj":"T4","obj":"http://purl.obolibrary.org/obo/CHEBI_64816"},{"id":"A6","pred":"ID:","subj":"T4","obj":"http://purl.obolibrary.org/obo/CHEBI_28748"},{"id":"A7","pred":"ID:","subj":"T7","obj":"D004317"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}

    TEST-OrganismTaxon

    {"project":"TEST-OrganismTaxon","denotations":[{"id":"T1","span":{"begin":122,"end":126},"obj":"OrganismTaxon"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}

    Test-GeneOrGeneProduct

    {"project":"Test-GeneOrGeneProduct","denotations":[{"id":"T1","span":{"begin":0,"end":33},"obj":"GeneOrGeneProduct"},{"id":"T2","span":{"begin":104,"end":107},"obj":"GeneOrGeneProduct"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}

    Test-merged-2

    {"project":"Test-merged-2","denotations":[{"id":"T50828","span":{"begin":45,"end":56},"obj":"ChemicalEntity"},{"id":"T82753","span":{"begin":156,"end":167},"obj":"ChemicalEntity"},{"id":"T35177","span":{"begin":169,"end":172},"obj":"ChemicalEntity"},{"id":"T96953","span":{"begin":0,"end":33},"obj":"GeneOrGeneProduct"},{"id":"T55833","span":{"begin":104,"end":107},"obj":"GeneOrGeneProduct"},{"id":"T34613","span":{"begin":122,"end":126},"obj":"OrganismTaxon"},{"id":"T1","span":{"begin":65,"end":84},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T2","span":{"begin":210,"end":224},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A15247","pred":"ID:","subj":"T50828","obj":"D004317"},{"id":"A78813","pred":"ID:","subj":"T50828","obj":"http://purl.obolibrary.org/obo/CHEBI_64816"},{"id":"A61879","pred":"ID:","subj":"T50828","obj":"http://purl.obolibrary.org/obo/CHEBI_28748"},{"id":"A1030","pred":"ID:","subj":"T82753","obj":"D004317"},{"id":"A60382","pred":"ID:","subj":"T82753","obj":"http://purl.obolibrary.org/obo/CHEBI_64816"},{"id":"A9009","pred":"ID:","subj":"T82753","obj":"http://purl.obolibrary.org/obo/CHEBI_28748"},{"id":"A7","pred":"ID:","subj":"T35177","obj":"D004317"},{"id":"A1","pred":"#label","subj":"T1","obj":"DISEASE"},{"id":"A2","pred":"#label","subj":"T2","obj":"D066126"}],"text":"Brain-derived neurotrophic factor attenuates doxorubicin-induced cardiac dysfunction through activating Akt signalling in rats.\nThe clinical application of doxorubicin (Dox) is limited by its adverse effect of cardiotoxicity. Previous studies have suggest"}