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Inflammaging

Id	Subject	Object	Predicate	Lexical cue
T1	0-77	Sentence	denotes	Identifying immune mechanisms mediating the hypertension during preeclampsia.
T2	78-242	Sentence	denotes	Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality.
T3	243-442	Sentence	denotes	Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction.
T4	443-543	Sentence	denotes	However, the pathophysiological mechanisms that lead to the development of PE are poorly understood.
T5	544-757	Sentence	denotes	Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy.
T6	758-945	Sentence	denotes	Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE.
T7	946-1133	Sentence	denotes	Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review.
T8	1134-1309	Sentence	denotes	Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role.
T1	0-77	Sentence	denotes	Identifying immune mechanisms mediating the hypertension during preeclampsia.
T2	78-242	Sentence	denotes	Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality.
T3	243-442	Sentence	denotes	Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction.
T4	443-543	Sentence	denotes	However, the pathophysiological mechanisms that lead to the development of PE are poorly understood.
T5	544-757	Sentence	denotes	Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy.
T6	758-945	Sentence	denotes	Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE.
T7	946-1133	Sentence	denotes	Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review.
T8	1134-1309	Sentence	denotes	Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role.

Preeclampsia

Id	Subject	Object	Predicate	Lexical cue
PD-Preeclampsia-B_T1	64-76	ORPHA:275555	denotes	preeclampsia
PD-Preeclampsia-B_T2	78-90	ORPHA:275555	denotes	Preeclampsia

Preeclampsia-compare

Id	Subject	Object	Predicate	Lexical cue
PD-Preeclampsia-B_T1	64-76	ORPHA:275555	denotes	preeclampsia
PD-Preeclampsia-B_T2	78-90	ORPHA:275555	denotes	Preeclampsia

sentences

Id	Subject	Object	Predicate	Lexical cue
TextSentencer_T1	0-77	Sentence	denotes	Identifying immune mechanisms mediating the hypertension during preeclampsia.
TextSentencer_T2	78-242	Sentence	denotes	Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality.
TextSentencer_T3	243-442	Sentence	denotes	Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction.
TextSentencer_T4	443-543	Sentence	denotes	However, the pathophysiological mechanisms that lead to the development of PE are poorly understood.
TextSentencer_T5	544-757	Sentence	denotes	Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy.
TextSentencer_T6	758-945	Sentence	denotes	Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE.
TextSentencer_T7	946-1133	Sentence	denotes	Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review.
TextSentencer_T8	1134-1309	Sentence	denotes	Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role.
T1	0-77	Sentence	denotes	Identifying immune mechanisms mediating the hypertension during preeclampsia.
T2	78-242	Sentence	denotes	Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality.
T3	243-442	Sentence	denotes	Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction.
T4	443-543	Sentence	denotes	However, the pathophysiological mechanisms that lead to the development of PE are poorly understood.
T5	544-757	Sentence	denotes	Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy.
T6	758-945	Sentence	denotes	Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE.
T7	946-1133	Sentence	denotes	Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review.
T8	1134-1309	Sentence	denotes	Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role.

performance-test

Id	Subject	Object	Predicate	Lexical cue
PD-UBERON-AE-B_T1	1262-1271	http://purl.obolibrary.org/obo/UBERON_0001987	denotes	placental