| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-138 |
Sentence |
denotes |
The Mechanism by Which MYCN Amplification Confers an Enhanced Sensitivity to a PCNA-Derived Cell Permeable Peptide in Neuroblastoma Cells. |
| TextSentencer_T2 |
139-218 |
Sentence |
denotes |
Dysregulated expression of MYC family genes is a hallmark of many malignancies. |
| TextSentencer_T3 |
219-333 |
Sentence |
denotes |
Unfortunately, these proteins are not amenable to blockade by small molecules or protein-based therapeutic agents. |
| TextSentencer_T4 |
334-410 |
Sentence |
denotes |
Therefore, we must find alternative approaches to target MYC-driven cancers. |
| TextSentencer_T5 |
411-501 |
Sentence |
denotes |
Amplification of MYCN, a MYC family member, predicts high-risk neuroblastoma (NB) disease. |
| TextSentencer_T6 |
502-697 |
Sentence |
denotes |
We have shown that R9-caPep blocks the interaction of PCNA with its binding partners and selectively kills human NB cells, especially those with MYCN amplification, and we now show the mechanism. |
| TextSentencer_T7 |
698-776 |
Sentence |
denotes |
We found elevated levels of DNA replication stress in MYCN-amplified NB cells. |
| TextSentencer_T8 |
777-1009 |
Sentence |
denotes |
R9-caPep exacerbated DNA replication stress in MYCN-amplified NB cells and NB cells with an augmented level of MYC by interfering with DNA replication fork extension, leading to Chk1 dependence and susceptibility to Chk1 inhibition. |
| TextSentencer_T9 |
1010-1073 |
Sentence |
denotes |
We describe how these effects may be exploited for treating NB. |
| T1 |
0-138 |
Sentence |
denotes |
The Mechanism by Which MYCN Amplification Confers an Enhanced Sensitivity to a PCNA-Derived Cell Permeable Peptide in Neuroblastoma Cells. |
| T2 |
139-218 |
Sentence |
denotes |
Dysregulated expression of MYC family genes is a hallmark of many malignancies. |
| T3 |
219-333 |
Sentence |
denotes |
Unfortunately, these proteins are not amenable to blockade by small molecules or protein-based therapeutic agents. |
| T4 |
334-410 |
Sentence |
denotes |
Therefore, we must find alternative approaches to target MYC-driven cancers. |
| T5 |
411-501 |
Sentence |
denotes |
Amplification of MYCN, a MYC family member, predicts high-risk neuroblastoma (NB) disease. |
| T6 |
502-697 |
Sentence |
denotes |
We have shown that R9-caPep blocks the interaction of PCNA with its binding partners and selectively kills human NB cells, especially those with MYCN amplification, and we now show the mechanism. |
| T7 |
698-776 |
Sentence |
denotes |
We found elevated levels of DNA replication stress in MYCN-amplified NB cells. |
| T8 |
777-1009 |
Sentence |
denotes |
R9-caPep exacerbated DNA replication stress in MYCN-amplified NB cells and NB cells with an augmented level of MYC by interfering with DNA replication fork extension, leading to Chk1 dependence and susceptibility to Chk1 inhibition. |
| T9 |
1010-1073 |
Sentence |
denotes |
We describe how these effects may be exploited for treating NB. |