PubMed:26555588
Annnotations
GoldHamster
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 0-8 | 7456 | denotes | Nitrogen |
T5 | 0-16 | D008466 | denotes | Nitrogen Mustard |
T6 | 0-16 | D008466 | denotes | Nitrogen Mustard |
T7 | 0-16 | CHEBI:37598 | denotes | Nitrogen Mustard |
T8 | 0-16 | CHEBI:28925 | denotes | Nitrogen Mustard |
T9 | 9-16 | D009149 | denotes | Mustard |
T10 | 25-39 | D065306 | denotes | Corneal Injury |
T11 | 25-39 | D065306 | denotes | Corneal Injury |
T16 | 49-52 | SO:0000352 | denotes | DNA |
T15 | 49-52 | CHEBI:16991 | denotes | DNA |
T17 | 49-52 | D004247 | denotes | DNA |
T14 | 49-52 | GO:0005574 | denotes | DNA |
T18 | 84-96 | GO:0006954 | denotes | Inflammation |
T19 | 84-96 | D007249 | denotes | Inflammation |
T20 | 84-96 | D007249 | denotes | Inflammation |
T21 | 229-235 | UBERON:0000479 | denotes | tissue |
T22 | 269-277 | 7456 | denotes | nitrogen |
T25 | 269-285 | D008466 | denotes | nitrogen mustard |
T26 | 269-285 | D008466 | denotes | nitrogen mustard |
T27 | 269-285 | CHEBI:37598 | denotes | nitrogen mustard |
T28 | 269-285 | CHEBI:28925 | denotes | nitrogen mustard |
T29 | 278-285 | D009149 | denotes | mustard |
T30 | 332-354 | D002619 | denotes | chemical warfare agent |
T33 | 355-361 | 10223 | denotes | sulfur |
T36 | 355-369 | D009151 | denotes | sulfur mustard |
T37 | 355-369 | CHEBI:25434 | denotes | sulfur mustard |
T38 | 355-369 | D009151 | denotes | sulfur mustard |
T39 | 362-369 | D009149 | denotes | mustard |
T40 | 464-470 | UBERON:0000479 | denotes | tissue |
T41 | 498-503 | D006801 | denotes | human |
T43 | 524-527 | CVCL_1272 | denotes | HCE |
T49 | 524-527 | CVCL_M619 | denotes | HCE |
T46 | 524-527 | C007907 | denotes | HCE |
T48 | 524-527 | C007907 | denotes | HCE |
T42 | 524-527 | O55236 | denotes | HCE |
T44 | 524-527 | PR:000014142 | denotes | HCE |
T45 | 524-527 | O60942 | denotes | HCE |
T47 | 524-527 | P02242 | denotes | HCE |
T51 | 579-585 | 813859 | denotes | rabbit |
T50 | 579-585 | D011817 | denotes | rabbit |
T52 | 579-585 | 9986 | denotes | rabbit |
T53 | 586-593 | UBERON:0000964 | denotes | corneas |
T54 | 628-648 | GO:0006915 | denotes | apoptotic cell death |
T56 | 717-751 | D042461 | denotes | vascular endothelial growth factor |
T58 | 717-751 | D042461 | denotes | vascular endothelial growth factor |
T57 | 717-751 | GO:0005172 | denotes | vascular endothelial growth factor |
T61 | 738-751 | P08441 | denotes | growth factor |
T62 | 738-751 | P08072 | denotes | growth factor |
T63 | 738-751 | Q776B5 | denotes | growth factor |
T64 | 738-751 | Q6RZT5 | denotes | growth factor |
T65 | 738-751 | Q8V307 | denotes | growth factor |
T78 | 753-769 | D051546 | denotes | cyclooxygenase 2 |
T67 | 753-769 | O62725 | denotes | cyclooxygenase 2 |
T68 | 753-769 | Q05769 | denotes | cyclooxygenase 2 |
T69 | 753-769 | P35355 | denotes | cyclooxygenase 2 |
T70 | 753-769 | P79208 | denotes | cyclooxygenase 2 |
T71 | 753-769 | P35354 | denotes | cyclooxygenase 2 |
T72 | 753-769 | O02768 | denotes | cyclooxygenase 2 |
T73 | 753-769 | PR:000013428 | denotes | cyclooxygenase 2 |
T74 | 753-769 | P27607 | denotes | cyclooxygenase 2 |
T75 | 753-769 | O19183 | denotes | cyclooxygenase 2 |
T76 | 753-769 | O62698 | denotes | cyclooxygenase 2 |
T77 | 753-769 | P70682 | denotes | cyclooxygenase 2 |
T79 | 775-781 | UBERON:0027368 | denotes | matrix |
T80 | 775-801 | D020780 | denotes | matrix metalloproteinase-9 |
T81 | 775-801 | P14780 | denotes | matrix metalloproteinase-9 |
T82 | 775-801 | P41246 | denotes | matrix metalloproteinase-9 |
T83 | 775-801 | P41245 | denotes | matrix metalloproteinase-9 |
T84 | 775-801 | P50282 | denotes | matrix metalloproteinase-9 |
T85 | 775-801 | PR:000010491 | denotes | matrix metalloproteinase-9 |
T86 | 775-801 | P52176 | denotes | matrix metalloproteinase-9 |
T87 | 775-801 | O18733 | denotes | matrix metalloproteinase-9 |
T92 | 806-809 | C007907 | denotes | HCE |
T94 | 806-809 | C007907 | denotes | HCE |
T88 | 806-809 | O55236 | denotes | HCE |
T90 | 806-809 | PR:000014142 | denotes | HCE |
T91 | 806-809 | O60942 | denotes | HCE |
T93 | 806-809 | P02242 | denotes | HCE |
T89 | 806-809 | CVCL_1272 | denotes | HCE |
T95 | 806-809 | CVCL_M619 | denotes | HCE |
T96 | 889-902 | UBERON:2000098 | denotes | proliferation |
T99 | 930-933 | SO:0000352 | denotes | DNA |
T98 | 930-933 | CHEBI:16991 | denotes | DNA |
T100 | 930-933 | D004247 | denotes | DNA |
T97 | 930-933 | GO:0005574 | denotes | DNA |
T101 | 968-971 | PR:P04637 | denotes | p53 |
T102 | 968-971 | PR:000003035 | denotes | p53 |
T103 | 968-971 | PR:P02340 | denotes | p53 |
T104 | 968-971 | PR:Q42578 | denotes | p53 |
T105 | 968-971 | PR:P10361 | denotes | p53 |
T112 | 972-975 | SO:0001444 | denotes | ser |
T106 | 972-975 | CHEBI:32838 | denotes | ser |
T107 | 972-975 | CHEBI:32839 | denotes | ser |
T109 | 972-975 | CHEBI:29999 | denotes | ser |
T113 | 972-975 | CHEBI:17115 | denotes | ser |
T110 | 972-975 | Q62230 | denotes | ser |
T111 | 972-975 | PR:000001931 | denotes | ser |
T114 | 972-975 | PR:P18168 | denotes | ser |
T108 | 972-975 | GO:0005790 | denotes | ser |
T115 | 985-988 | PR:P04637 | denotes | p53 |
T116 | 985-988 | PR:000003035 | denotes | p53 |
T117 | 985-988 | PR:P02340 | denotes | p53 |
T118 | 985-988 | PR:Q42578 | denotes | p53 |
T119 | 985-988 | PR:P10361 | denotes | p53 |
T121 | 994-999 | P27661 | denotes | H2A.X |
T122 | 994-999 | PR:P27661 | denotes | H2A.X |
T123 | 994-999 | Q7ZUY3 | denotes | H2A.X |
T124 | 994-999 | Q54WG6 | denotes | H2A.X |
T125 | 994-999 | PR:000008418 | denotes | H2A.X |
T126 | 994-999 | Q6GM86 | denotes | H2A.X |
T127 | 994-999 | P16104 | denotes | H2A.X |
T134 | 1000-1003 | SO:0001444 | denotes | ser |
T128 | 1000-1003 | CHEBI:32838 | denotes | ser |
T129 | 1000-1003 | CHEBI:32839 | denotes | ser |
T131 | 1000-1003 | CHEBI:29999 | denotes | ser |
T135 | 1000-1003 | CHEBI:17115 | denotes | ser |
T132 | 1000-1003 | Q62230 | denotes | ser |
T133 | 1000-1003 | PR:000001931 | denotes | ser |
T136 | 1000-1003 | PR:P18168 | denotes | ser |
T130 | 1000-1003 | GO:0005790 | denotes | ser |
T139 | 1040-1049 | P55866 | denotes | caspase-3 |
T140 | 1040-1049 | Q8MJC3 | denotes | caspase-3 |
T141 | 1040-1049 | Q2PFV2 | denotes | caspase-3 |
T142 | 1040-1049 | Q08DY9 | denotes | caspase-3 |
T143 | 1040-1049 | Q5IS54 | denotes | caspase-3 |
T144 | 1040-1049 | Q8MKI5 | denotes | caspase-3 |
T145 | 1040-1049 | PR:000002312 | denotes | caspase-3 |
T146 | 1040-1049 | Q8MJU1 | denotes | caspase-3 |
T147 | 1040-1049 | Q60431 | denotes | caspase-3 |
T148 | 1040-1049 | P42574 | denotes | caspase-3 |
T149 | 1040-1049 | Q5IS99 | denotes | caspase-3 |
T150 | 1040-1049 | Q95ND5 | denotes | caspase-3 |
T151 | 1040-1049 | P70677 | denotes | caspase-3 |
T152 | 1040-1049 | P55213 | denotes | caspase-3 |
T138 | 1040-1049 | D053148 | denotes | caspase-3 |
T153 | 1054-1058 | CL:0000775 | denotes | poly |
T154 | 1054-1058 | CL:0000096 | denotes | poly |
T158 | 1054-1080 | P35875 | denotes | poly ADP ribose polymerase |
T159 | 1054-1080 | Q11208 | denotes | poly ADP ribose polymerase |
T160 | 1054-1080 | Q9VBP3 | denotes | poly ADP ribose polymerase |
T157 | 1054-1080 | D011065 | denotes | poly ADP ribose polymerase |
T178 | 1157-1163 | 813859 | denotes | rabbit |
T177 | 1157-1163 | D011817 | denotes | rabbit |
T179 | 1157-1163 | 9986 | denotes | rabbit |
T180 | 1164-1170 | UBERON:0000964 | denotes | cornea |
T181 | 1175-1178 | O55236 | denotes | HCE |
T183 | 1175-1178 | PR:000014142 | denotes | HCE |
T184 | 1175-1178 | O60942 | denotes | HCE |
T186 | 1175-1178 | P02242 | denotes | HCE |
T185 | 1175-1178 | C007907 | denotes | HCE |
T187 | 1175-1178 | C007907 | denotes | HCE |
T182 | 1175-1178 | CVCL_1272 | denotes | HCE |
T188 | 1175-1178 | CVCL_M619 | denotes | HCE |
T190 | 1197-1203 | 813859 | denotes | rabbit |
T189 | 1197-1203 | D011817 | denotes | rabbit |
T191 | 1197-1203 | 9986 | denotes | rabbit |
T192 | 1204-1210 | UBERON:0000964 | denotes | cornea |
T194 | 1246-1262 | O62725 | denotes | cyclooxygenase 2 |
T195 | 1246-1262 | Q05769 | denotes | cyclooxygenase 2 |
T196 | 1246-1262 | P35355 | denotes | cyclooxygenase 2 |
T197 | 1246-1262 | P79208 | denotes | cyclooxygenase 2 |
T198 | 1246-1262 | P35354 | denotes | cyclooxygenase 2 |
T199 | 1246-1262 | O02768 | denotes | cyclooxygenase 2 |
T200 | 1246-1262 | PR:000013428 | denotes | cyclooxygenase 2 |
T201 | 1246-1262 | P27607 | denotes | cyclooxygenase 2 |
T202 | 1246-1262 | O19183 | denotes | cyclooxygenase 2 |
T203 | 1246-1262 | O62698 | denotes | cyclooxygenase 2 |
T204 | 1246-1262 | P70682 | denotes | cyclooxygenase 2 |
T205 | 1246-1262 | D051546 | denotes | cyclooxygenase 2 |
T206 | 1264-1270 | UBERON:0027368 | denotes | matrix |
T208 | 1264-1290 | P14780 | denotes | matrix metalloproteinase-9 |
T209 | 1264-1290 | P41246 | denotes | matrix metalloproteinase-9 |
T210 | 1264-1290 | P41245 | denotes | matrix metalloproteinase-9 |
T211 | 1264-1290 | P50282 | denotes | matrix metalloproteinase-9 |
T212 | 1264-1290 | PR:000010491 | denotes | matrix metalloproteinase-9 |
T213 | 1264-1290 | P52176 | denotes | matrix metalloproteinase-9 |
T214 | 1264-1290 | O18733 | denotes | matrix metalloproteinase-9 |
T207 | 1264-1290 | D020780 | denotes | matrix metalloproteinase-9 |
T215 | 1296-1330 | D042461 | denotes | vascular endothelial growth factor |
T217 | 1296-1330 | D042461 | denotes | vascular endothelial growth factor |
T216 | 1296-1330 | GO:0005172 | denotes | vascular endothelial growth factor |
T220 | 1317-1330 | P08441 | denotes | growth factor |
T221 | 1317-1330 | P08072 | denotes | growth factor |
T222 | 1317-1330 | Q776B5 | denotes | growth factor |
T223 | 1317-1330 | Q6RZT5 | denotes | growth factor |
T224 | 1317-1330 | Q8V307 | denotes | growth factor |
T226 | 1341-1344 | CVCL_1272 | denotes | HCE |
T232 | 1341-1344 | CVCL_M619 | denotes | HCE |
T225 | 1341-1344 | O55236 | denotes | HCE |
T227 | 1341-1344 | PR:000014142 | denotes | HCE |
T228 | 1341-1344 | O60942 | denotes | HCE |
T230 | 1341-1344 | P02242 | denotes | HCE |
T229 | 1341-1344 | C007907 | denotes | HCE |
T231 | 1341-1344 | C007907 | denotes | HCE |
T233 | 1379-1388 | CHEBI:25367 | denotes | molecules |
T234 | 1432-1444 | GO:0006954 | denotes | inflammation |
T235 | 1432-1444 | D007249 | denotes | inflammation |
T236 | 1432-1444 | D007249 | denotes | inflammation |
T237 | 1540-1559 | P56432 | denotes | activator protein 1 |
T238 | 1540-1559 | P05627 | denotes | activator protein 1 |
T239 | 1540-1559 | O77627 | denotes | activator protein 1 |
T240 | 1540-1559 | P05412 | denotes | activator protein 1 |
T241 | 1540-1559 | P17325 | denotes | activator protein 1 |
T242 | 1540-1559 | PR:000009232 | denotes | activator protein 1 |
T243 | 1540-1559 | D018808 | denotes | activator protein 1 |
T248 | 1550-1557 | SO:0000104 | denotes | protein |
T244 | 1550-1557 | GO:0003675 | denotes | protein |
T250 | 1560-1573 | GO:0006351 | denotes | transcription |
T251 | 1560-1580 | GO:0000981 | denotes | transcription factor |
T252 | 1581-1589 | CHEBI:36080 | denotes | proteins |
T253 | 1594-1602 | SO:0001635 | denotes | upstream |
T254 | 1603-1612 | GO:0023052 | denotes | signaling |
T256 | 1632-1665 | D020928 | denotes | mitogen-activated protein kinases |
T261 | 1650-1657 | SO:0000104 | denotes | protein |
T260 | 1650-1657 | PR:000000001 | denotes | protein |
T257 | 1650-1657 | GO:0003675 | denotes | protein |
T263 | 1670-1673 | PR:000029189 | denotes | Akt |
T264 | 1670-1673 | PR:P54644 | denotes | Akt |
T265 | 1670-1673 | PR:P31750 | denotes | Akt |
T266 | 1670-1673 | PR:000002190 | denotes | Akt |
T267 | 1670-1673 | Q8INB9 | denotes | Akt |
T272 | 1674-1681 | SO:0000104 | denotes | protein |
T271 | 1674-1681 | PR:000000001 | denotes | protein |
T268 | 1674-1681 | GO:0003675 | denotes | protein |
T273 | 1674-1688 | D011494 | denotes | protein kinase |
T274 | 1756-1770 | D065306 | denotes | corneal injury |
T275 | 1756-1770 | D065306 | denotes | corneal injury |
T276 | 1902-1918 | D065306 | denotes | corneal injuries |
T277 | 1902-1918 | D065306 | denotes | corneal injuries |
T278 | 1988-1996 | CHEBI:78592 | denotes | vesicant |
PubMed_Structured_Abstracts
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 162-370 | OBJECTIVE | denotes | To evaluate the toxic effects and associated mechanisms in corneal tissue exposed to the vesicating agent, nitrogen mustard (NM), a bifunctional alkylating analog of the chemical warfare agent sulfur mustard. |
T2 | 380-549 | METHODS | denotes | Toxic effects and associated mechanisms were examined in maximally affected corneal tissue using corneal cultures and human corneal epithelial (HCE) cells exposed to NM. |
T3 | 559-1771 | RESULTS | denotes | Analysis of ex vivo rabbit corneas showed that NM exposure increased apoptotic cell death, epithelial thickness, epithelial-stromal separation, and levels of vascular endothelial growth factor, cyclooxygenase 2, and matrix metalloproteinase-9. In HCE cells, NM exposure resulted in a dose-dependent decrease in cell viability and proliferation, which was associated with DNA damage in terms of an increase in p53 ser15, total p53, and H2A.X ser139 levels. NM exposure also induced caspase-3 and poly ADP ribose polymerase cleavage, suggesting their involvement in NM-induced apoptotic death in the rabbit cornea and HCE cells. Similar to rabbit cornea, NM exposure caused an increase in cyclooxygenase 2, matrix metalloproteinase-9, and vascular endothelial growth factor levels in HCE cells, indicating a role of these molecules and related pathways in NM-induced corneal inflammation, epithelial-stromal separation, and neovascularization. NM exposure also induced activation of activator protein 1 transcription factor proteins and upstream signaling pathways including mitogen-activated protein kinases and Akt protein kinase, suggesting that these could be key factors involved in NM-induced corneal injury. |
T4 | 1785-2086 | CONCLUSIONS | denotes | Results from this study provide insight into the molecular targets and pathways that could be involved in NM-induced corneal injuries laying the background for further investigation of these pathways in vesicant-induced ocular injuries, which could be helpful in the development of targeted therapies. |
Goldhamster2_Cellosaurus
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 292-293 | CVCL_6479|Finite_cell_line|Mus musculus | denotes | a |
T2 | 524-527 | CVCL_1272|Transformed_cell_line|Homo sapiens | denotes | HCE |
T3 | 524-527 | CVCL_M619|Cancer_cell_line|Homo sapiens | denotes | HCE |
T4 | 806-809 | CVCL_1272|Transformed_cell_line|Homo sapiens | denotes | HCE |
T5 | 806-809 | CVCL_M619|Cancer_cell_line|Homo sapiens | denotes | HCE |
T6 | 841-842 | CVCL_6479|Finite_cell_line|Mus musculus | denotes | a |
T7 | 953-955 | CVCL_8754|Cancer_cell_line|Homo sapiens | denotes | an |
T8 | 953-955 | CVCL_H241|Cancer_cell_line|Homo sapiens | denotes | an |
T9 | 1175-1178 | CVCL_1272|Transformed_cell_line|Homo sapiens | denotes | HCE |
T10 | 1175-1178 | CVCL_M619|Cancer_cell_line|Homo sapiens | denotes | HCE |
T11 | 1231-1233 | CVCL_8754|Cancer_cell_line|Homo sapiens | denotes | an |
T12 | 1231-1233 | CVCL_H241|Cancer_cell_line|Homo sapiens | denotes | an |
T13 | 1341-1344 | CVCL_1272|Transformed_cell_line|Homo sapiens | denotes | HCE |
T14 | 1341-1344 | CVCL_M619|Cancer_cell_line|Homo sapiens | denotes | HCE |
T15 | 1363-1364 | CVCL_6479|Finite_cell_line|Mus musculus | denotes | a |
T16 | 1526-1536 | CVCL_C410|Hybridoma|Mus musculus | denotes | activation |
T17 | 1540-1549 | CVCL_C410|Hybridoma|Mus musculus | denotes | activator |
T18 | 1640-1649 | CVCL_C410|Hybridoma|Mus musculus | denotes | activated |
Inflammaging
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 0-152 | Sentence | denotes | Nitrogen Mustard-Induced Corneal Injury Involves DNA Damage and Pathways Related to Inflammation, Epithelial-Stromal Separation, and Neovascularization. |
T2 | 153-161 | Sentence | denotes | PURPOSE: |
T3 | 162-370 | Sentence | denotes | To evaluate the toxic effects and associated mechanisms in corneal tissue exposed to the vesicating agent, nitrogen mustard (NM), a bifunctional alkylating analog of the chemical warfare agent sulfur mustard. |
T4 | 371-379 | Sentence | denotes | METHODS: |
T5 | 380-549 | Sentence | denotes | Toxic effects and associated mechanisms were examined in maximally affected corneal tissue using corneal cultures and human corneal epithelial (HCE) cells exposed to NM. |
T6 | 550-558 | Sentence | denotes | RESULTS: |
T7 | 559-802 | Sentence | denotes | Analysis of ex vivo rabbit corneas showed that NM exposure increased apoptotic cell death, epithelial thickness, epithelial-stromal separation, and levels of vascular endothelial growth factor, cyclooxygenase 2, and matrix metalloproteinase-9. |
T8 | 803-1014 | Sentence | denotes | In HCE cells, NM exposure resulted in a dose-dependent decrease in cell viability and proliferation, which was associated with DNA damage in terms of an increase in p53 ser15, total p53, and H2A.X ser139 levels. |
T9 | 1015-1185 | Sentence | denotes | NM exposure also induced caspase-3 and poly ADP ribose polymerase cleavage, suggesting their involvement in NM-induced apoptotic death in the rabbit cornea and HCE cells. |
T10 | 1186-1500 | Sentence | denotes | Similar to rabbit cornea, NM exposure caused an increase in cyclooxygenase 2, matrix metalloproteinase-9, and vascular endothelial growth factor levels in HCE cells, indicating a role of these molecules and related pathways in NM-induced corneal inflammation, epithelial-stromal separation, and neovascularization. |
T11 | 1501-1771 | Sentence | denotes | NM exposure also induced activation of activator protein 1 transcription factor proteins and upstream signaling pathways including mitogen-activated protein kinases and Akt protein kinase, suggesting that these could be key factors involved in NM-induced corneal injury. |
T12 | 1772-1784 | Sentence | denotes | CONCLUSIONS: |
T13 | 1785-2086 | Sentence | denotes | Results from this study provide insight into the molecular targets and pathways that could be involved in NM-induced corneal injuries laying the background for further investigation of these pathways in vesicant-induced ocular injuries, which could be helpful in the development of targeted therapies. |
T1 | 0-152 | Sentence | denotes | Nitrogen Mustard-Induced Corneal Injury Involves DNA Damage and Pathways Related to Inflammation, Epithelial-Stromal Separation, and Neovascularization. |
T2 | 153-161 | Sentence | denotes | PURPOSE: |
T3 | 162-370 | Sentence | denotes | To evaluate the toxic effects and associated mechanisms in corneal tissue exposed to the vesicating agent, nitrogen mustard (NM), a bifunctional alkylating analog of the chemical warfare agent sulfur mustard. |
T4 | 371-379 | Sentence | denotes | METHODS: |
T5 | 380-549 | Sentence | denotes | Toxic effects and associated mechanisms were examined in maximally affected corneal tissue using corneal cultures and human corneal epithelial (HCE) cells exposed to NM. |
T6 | 550-558 | Sentence | denotes | RESULTS: |
T7 | 559-802 | Sentence | denotes | Analysis of ex vivo rabbit corneas showed that NM exposure increased apoptotic cell death, epithelial thickness, epithelial-stromal separation, and levels of vascular endothelial growth factor, cyclooxygenase 2, and matrix metalloproteinase-9. |
T8 | 803-1014 | Sentence | denotes | In HCE cells, NM exposure resulted in a dose-dependent decrease in cell viability and proliferation, which was associated with DNA damage in terms of an increase in p53 ser15, total p53, and H2A.X ser139 levels. |
T9 | 1015-1185 | Sentence | denotes | NM exposure also induced caspase-3 and poly ADP ribose polymerase cleavage, suggesting their involvement in NM-induced apoptotic death in the rabbit cornea and HCE cells. |
T10 | 1186-1500 | Sentence | denotes | Similar to rabbit cornea, NM exposure caused an increase in cyclooxygenase 2, matrix metalloproteinase-9, and vascular endothelial growth factor levels in HCE cells, indicating a role of these molecules and related pathways in NM-induced corneal inflammation, epithelial-stromal separation, and neovascularization. |
T11 | 1501-1771 | Sentence | denotes | NM exposure also induced activation of activator protein 1 transcription factor proteins and upstream signaling pathways including mitogen-activated protein kinases and Akt protein kinase, suggesting that these could be key factors involved in NM-induced corneal injury. |
T12 | 1772-1784 | Sentence | denotes | CONCLUSIONS: |
T13 | 1785-2086 | Sentence | denotes | Results from this study provide insight into the molecular targets and pathways that could be involved in NM-induced corneal injuries laying the background for further investigation of these pathways in vesicant-induced ocular injuries, which could be helpful in the development of targeted therapies. |