PubMed:26515425
Annnotations
Inflammaging
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 0-111 | Sentence | denotes | Amniotic mesenchymal cells from pre-eclamptic placentae maintain immunomodulatory features as healthy controls. |
T2 | 112-249 | Sentence | denotes | Pre-eclampsia (PE) is one of the most severe syndromes in human pregnancy, and the underlying mechanisms of PE have yet to be determined. |
T3 | 250-434 | Sentence | denotes | Pre-eclampsia is characterized by the alteration of the immune system's activation status, an increase in inflammatory Th1/Th17/APC cells, and a decrease in Th2/Treg subsets/cytokines. |
T4 | 435-639 | Sentence | denotes | Moreover, inflammatory infiltrates have been detected in the amniotic membranes of pre-eclamptic placentae, and to this date limited data are available regarding the role of amniotic membrane cells in PE. |
T5 | 640-844 | Sentence | denotes | Interestingly, we and others have previously shown that human amniotic mesenchymal stromal cells (hAMSC) possess anti-inflammatory properties towards almost all immune cells described to be altered in PE. |
T6 | 845-943 | Sentence | denotes | In this study we investigated whether the immunomodulatory properties of hAMSC were altered in PE. |
T7 | 944-1174 | Sentence | denotes | We performed a comprehensive study of cell phenotype and investigated the in vitro immunomodulatory properties of hAMSC isolated from pre-eclamptic pregnancies (PE-hAMSC), comparing them to hAMSC from normal pregnancies (N-hAMSC). |
T8 | 1175-1366 | Sentence | denotes | We demonstrate that PE-hAMSC inhibit CD4/CD8 T-cell proliferation, suppress Th1/Th2/Th17 polarization, induce Treg and block dendritic cells and M1 differentiation switching them to M2 cells. |
T9 | 1367-1610 | Sentence | denotes | Notably, PE-hAMSC generated a more prominent induction of Treg and higher suppression of interferon-γ when compared to N-hAMSC, and this was associated with higher transforming growth factor-β1 secretion and PD-L2/PD-L1 expression in PE-hAMSC. |
T10 | 1611-1743 | Sentence | denotes | In conclusion, for the first time we demonstrate that there is no intrinsic impairment of the immunomodulatory features of PE-hAMSC. |
T11 | 1744-1938 | Sentence | denotes | Our results suggest that amniotic mesenchymal stromal cells do not contribute to the disease, but conversely, could participate in offsetting the inflammatory environment which characterizes PE. |
T1 | 0-111 | Sentence | denotes | Amniotic mesenchymal cells from pre-eclamptic placentae maintain immunomodulatory features as healthy controls. |
T2 | 112-249 | Sentence | denotes | Pre-eclampsia (PE) is one of the most severe syndromes in human pregnancy, and the underlying mechanisms of PE have yet to be determined. |
T3 | 250-434 | Sentence | denotes | Pre-eclampsia is characterized by the alteration of the immune system's activation status, an increase in inflammatory Th1/Th17/APC cells, and a decrease in Th2/Treg subsets/cytokines. |
T4 | 435-639 | Sentence | denotes | Moreover, inflammatory infiltrates have been detected in the amniotic membranes of pre-eclamptic placentae, and to this date limited data are available regarding the role of amniotic membrane cells in PE. |
T5 | 640-844 | Sentence | denotes | Interestingly, we and others have previously shown that human amniotic mesenchymal stromal cells (hAMSC) possess anti-inflammatory properties towards almost all immune cells described to be altered in PE. |
T6 | 845-943 | Sentence | denotes | In this study we investigated whether the immunomodulatory properties of hAMSC were altered in PE. |
T7 | 944-1174 | Sentence | denotes | We performed a comprehensive study of cell phenotype and investigated the in vitro immunomodulatory properties of hAMSC isolated from pre-eclamptic pregnancies (PE-hAMSC), comparing them to hAMSC from normal pregnancies (N-hAMSC). |
T8 | 1175-1366 | Sentence | denotes | We demonstrate that PE-hAMSC inhibit CD4/CD8 T-cell proliferation, suppress Th1/Th2/Th17 polarization, induce Treg and block dendritic cells and M1 differentiation switching them to M2 cells. |
T9 | 1367-1610 | Sentence | denotes | Notably, PE-hAMSC generated a more prominent induction of Treg and higher suppression of interferon-γ when compared to N-hAMSC, and this was associated with higher transforming growth factor-β1 secretion and PD-L2/PD-L1 expression in PE-hAMSC. |
T10 | 1611-1743 | Sentence | denotes | In conclusion, for the first time we demonstrate that there is no intrinsic impairment of the immunomodulatory features of PE-hAMSC. |
T11 | 1744-1938 | Sentence | denotes | Our results suggest that amniotic mesenchymal stromal cells do not contribute to the disease, but conversely, could participate in offsetting the inflammatory environment which characterizes PE. |
sentences
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
TextSentencer_T1 | 0-111 | Sentence | denotes | Amniotic mesenchymal cells from pre-eclamptic placentae maintain immunomodulatory features as healthy controls. |
TextSentencer_T2 | 112-249 | Sentence | denotes | Pre-eclampsia (PE) is one of the most severe syndromes in human pregnancy, and the underlying mechanisms of PE have yet to be determined. |
TextSentencer_T3 | 250-434 | Sentence | denotes | Pre-eclampsia is characterized by the alteration of the immune system's activation status, an increase in inflammatory Th1/Th17/APC cells, and a decrease in Th2/Treg subsets/cytokines. |
TextSentencer_T4 | 435-639 | Sentence | denotes | Moreover, inflammatory infiltrates have been detected in the amniotic membranes of pre-eclamptic placentae, and to this date limited data are available regarding the role of amniotic membrane cells in PE. |
TextSentencer_T5 | 640-844 | Sentence | denotes | Interestingly, we and others have previously shown that human amniotic mesenchymal stromal cells (hAMSC) possess anti-inflammatory properties towards almost all immune cells described to be altered in PE. |
TextSentencer_T6 | 845-943 | Sentence | denotes | In this study we investigated whether the immunomodulatory properties of hAMSC were altered in PE. |
TextSentencer_T7 | 944-1174 | Sentence | denotes | We performed a comprehensive study of cell phenotype and investigated the in vitro immunomodulatory properties of hAMSC isolated from pre-eclamptic pregnancies (PE-hAMSC), comparing them to hAMSC from normal pregnancies (N-hAMSC). |
TextSentencer_T8 | 1175-1366 | Sentence | denotes | We demonstrate that PE-hAMSC inhibit CD4/CD8 T-cell proliferation, suppress Th1/Th2/Th17 polarization, induce Treg and block dendritic cells and M1 differentiation switching them to M2 cells. |
TextSentencer_T9 | 1367-1610 | Sentence | denotes | Notably, PE-hAMSC generated a more prominent induction of Treg and higher suppression of interferon-γ when compared to N-hAMSC, and this was associated with higher transforming growth factor-β1 secretion and PD-L2/PD-L1 expression in PE-hAMSC. |
TextSentencer_T10 | 1611-1743 | Sentence | denotes | In conclusion, for the first time we demonstrate that there is no intrinsic impairment of the immunomodulatory features of PE-hAMSC. |
TextSentencer_T11 | 1744-1938 | Sentence | denotes | Our results suggest that amniotic mesenchymal stromal cells do not contribute to the disease, but conversely, could participate in offsetting the inflammatory environment which characterizes PE. |
T1 | 0-111 | Sentence | denotes | Amniotic mesenchymal cells from pre-eclamptic placentae maintain immunomodulatory features as healthy controls. |
T2 | 112-249 | Sentence | denotes | Pre-eclampsia (PE) is one of the most severe syndromes in human pregnancy, and the underlying mechanisms of PE have yet to be determined. |
T3 | 250-434 | Sentence | denotes | Pre-eclampsia is characterized by the alteration of the immune system's activation status, an increase in inflammatory Th1/Th17/APC cells, and a decrease in Th2/Treg subsets/cytokines. |
T4 | 435-639 | Sentence | denotes | Moreover, inflammatory infiltrates have been detected in the amniotic membranes of pre-eclamptic placentae, and to this date limited data are available regarding the role of amniotic membrane cells in PE. |
T5 | 640-844 | Sentence | denotes | Interestingly, we and others have previously shown that human amniotic mesenchymal stromal cells (hAMSC) possess anti-inflammatory properties towards almost all immune cells described to be altered in PE. |
T6 | 845-943 | Sentence | denotes | In this study we investigated whether the immunomodulatory properties of hAMSC were altered in PE. |
T7 | 944-1174 | Sentence | denotes | We performed a comprehensive study of cell phenotype and investigated the in vitro immunomodulatory properties of hAMSC isolated from pre-eclamptic pregnancies (PE-hAMSC), comparing them to hAMSC from normal pregnancies (N-hAMSC). |
T8 | 1175-1366 | Sentence | denotes | We demonstrate that PE-hAMSC inhibit CD4/CD8 T-cell proliferation, suppress Th1/Th2/Th17 polarization, induce Treg and block dendritic cells and M1 differentiation switching them to M2 cells. |
T9 | 1367-1610 | Sentence | denotes | Notably, PE-hAMSC generated a more prominent induction of Treg and higher suppression of interferon-γ when compared to N-hAMSC, and this was associated with higher transforming growth factor-β1 secretion and PD-L2/PD-L1 expression in PE-hAMSC. |
T10 | 1611-1743 | Sentence | denotes | In conclusion, for the first time we demonstrate that there is no intrinsic impairment of the immunomodulatory features of PE-hAMSC. |
T11 | 1744-1938 | Sentence | denotes | Our results suggest that amniotic mesenchymal stromal cells do not contribute to the disease, but conversely, could participate in offsetting the inflammatory environment which characterizes PE. |
Preeclampsia
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
PD-Preeclampsia-B_T1 | 112-125 | ORPHA:275555 | denotes | Pre-eclampsia |
PD-Preeclampsia-B_T2 | 250-263 | ORPHA:275555 | denotes | Pre-eclampsia |
Preeclampsia-compare
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
PD-Preeclampsia-B_T1 | 32-45 | ORPHA:275555 | denotes | pre-eclamptic |
PD-Preeclampsia-B_T2 | 112-125 | ORPHA:275555 | denotes | Pre-eclampsia |
PD-Preeclampsia-B_T3 | 250-263 | ORPHA:275555 | denotes | Pre-eclampsia |
PD-Preeclampsia-B_T4 | 518-531 | ORPHA:275555 | denotes | pre-eclamptic |
PD-Preeclampsia-B_T5 | 1078-1091 | ORPHA:275555 | denotes | pre-eclamptic |
UBERON-AE
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
PD-UBERON-AE-B_T1 | 9-20 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T2 | 711-722 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T3 | 1778-1789 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T4 | 46-55 | http://purl.obolibrary.org/obo/UBERON_0001987 | denotes | placentae |
PD-UBERON-AE-B_T5 | 532-541 | http://purl.obolibrary.org/obo/UBERON_0001987 | denotes | placentae |
PD-UBERON-AE-B_T6 | 306-321 | http://purl.obolibrary.org/obo/UBERON_0002405 | denotes | immune system's |
performance-test
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
PD-UBERON-AE-B_T1 | 46-55 | http://purl.obolibrary.org/obo/UBERON_0001987 | denotes | placentae |
PD-UBERON-AE-B_T2 | 532-541 | http://purl.obolibrary.org/obo/UBERON_0001987 | denotes | placentae |
PD-UBERON-AE-B_T3 | 9-20 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T4 | 711-722 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T5 | 1778-1789 | http://purl.obolibrary.org/obo/UBERON_0003104 | denotes | mesenchymal |
PD-UBERON-AE-B_T6 | 306-321 | http://purl.obolibrary.org/obo/UBERON_0002405 | denotes | immune system's |