PubMed:26476017 JSON TXT

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pqqtest_sentence

21k_plant_trait_mention

OryzaGP_2021_v2

OryzaGP_2021_FLAIR

{"project":"OryzaGP_2021_FLAIR","denotations":[{"id":"M_0","span":{"begin":37,"end":58},"obj":"hunflair:NA:Gene"},{"id":"M_1","span":{"begin":422,"end":443},"obj":"hunflair:NA:Gene"},{"id":"M_2","span":{"begin":337,"end":341},"obj":"hunflair:NA:Gene"},{"id":"M_3","span":{"begin":0,"end":4},"obj":"hunflair:NA:Species"},{"id":"M_4","span":{"begin":97,"end":101},"obj":"hunflair:NA:Species"},{"id":"M_5","span":{"begin":557,"end":561},"obj":"hunflair:NA:Species"},{"id":"M_6","span":{"begin":70,"end":78},"obj":"hunflair:NA:Chemical"},{"id":"M_7","span":{"begin":138,"end":146},"obj":"hunflair:NA:Chemical"},{"id":"M_8","span":{"begin":148,"end":150},"obj":"hunflair:NA:Chemical"},{"id":"M_9","span":{"begin":226,"end":228},"obj":"hunflair:NA:Chemical"},{"id":"M_10","span":{"begin":553,"end":555},"obj":"hunflair:NA:Chemical"},{"id":"M_11","span":{"begin":617,"end":619},"obj":"hunflair:NA:Chemical"},{"id":"M_12","span":{"begin":691,"end":693},"obj":"hunflair:NA:Chemical"},{"id":"M_13","span":{"begin":755,"end":757},"obj":"hunflair:NA:Chemical"},{"id":"M_14","span":{"begin":1026,"end":1028},"obj":"hunflair:NA:Chemical"},{"id":"M_15","span":{"begin":1377,"end":1379},"obj":"hunflair:NA:Chemical"},{"id":"M_16","span":{"begin":796,"end":800},"obj":"hunflair:NA:Species"},{"id":"M_17","span":{"begin":14,"end":18},"obj":"hunflair:NA:Gene"},{"id":"M_18","span":{"begin":331,"end":335},"obj":"hunflair:NA:Gene"},{"id":"M_19","span":{"begin":621,"end":625},"obj":"hunflair:NA:Gene"},{"id":"M_20","span":{"begin":857,"end":861},"obj":"hunflair:NA:Gene"},{"id":"M_21","span":{"begin":919,"end":923},"obj":"hunflair:NA:Gene"},{"id":"M_22","span":{"begin":970,"end":974},"obj":"hunflair:NA:Gene"},{"id":"M_23","span":{"begin":1105,"end":1109},"obj":"hunflair:NA:Gene"},{"id":"M_24","span":{"begin":1120,"end":1124},"obj":"hunflair:NA:Gene"},{"id":"M_25","span":{"begin":1160,"end":1164},"obj":"hunflair:NA:Gene"},{"id":"M_26","span":{"begin":1228,"end":1232},"obj":"hunflair:NA:Gene"},{"id":"M_27","span":{"begin":1294,"end":1298},"obj":"hunflair:NA:Gene"},{"id":"M_28","span":{"begin":310,"end":330},"obj":"hunflair:NA:Gene"},{"id":"M_29","span":{"begin":5,"end":9},"obj":"hunflair:NA:Gene"},{"id":"M_30","span":{"begin":848,"end":852},"obj":"hunflair:NA:Gene"},{"id":"M_31","span":{"begin":1053,"end":1057},"obj":"hunflair:NA:Gene"},{"id":"M_32","span":{"begin":1059,"end":1063},"obj":"hunflair:NA:Gene"},{"id":"M_33","span":{"begin":1285,"end":1289},"obj":"hunflair:NA:Gene"},{"id":"M_34","span":{"begin":583,"end":586},"obj":"hunflair:NA:Gene"}],"text":"Rice ASR1 and ASR5 are complementary transcription factors regulating aluminum responsive genes.\nRice is the most tolerant staple crop to aluminum (Al) toxicity, which is a limiting stress for grain production worldwide. This Al tolerance is the result of combined mechanisms that are triggered in part by the transcription factor ASR5. ASRs are dual target proteins that participate as chaperones in the cytoplasm and as transcription factors in the nucleus. Moreover, these proteins respond to biotic and abiotic stresses, including salt, drought and Al. Rice plants with silenced ASR genes are highly sensitive to Al. ASR5, a well-characterized protein, binds to specific cis elements in Al responsive genes and regulates their expression. Because the Al sensitive phenotype found in silenced rice plants could be due to the mutual silencing of ASR1 and ASR5, we investigated the effect of the specific silencing of ASR5. Plants with artificial microRNA silencing of ASR5 present a non-transformed phenotype in response to Al due to the induction of ASR1. ASR1 has the same subcellular localization as ASR5, binds to ASR5 cis-regulatory elements, regulates ASR5 regulated genes in a non-preferential manner and might replace ASR5 under certain conditions. Our results indicate that ASR1 and ASR5 act in concert and complementarily to regulate gene expression in response to Al."}

OryzaGP_2021

OryzaGP

{"project":"OryzaGP","denotations":[{"id":"T1","span":{"begin":5,"end":9},"obj":"gene"},{"id":"T2","span":{"begin":14,"end":18},"obj":"gene"},{"id":"T3","span":{"begin":848,"end":852},"obj":"gene"},{"id":"T4","span":{"begin":1285,"end":1289},"obj":"gene"},{"id":"T5","span":{"begin":1294,"end":1298},"obj":"gene"}],"text":"Rice ASR1 and ASR5 are complementary transcription factors regulating aluminum responsive genes.\nRice is the most tolerant staple crop to aluminum (Al) toxicity, which is a limiting stress for grain production worldwide. This Al tolerance is the result of combined mechanisms that are triggered in part by the transcription factor ASR5. ASRs are dual target proteins that participate as chaperones in the cytoplasm and as transcription factors in the nucleus. Moreover, these proteins respond to biotic and abiotic stresses, including salt, drought and Al. Rice plants with silenced ASR genes are highly sensitive to Al. ASR5, a well-characterized protein, binds to specific cis elements in Al responsive genes and regulates their expression. Because the Al sensitive phenotype found in silenced rice plants could be due to the mutual silencing of ASR1 and ASR5, we investigated the effect of the specific silencing of ASR5. Plants with artificial microRNA silencing of ASR5 present a non-transformed phenotype in response to Al due to the induction of ASR1. ASR1 has the same subcellular localization as ASR5, binds to ASR5 cis-regulatory elements, regulates ASR5 regulated genes in a non-preferential manner and might replace ASR5 under certain conditions. Our results indicate that ASR1 and ASR5 act in concert and complementarily to regulate gene expression in response to Al."}

Oryza_sentences

{"project":"Oryza_sentences","blocks":[{"id":"T1","span":{"begin":0,"end":96},"obj":"Sentence"},{"id":"T2","span":{"begin":97,"end":220},"obj":"Sentence"},{"id":"T3","span":{"begin":221,"end":336},"obj":"Sentence"},{"id":"T4","span":{"begin":337,"end":459},"obj":"Sentence"},{"id":"T5","span":{"begin":460,"end":742},"obj":"Sentence"},{"id":"T6","span":{"begin":743,"end":924},"obj":"Sentence"},{"id":"T7","span":{"begin":925,"end":1058},"obj":"Sentence"},{"id":"T8","span":{"begin":1059,"end":1258},"obj":"Sentence"},{"id":"T9","span":{"begin":1259,"end":1380},"obj":"Sentence"}],"text":"Rice ASR1 and ASR5 are complementary transcription factors regulating aluminum responsive genes.\nRice is the most tolerant staple crop to aluminum (Al) toxicity, which is a limiting stress for grain production worldwide. This Al tolerance is the result of combined mechanisms that are triggered in part by the transcription factor ASR5. ASRs are dual target proteins that participate as chaperones in the cytoplasm and as transcription factors in the nucleus. Moreover, these proteins respond to biotic and abiotic stresses, including salt, drought and Al. Rice plants with silenced ASR genes are highly sensitive to Al. ASR5, a well-characterized protein, binds to specific cis elements in Al responsive genes and regulates their expression. Because the Al sensitive phenotype found in silenced rice plants could be due to the mutual silencing of ASR1 and ASR5, we investigated the effect of the specific silencing of ASR5. Plants with artificial microRNA silencing of ASR5 present a non-transformed phenotype in response to Al due to the induction of ASR1. ASR1 has the same subcellular localization as ASR5, binds to ASR5 cis-regulatory elements, regulates ASR5 regulated genes in a non-preferential manner and might replace ASR5 under certain conditions. Our results indicate that ASR1 and ASR5 act in concert and complementarily to regulate gene expression in response to Al."}