PubMed:26102294 / 1599-1841 JSONTXT

{"project":"LitCoin-sentences","denotations":[{"id":"T10","span":{"begin":0,"end":242},"obj":"Sentence"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin-entities","denotations":[{"id":"10733","span":{"begin":36,"end":41},"obj":"GeneOrGeneProduct"},{"id":"10734","span":{"begin":76,"end":80},"obj":"CellLine"},{"id":"10735","span":{"begin":179,"end":186},"obj":"GeneOrGeneProduct"},{"id":"10736","span":{"begin":204,"end":208},"obj":"GeneOrGeneProduct"}],"attributes":[{"id":"A30","pred":"db_id","subj":"10733","obj":"NCBIGene:5460"},{"id":"A31","pred":"db_id","subj":"10734","obj":"NCBITaxon:9606"},{"id":"A32","pred":"db_id","subj":"10735","obj":"NCBIGene:1026"},{"id":"A33","pred":"db_id","subj":"10736","obj":"NCBIGene:5562"}],"namespaces":[{"prefix":"_base","uri":"https://w3id.org/biolink/vocab/"},{"prefix":"MESH","uri":"http://id.nlm.nih.gov/mesh/"},{"prefix":"NCBITaxon","uri":"https://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id="},{"prefix":"NCBIGene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"OMIM","uri":"https://www.omim.org/entry/"},{"prefix":"DBSNP","uri":"https://www.ncbi.nlm.nih.gov/snp/"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin-GeneOrGeneProduct-v2","denotations":[{"id":"T20","span":{"begin":99,"end":103},"obj":"GeneOrGeneProduct"},{"id":"T21","span":{"begin":179,"end":186},"obj":"GeneOrGeneProduct"},{"id":"T22","span":{"begin":204,"end":208},"obj":"GeneOrGeneProduct"},{"id":"T23","span":{"begin":232,"end":241},"obj":"GeneOrGeneProduct"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin-GeneOrGeneProduct-v0","denotations":[{"id":"T39","span":{"begin":16,"end":24},"obj":"GeneOrGeneProduct"},{"id":"T40","span":{"begin":66,"end":72},"obj":"GeneOrGeneProduct"},{"id":"T41","span":{"begin":81,"end":86},"obj":"GeneOrGeneProduct"},{"id":"T42","span":{"begin":99,"end":103},"obj":"GeneOrGeneProduct"},{"id":"T43","span":{"begin":179,"end":186},"obj":"GeneOrGeneProduct"},{"id":"T44","span":{"begin":204,"end":208},"obj":"GeneOrGeneProduct"},{"id":"T45","span":{"begin":209,"end":214},"obj":"GeneOrGeneProduct"},{"id":"T46","span":{"begin":223,"end":231},"obj":"GeneOrGeneProduct"},{"id":"T47","span":{"begin":232,"end":241},"obj":"GeneOrGeneProduct"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin-GeneOrGeneProduct-v3","denotations":[{"id":"T11","span":{"begin":179,"end":186},"obj":"GeneOrGeneProduct"},{"id":"T12","span":{"begin":204,"end":208},"obj":"GeneOrGeneProduct"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin_CellLine","denotations":[{"id":"T3","span":{"begin":76,"end":80},"obj":"CellLine"}],"attributes":[{"id":"A3","pred":"cellosaurus_accession_id","subj":"T3","obj":"CVCL_0479"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}

{"project":"LitCoin-training-merged","denotations":[{"id":"T3","span":{"begin":76,"end":80},"obj":"CellLine"},{"id":"T37772","span":{"begin":204,"end":208},"obj":"GeneOrGeneProduct"},{"id":"T11","span":{"begin":179,"end":186},"obj":"GeneOrGeneProduct"}],"attributes":[{"id":"A3","pred":"cellosaurus_accession_id","subj":"T3","obj":"CVCL_0479"}],"text":"Together, these findings imply that OCT4A induction following DNA damage in PA-1 cells, performs a cell stress, rather than self-renewal, function by moderating the expression of p21Cip1, which alongside AMPK helps to then regulate autophagy."}