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PubMed:25627160 / 110-592 JSONTXT

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PubMed_Structured_Abstracts

Id Subject Object Predicate Lexical cue
T1 0-482 BACKGROUND denotes The molecular mechanisms underlying autistic behaviors remain to be elucidated. Mutations in genes linked to autism adversely affect molecules regulating dendritic spine formation, function and plasticity, and some increase the mammalian target of rapamycin, mTOR, a regulator of protein synthesis at spines. Here, we investigated whether the Akt/mTOR pathway is disrupted in idiopathic autism and in rats exposed to valproic acid, an animal model exhibiting autistic-like behavior.

PubmedHPO

Id Subject Object Predicate Lexical cue
T1 36-54 HP_0000729 denotes autistic behaviors
T2 36-44 HP_0000717 denotes autistic
T3 109-115 HP_0000717 denotes autism
T4 387-393 HP_0000717 denotes autism
T5 459-467 HP_0000717 denotes autistic

NEUROSES

Id Subject Object Predicate Lexical cue
T5 406-413 PATO_0001646 denotes exposed
T9 406-413 PATO_0002425 denotes exposed
T17 133-142 CHEBI_25367 denotes molecules
T19 154-163 PATO_0002045 denotes dendritic
T20 181-189 PATO_0000173 denotes function
T22 248-257 CHEBI_9168 denotes rapamycin
T23 280-287 CHEBI_16541 denotes protein
T26 280-287 CHEBI_36080 denotes protein
T29 363-372 PATO_0001507 denotes disrupted

BLAH2015_Annotations_test_5

Id Subject Object Predicate Lexical cue
T10 133-142 CHEBI_25367 denotes molecules
T12 154-163 PATO_0002045 denotes dendritic
T13 181-189 PATO_0000173 denotes function
T15 248-257 CHEBI_9168 denotes rapamycin
T16 280-287 CHEBI_36080 denotes protein
T35 280-287 PM7566 denotes protein
T19 280-287 CHEBI_16541 denotes protein
T22 363-372 PATO_0001507 denotes disrupted
T2 406-413 PATO_0002425 denotes exposed
T6 417-430 CHEBI_39867 denotes valproic acid