PubMed:25174649
Annnotations
test-210614
Id | Subject | Object | Predicate | Lexical cue | proteinmutation |
---|---|---|---|---|---|
25174649_0 | 100-106 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_1 | 840-846 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_2 | 676-682 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_3 | 626-632 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_4 | 489-495 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_5 | 389-395 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_6 | 266-272 | ProteinMutation | denotes | G2019S | rs34637584 |
PubTator4TogoVar
Id | Subject | Object | Predicate | Lexical cue | proteinmutation |
---|---|---|---|---|---|
25174649_0 | 100-106 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_1 | 840-846 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_2 | 676-682 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_3 | 626-632 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_4 | 489-495 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_5 | 389-395 | ProteinMutation | denotes | G2019S | rs34637584 |
25174649_6 | 266-272 | ProteinMutation | denotes | G2019S | rs34637584 |
c_corpus
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 57-88 | SO:0000153 | denotes | bacterial artificial chromosome |
T2 | 67-77 | 32630 | denotes | artificial |
T3 | 78-88 | GO:0005694 | denotes | chromosome |
T5 | 89-99 | SO:0000781 | denotes | transgenic |
T6 | 105-114 | CHEBI:15603 | denotes | S leucine |
T8 | 107-114 | 6308 | denotes | leucine |
T9 | 107-114 | SO:0001437 | denotes | leucine |
T13 | 138-142 | 10118 | denotes | rats |
T14 | 138-142 | 10116 | denotes | rats |
T15 | 138-142 | D051381 | denotes | rats |
T17 | 157-164 | 6308 | denotes | leucine |
T18 | 157-164 | SO:0001437 | denotes | leucine |
T16 | 157-164 | CHEBI:15603 | denotes | leucine |
T19 | 157-164 | D007930 | denotes | leucine |
T20 | 157-164 | CHEBI:25017 | denotes | leucine |
T21 | 157-164 | D007930 | denotes | leucine |
T22 | 189-194 | PR:Q5S006 | denotes | LRRK2 |
T23 | 189-194 | PR:000003033 | denotes | LRRK2 |
T24 | 189-194 | PR:Q5S007 | denotes | LRRK2 |
T25 | 202-220 | C566739 | denotes | autosomal dominant |
T26 | 221-231 | D000067562 | denotes | late-onset |
T27 | 221-231 | D000067562 | denotes | late-onset |
T32 | 232-251 | D010300 | denotes | Parkinson's disease |
T33 | 232-251 | D010300 | denotes | Parkinson's disease |
T36 | 273-281 | SO:0000109 | denotes | mutation |
T37 | 296-302 | SO:0000417 | denotes | domain |
T38 | 306-311 | PR:Q5S006 | denotes | LRRK2 |
T39 | 306-311 | PR:000003033 | denotes | LRRK2 |
T40 | 306-311 | PR:Q5S007 | denotes | LRRK2 |
T41 | 370-385 | GO:0016301 | denotes | kinase activity |
T42 | 396-401 | PR:Q5S006 | denotes | LRRK2 |
T43 | 396-401 | PR:000003033 | denotes | LRRK2 |
T44 | 396-401 | PR:Q5S007 | denotes | LRRK2 |
T45 | 431-439 | CHEBI:78059 | denotes | carriers |
T46 | 496-501 | PR:Q5S006 | denotes | LRRK2 |
T47 | 496-501 | PR:000003033 | denotes | LRRK2 |
T48 | 496-501 | PR:Q5S007 | denotes | LRRK2 |
T49 | 573-604 | SO:0000153 | denotes | bacterial artificial chromosome |
T50 | 583-593 | 32630 | denotes | artificial |
T51 | 594-604 | GO:0005694 | denotes | chromosome |
T53 | 605-608 | 10116 | denotes | rat |
T57 | 605-608 | D051381 | denotes | rat |
T54 | 605-608 | PR:P63003-1 | denotes | rat |
T55 | 605-608 | PR:Q2G0B1 | denotes | rat |
T56 | 605-608 | PR:Q8VHJ4 | denotes | rat |
T58 | 620-625 | D006801 | denotes | human |
T59 | 633-638 | PR:Q5S006 | denotes | LRRK2 |
T60 | 633-638 | PR:000003033 | denotes | LRRK2 |
T61 | 633-638 | PR:Q5S007 | denotes | LRRK2 |
T62 | 683-688 | PR:Q5S006 | denotes | LRRK2 |
T63 | 683-688 | PR:000003033 | denotes | LRRK2 |
T64 | 683-688 | PR:Q5S007 | denotes | LRRK2 |
T65 | 740-749 | SO:0000817 | denotes | wild-type |
T67 | 750-753 | PR:P63003-1 | denotes | rat |
T68 | 750-753 | PR:Q2G0B1 | denotes | rat |
T69 | 750-753 | PR:Q8VHJ4 | denotes | rat |
T66 | 750-753 | 10116 | denotes | rat |
T70 | 750-753 | D051381 | denotes | rat |
T71 | 754-759 | PR:Q5S006 | denotes | LRRK2 |
T72 | 754-759 | PR:000003033 | denotes | LRRK2 |
T73 | 754-759 | PR:Q5S007 | denotes | LRRK2 |
T74 | 847-852 | PR:Q5S006 | denotes | LRRK2 |
T75 | 847-852 | PR:000003033 | denotes | LRRK2 |
T76 | 847-852 | PR:Q5S007 | denotes | LRRK2 |
T77 | 881-889 | UBERON:0002435 | denotes | striatum |
T78 | 881-889 | UBERON:0005383 | denotes | striatum |
T79 | 894-910 | UBERON:0002038 | denotes | substantia nigra |
T80 | 922-953 | D052247 | denotes | inducible nitric oxide synthase |
T84 | 932-944 | 7442 | denotes | nitric oxide |
T85 | 932-953 | O61309 | denotes | nitric oxide synthase |
T86 | 932-953 | Q8T8C0 | denotes | nitric oxide synthase |
T87 | 932-953 | Q9I9M2 | denotes | nitric oxide synthase |
T88 | 932-953 | Q27571 | denotes | nitric oxide synthase |
T90 | 932-953 | O61608 | denotes | nitric oxide synthase |
T97 | 975-983 | 3628 | denotes | dopamine |
T93 | 975-983 | CHEBI:59905 | denotes | dopamine |
T94 | 975-983 | CHEBI:18243 | denotes | dopamine |
T95 | 975-983 | D004298 | denotes | dopamine |
T96 | 975-983 | D004298 | denotes | dopamine |
T98 | 1051-1061 | SO:0000781 | denotes | transgenic |
T99 | 1051-1066 | D055647 | denotes | transgenic rats |
T103 | 1081-1090 | SO:0000817 | denotes | wild-type |
T104 | 1306-1313 | D004194 | denotes | disease |
T105 | 1306-1313 | D004194 | denotes | disease |
T106 | 1393-1397 | SO:0000704 | denotes | gene |
Inflammaging
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 0-143 | Sentence | denotes | Behavioral, neurochemical, and pathologic alterations in bacterial artificial chromosome transgenic G2019S leucine-rich repeated kinase 2 rats. |
T2 | 144-360 | Sentence | denotes | Mutations in leucine-rich repeated kinase 2 (LRRK2) cause autosomal dominant late-onset Parkinson's disease (PD), and the G2019S mutation in the kinase domain of LRRK2 is the most common genetic cause of familial PD. |
T3 | 361-515 | Sentence | denotes | Enhanced kinase activity of G2019S LRRK2 is a suspected mechanism for carriers to develop PD but pathophysiological function of G2019S LRRK2 is not clear. |
T4 | 516-639 | Sentence | denotes | The objective of the present study was to characterize a bacterial artificial chromosome rat expressing human G2019S LRRK2. |
T5 | 640-760 | Sentence | denotes | Immunoblotting analysis showed that G2019S LRRK2 expression was approximately 5-8 times higher than wild-type rat LRRK2. |
T6 | 761-1171 | Sentence | denotes | At ages of 4, 8, and 12 months, our characterization showed that expression of G2019S LRRK2 induced oxidative stress in striatum and substantia nigra, increased inducible nitric oxide synthase expression in nigral dopamine neurons, and abnormal morphology of nigral dopaminergic neurons in transgenic rats compared with wild-type, without inducing overt neurodegeneration in nigrostriatal dopaminergic neurons. |
T7 | 1172-1430 | Sentence | denotes | Thus, we conclude that although this model does not reproduce the key features of end-stage PD, important preclinical features of the disease are evident, which may be useful in studying the earliest stages of PD and for gene-environment interaction studies. |
T1 | 0-143 | Sentence | denotes | Behavioral, neurochemical, and pathologic alterations in bacterial artificial chromosome transgenic G2019S leucine-rich repeated kinase 2 rats. |
T2 | 144-360 | Sentence | denotes | Mutations in leucine-rich repeated kinase 2 (LRRK2) cause autosomal dominant late-onset Parkinson's disease (PD), and the G2019S mutation in the kinase domain of LRRK2 is the most common genetic cause of familial PD. |
T3 | 361-515 | Sentence | denotes | Enhanced kinase activity of G2019S LRRK2 is a suspected mechanism for carriers to develop PD but pathophysiological function of G2019S LRRK2 is not clear. |
T4 | 516-639 | Sentence | denotes | The objective of the present study was to characterize a bacterial artificial chromosome rat expressing human G2019S LRRK2. |
T5 | 640-760 | Sentence | denotes | Immunoblotting analysis showed that G2019S LRRK2 expression was approximately 5-8 times higher than wild-type rat LRRK2. |
T6 | 761-1171 | Sentence | denotes | At ages of 4, 8, and 12 months, our characterization showed that expression of G2019S LRRK2 induced oxidative stress in striatum and substantia nigra, increased inducible nitric oxide synthase expression in nigral dopamine neurons, and abnormal morphology of nigral dopaminergic neurons in transgenic rats compared with wild-type, without inducing overt neurodegeneration in nigrostriatal dopaminergic neurons. |
T7 | 1172-1430 | Sentence | denotes | Thus, we conclude that although this model does not reproduce the key features of end-stage PD, important preclinical features of the disease are evident, which may be useful in studying the earliest stages of PD and for gene-environment interaction studies. |
PubmedHPO
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 202-220 | HP_0000006 | denotes | autosomal dominant |
T2 | 202-225 | HP_0000006 | denotes | autosomal dominant late |
T3 | 232-241 | HP_0001300 | denotes | Parkinson |
T4 | 1115-1132 | HP_0002180 | denotes | neurodegeneration |
UseCases_ArguminSci_Discourse
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 0-143 | DRI_Background | denotes | Behavioral, neurochemical, and pathologic alterations in bacterial artificial chromosome transgenic G2019S leucine-rich repeated kinase 2 rats. |
T2 | 144-360 | DRI_Background | denotes | Mutations in leucine-rich repeated kinase 2 (LRRK2) cause autosomal dominant late-onset Parkinson's disease (PD), and the G2019S mutation in the kinase domain of LRRK2 is the most common genetic cause of familial PD. |
T3 | 361-388 | DRI_Background | denotes | Enhanced kinase activity of |
T4 | 389-401 | Token_Label.OUTSIDE | denotes | G2019S LRRK2 |
T5 | 402-488 | DRI_Background | denotes | is a suspected mechanism for carriers to develop PD but pathophysiological function of |
T6 | 489-501 | Token_Label.OUTSIDE | denotes | G2019S LRRK2 |
T7 | 502-515 | DRI_Background | denotes | is not clear. |
T8 | 516-625 | DRI_Background | denotes | The objective of the present study was to characterize a bacterial artificial chromosome rat expressing human |
T9 | 626-638 | Token_Label.OUTSIDE | denotes | G2019S LRRK2 |
T10 | 638-639 | DRI_Background | denotes | . |
T11 | 640-675 | DRI_Outcome | denotes | Immunoblotting analysis showed that |
T12 | 676-688 | Token_Label.OUTSIDE | denotes | G2019S LRRK2 |
T13 | 689-760 | DRI_Outcome | denotes | expression was approximately 5-8 times higher than wild-type rat LRRK2. |
T14 | 761-839 | DRI_Outcome | denotes | At ages of 4, 8, and 12 months, our characterization showed that expression of |
T15 | 840-852 | Token_Label.OUTSIDE | denotes | G2019S LRRK2 |
T16 | 853-1171 | DRI_Outcome | denotes | induced oxidative stress in striatum and substantia nigra, increased inducible nitric oxide synthase expression in nigral dopamine neurons, and abnormal morphology of nigral dopaminergic neurons in transgenic rats compared with wild-type, without inducing overt neurodegeneration in nigrostriatal dopaminergic neurons. |
T17 | 1172-1430 | DRI_Challenge | denotes | Thus, we conclude that although this model does not reproduce the key features of end-stage PD, important preclinical features of the disease are evident, which may be useful in studying the earliest stages of PD and for gene-environment interaction studies. |
PubMed_ArguminSci
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 144-360 | DRI_Background | denotes | Mutations in leucine-rich repeated kinase 2 (LRRK2) cause autosomal dominant late-onset Parkinson's disease (PD), and the G2019S mutation in the kinase domain of LRRK2 is the most common genetic cause of familial PD. |
T2 | 361-388 | DRI_Background | denotes | Enhanced kinase activity of |
T3 | 402-488 | DRI_Background | denotes | is a suspected mechanism for carriers to develop PD but pathophysiological function of |
T4 | 502-515 | DRI_Background | denotes | is not clear. |
T5 | 516-625 | DRI_Background | denotes | The objective of the present study was to characterize a bacterial artificial chromosome rat expressing human |
T6 | 638-639 | DRI_Background | denotes | . |
T7 | 640-675 | DRI_Outcome | denotes | Immunoblotting analysis showed that |
T8 | 689-760 | DRI_Outcome | denotes | expression was approximately 5-8 times higher than wild-type rat LRRK2. |
T9 | 761-839 | DRI_Outcome | denotes | At ages of 4, 8, and 12 months, our characterization showed that expression of |
T10 | 853-1171 | DRI_Outcome | denotes | induced oxidative stress in striatum and substantia nigra, increased inducible nitric oxide synthase expression in nigral dopamine neurons, and abnormal morphology of nigral dopaminergic neurons in transgenic rats compared with wild-type, without inducing overt neurodegeneration in nigrostriatal dopaminergic neurons. |
T11 | 1172-1430 | DRI_Challenge | denotes | Thus, we conclude that although this model does not reproduce the key features of end-stage PD, important preclinical features of the disease are evident, which may be useful in studying the earliest stages of PD and for gene-environment interaction studies. |
DisGeNET5_variant_disease
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
25174649-2#28#34#geners34637584 | 389-395 | geners34637584 | denotes | G2019S |
25174649-2#128#134#geners34637584 | 489-495 | geners34637584 | denotes | G2019S |
25174649-2#90#92#diseaseC4274355 | 451-453 | diseaseC4274355 | denotes | PD |
28#34#geners3463758490#92#diseaseC4274355 | 25174649-2#28#34#geners34637584 | 25174649-2#90#92#diseaseC4274355 | associated_with | G2019S,PD |
128#134#geners3463758490#92#diseaseC4274355 | 25174649-2#128#134#geners34637584 | 25174649-2#90#92#diseaseC4274355 | associated_with | G2019S,PD |
DisGeNET5_gene_disease
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
25174649-2#35#40#gene120892 | 396-401 | gene120892 | denotes | LRRK2 |
25174649-2#135#140#gene120892 | 496-501 | gene120892 | denotes | LRRK2 |
25174649-2#90#92#diseaseC4274355 | 451-453 | diseaseC4274355 | denotes | PD |
35#40#gene12089290#92#diseaseC4274355 | 25174649-2#35#40#gene120892 | 25174649-2#90#92#diseaseC4274355 | associated_with | LRRK2,PD |
135#140#gene12089290#92#diseaseC4274355 | 25174649-2#135#140#gene120892 | 25174649-2#90#92#diseaseC4274355 | associated_with | LRRK2,PD |