| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-137 |
Sentence |
denotes |
Transcription factor C/EBPα-induced microRNA-30c inactivates Notch1 during granulopoiesis and is downregulated in acute myeloid leukemia. |
| TextSentencer_T2 |
138-305 |
Sentence |
denotes |
The transcription factor CCAAT enhancer binding protein α (C/EBPα) is a master regulator in granulopoiesis and is frequently disrupted in acute myeloid leukemia (AML). |
| TextSentencer_T3 |
306-421 |
Sentence |
denotes |
We have previously shown that C/EBPα exerts its effects by regulating microRNAs (miRs) such as miR-223 and miR-34a. |
| TextSentencer_T4 |
422-507 |
Sentence |
denotes |
Here, we confirm miR-30c as a novel important target of C/EBPα during granulopoiesis. |
| TextSentencer_T5 |
508-718 |
Sentence |
denotes |
Thus, wild-type C/EBPα-p42 directly upregulates miR-30c expression, whereas C/EBPα-p30, found in AML, does not. miR-30c is downregulated in AML, especially in normal karyotype AML patients with CEBPA mutations. |
| TextSentencer_T6 |
719-835 |
Sentence |
denotes |
An induced C/EBPα knockout in mice leads to a significant downregulation of miR-30c expression in bone marrow cells. |
| TextSentencer_T7 |
836-887 |
Sentence |
denotes |
We identified NOTCH1 as a direct target of miR-30c. |
| TextSentencer_T8 |
888-1040 |
Sentence |
denotes |
Finally, a block of miR-30c prevents C/EBPα-induced downregulation of Notch1 protein and leads to a reduced CD11b expression in myeloid differentiation. |
| TextSentencer_T9 |
1041-1228 |
Sentence |
denotes |
Our study presents the first evidence that C/EBPα, miR-30c, and Notch1 together play a critical role in granulocytic differentiation and AML, and particularly in AML with CEBPA mutations. |
| TextSentencer_T10 |
1229-1370 |
Sentence |
denotes |
These data reveal the importance of deregulated miRNA expression in leukemia and may provide novel biomarkers and therapeutic targets in AML. |
| T1 |
0-137 |
Sentence |
denotes |
Transcription factor C/EBPα-induced microRNA-30c inactivates Notch1 during granulopoiesis and is downregulated in acute myeloid leukemia. |
| T2 |
138-305 |
Sentence |
denotes |
The transcription factor CCAAT enhancer binding protein α (C/EBPα) is a master regulator in granulopoiesis and is frequently disrupted in acute myeloid leukemia (AML). |
| T3 |
306-421 |
Sentence |
denotes |
We have previously shown that C/EBPα exerts its effects by regulating microRNAs (miRs) such as miR-223 and miR-34a. |
| T4 |
422-507 |
Sentence |
denotes |
Here, we confirm miR-30c as a novel important target of C/EBPα during granulopoiesis. |
| T5 |
508-718 |
Sentence |
denotes |
Thus, wild-type C/EBPα-p42 directly upregulates miR-30c expression, whereas C/EBPα-p30, found in AML, does not. miR-30c is downregulated in AML, especially in normal karyotype AML patients with CEBPA mutations. |
| T6 |
719-835 |
Sentence |
denotes |
An induced C/EBPα knockout in mice leads to a significant downregulation of miR-30c expression in bone marrow cells. |
| T7 |
836-887 |
Sentence |
denotes |
We identified NOTCH1 as a direct target of miR-30c. |
| T8 |
888-1040 |
Sentence |
denotes |
Finally, a block of miR-30c prevents C/EBPα-induced downregulation of Notch1 protein and leads to a reduced CD11b expression in myeloid differentiation. |
| T9 |
1041-1228 |
Sentence |
denotes |
Our study presents the first evidence that C/EBPα, miR-30c, and Notch1 together play a critical role in granulocytic differentiation and AML, and particularly in AML with CEBPA mutations. |
| T10 |
1229-1370 |
Sentence |
denotes |
These data reveal the importance of deregulated miRNA expression in leukemia and may provide novel biomarkers and therapeutic targets in AML. |