| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-122 |
Sentence |
denotes |
Tryptophan catabolites from microbiota engage aryl hydrocarbon receptor and balance mucosal reactivity via interleukin-22. |
| T2 |
123-338 |
Sentence |
denotes |
Endogenous tryptophan (Trp) metabolites have an important role in mammalian gut immune homeostasis, yet the potential contribution of Trp metabolites from resident microbiota has never been addressed experimentally. |
| T3 |
339-456 |
Sentence |
denotes |
Here, we describe a metabolic pathway whereby Trp metabolites from the microbiota balance mucosal reactivity in mice. |
| T4 |
457-731 |
Sentence |
denotes |
Switching from sugar to Trp as an energy source (e.g., under conditions of unrestricted Trp availability), highly adaptive lactobacilli are expanded and produce an aryl hydrocarbon receptor (AhR) ligand-indole-3-aldehyde-that contributes to AhR-dependent Il22 transcription. |
| T5 |
732-948 |
Sentence |
denotes |
The resulting IL-22-dependent balanced mucosal response allows for survival of mixed microbial communities yet provides colonization resistance to the fungus Candida albicans and mucosal protection from inflammation. |
| T6 |
949-1122 |
Sentence |
denotes |
Thus, the microbiota-AhR axis might represent an important strategy pursued by coevolutive commensalism for fine tuning host mucosal reactivity contingent on Trp catabolism. |
| T1 |
0-122 |
Sentence |
denotes |
Tryptophan catabolites from microbiota engage aryl hydrocarbon receptor and balance mucosal reactivity via interleukin-22. |
| T2 |
123-338 |
Sentence |
denotes |
Endogenous tryptophan (Trp) metabolites have an important role in mammalian gut immune homeostasis, yet the potential contribution of Trp metabolites from resident microbiota has never been addressed experimentally. |
| T3 |
339-456 |
Sentence |
denotes |
Here, we describe a metabolic pathway whereby Trp metabolites from the microbiota balance mucosal reactivity in mice. |
| T4 |
457-731 |
Sentence |
denotes |
Switching from sugar to Trp as an energy source (e.g., under conditions of unrestricted Trp availability), highly adaptive lactobacilli are expanded and produce an aryl hydrocarbon receptor (AhR) ligand-indole-3-aldehyde-that contributes to AhR-dependent Il22 transcription. |
| T5 |
732-948 |
Sentence |
denotes |
The resulting IL-22-dependent balanced mucosal response allows for survival of mixed microbial communities yet provides colonization resistance to the fungus Candida albicans and mucosal protection from inflammation. |
| T6 |
949-1122 |
Sentence |
denotes |
Thus, the microbiota-AhR axis might represent an important strategy pursued by coevolutive commensalism for fine tuning host mucosal reactivity contingent on Trp catabolism. |