PubMed:23811795 / 1352-1589 JSONTXT

{"project":"sentences","denotations":[{"id":"TextSentencer_T8","span":{"begin":0,"end":237},"obj":"Sentence"},{"id":"T8","span":{"begin":0,"end":237},"obj":"Sentence"},{"id":"T8","span":{"begin":0,"end":237},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"GlycoBiology-FMA","denotations":[{"id":"_T17","span":{"begin":5,"end":13},"obj":"FMAID:82768"},{"id":"_T18","span":{"begin":5,"end":13},"obj":"FMAID:196757"},{"id":"_T19","span":{"begin":231,"end":236},"obj":"FMAID:68646"},{"id":"_T20","span":{"begin":231,"end":236},"obj":"FMAID:169002"}],"namespaces":[{"prefix":"FMAID","uri":"http://purl.org/sig/ont/fma/fma"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"uniprot-human","denotations":[{"id":"T9","span":{"begin":0,"end":4},"obj":"http://www.uniprot.org/uniprot/Q9GZX1"},{"id":"T17","span":{"begin":175,"end":179},"obj":"http://www.uniprot.org/uniprot/Q16512"},{"id":"T26","span":{"begin":175,"end":179},"obj":"http://www.uniprot.org/uniprot/Q13153"},{"id":"T53","span":{"begin":5,"end":31},"obj":"http://www.uniprot.org/uniprot/Q8TAI4"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"GlycoBiology-NCBITAXON","denotations":[{"id":"T8","span":{"begin":231,"end":236},"obj":"http://purl.bioontology.org/ontology/STY/T025"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"GO-BP","denotations":[{"id":"T6","span":{"begin":102,"end":109},"obj":"http://purl.obolibrary.org/obo/GO_0043276"},{"id":"T11","span":{"begin":0,"end":4},"obj":"http://purl.obolibrary.org/obo/GO_0005006"},{"id":"T28","span":{"begin":91,"end":97},"obj":"http://purl.obolibrary.org/obo/GO_0040007"},{"id":"T46","span":{"begin":14,"end":31},"obj":"http://purl.obolibrary.org/obo/GO_0033673"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"GO-CC","denotations":[{"id":"T5","span":{"begin":231,"end":236},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}

{"project":"mondo_disease","denotations":[{"id":"T9","span":{"begin":222,"end":230},"obj":"Disease"}],"attributes":[{"id":"A9","pred":"mondo_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/MONDO_0007256"}],"text":"EGFR tyrosine kinase inhibitors AG1478 and Iressa treatment declined anchorage-independent growth and anoikis resistance, which could be rescued by constitutive active mutant PAK1 T423E coexpression in Mgat5-overexpressed hepatoma cells."}