| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-70 |
Sentence |
denotes |
Genotype-phenotype correlation in multiple endocrine neoplasia type 2. |
| TextSentencer_T2 |
71-320 |
Sentence |
denotes |
Multiple endocrine neoplasia type 2 is an autosomal-dominant hereditary cancer syndrome caused by missense gain-of-function mutations of the rearranged during transfection proto-oncogene, which encodes the receptor tyrosine kinase, on chromosome 10. |
| TextSentencer_T3 |
321-466 |
Sentence |
denotes |
It has a strong penetrance of medullary thyroid carcinomas and can be associated with bilateral pheochromocytoma and primary hyperparathyroidism. |
| TextSentencer_T4 |
467-688 |
Sentence |
denotes |
Multiple endocrine neoplasia type 2 is divided into three varieties depending on its clinical features: multiple endocrine neoplasia type 2A, multiple endocrine neoplasia type 2B, and familial medullary thyroid carcinoma. |
| TextSentencer_T5 |
689-895 |
Sentence |
denotes |
The specific rearranged during transfection mutation may suggest a predilection toward a particular phenotype and clinical course of medullary thyroid carcinoma, with strong genotype-phenotype correlations. |
| TextSentencer_T6 |
896-1191 |
Sentence |
denotes |
Offering rearranged during transfection testing is the best practice for the clinical management of patients at risk of developing multiple endocrine neoplasia type 2, and multiple endocrine neoplasia type 2 has become a classic model for the integration of molecular medicine into patient care. |
| TextSentencer_T7 |
1192-1412 |
Sentence |
denotes |
Recommendations on the timing of prophylactic thyroidectomy and extent of surgery are based on the classification of rearranged during transfection mutations into risk levels according to genotype-phenotype correlations. |
| TextSentencer_T8 |
1413-1643 |
Sentence |
denotes |
Earlier identification of patients with hereditary medullary thyroid carcinoma can change the presentation from clinical tumor to preclinical disease, resulting in a high cure rate of affected patients and a much better prognoses. |
| T1 |
0-70 |
Sentence |
denotes |
Genotype-phenotype correlation in multiple endocrine neoplasia type 2. |
| T2 |
71-320 |
Sentence |
denotes |
Multiple endocrine neoplasia type 2 is an autosomal-dominant hereditary cancer syndrome caused by missense gain-of-function mutations of the rearranged during transfection proto-oncogene, which encodes the receptor tyrosine kinase, on chromosome 10. |
| T3 |
321-466 |
Sentence |
denotes |
It has a strong penetrance of medullary thyroid carcinomas and can be associated with bilateral pheochromocytoma and primary hyperparathyroidism. |
| T4 |
467-688 |
Sentence |
denotes |
Multiple endocrine neoplasia type 2 is divided into three varieties depending on its clinical features: multiple endocrine neoplasia type 2A, multiple endocrine neoplasia type 2B, and familial medullary thyroid carcinoma. |
| T5 |
689-895 |
Sentence |
denotes |
The specific rearranged during transfection mutation may suggest a predilection toward a particular phenotype and clinical course of medullary thyroid carcinoma, with strong genotype-phenotype correlations. |
| T6 |
896-1191 |
Sentence |
denotes |
Offering rearranged during transfection testing is the best practice for the clinical management of patients at risk of developing multiple endocrine neoplasia type 2, and multiple endocrine neoplasia type 2 has become a classic model for the integration of molecular medicine into patient care. |
| T7 |
1192-1412 |
Sentence |
denotes |
Recommendations on the timing of prophylactic thyroidectomy and extent of surgery are based on the classification of rearranged during transfection mutations into risk levels according to genotype-phenotype correlations. |
| T8 |
1413-1643 |
Sentence |
denotes |
Earlier identification of patients with hereditary medullary thyroid carcinoma can change the presentation from clinical tumor to preclinical disease, resulting in a high cure rate of affected patients and a much better prognoses. |