| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-89 |
Sentence |
denotes |
Novel compound heterozygous mutation of MLYCD in a Chinese patient with malonic aciduria. |
| TextSentencer_T2 |
90-306 |
Sentence |
denotes |
A 3-year-old Chinese boy presented with prominent clinical features of malonic aciduria, including developmental delay, short stature, brain abnormalities and massive excretion of malonic acid and methylmalonic acid. |
| TextSentencer_T3 |
307-595 |
Sentence |
denotes |
Molecular characterization by DNA sequencing analysis and multiplex ligation-dependent probe amplification of the MLYCD gene revealed a heterozygous mutation (c.920T>G, p.Leu307Arg) in the patient and his father and a heterozygous deletion comprising exon 1 in the patient and his mother. |
| TextSentencer_T4 |
596-704 |
Sentence |
denotes |
The missense mutation (c.920T>G) was not found in 100 healthy controls and has not been reported previously. |
| TextSentencer_T5 |
705-813 |
Sentence |
denotes |
Our findings expand the number of reported cases and add a novel entry to the repertoire of MLYCD mutations. |
| T1 |
0-89 |
Sentence |
denotes |
Novel compound heterozygous mutation of MLYCD in a Chinese patient with malonic aciduria. |
| T2 |
90-306 |
Sentence |
denotes |
A 3-year-old Chinese boy presented with prominent clinical features of malonic aciduria, including developmental delay, short stature, brain abnormalities and massive excretion of malonic acid and methylmalonic acid. |
| T3 |
307-595 |
Sentence |
denotes |
Molecular characterization by DNA sequencing analysis and multiplex ligation-dependent probe amplification of the MLYCD gene revealed a heterozygous mutation (c.920T>G, p.Leu307Arg) in the patient and his father and a heterozygous deletion comprising exon 1 in the patient and his mother. |
| T4 |
596-704 |
Sentence |
denotes |
The missense mutation (c.920T>G) was not found in 100 healthy controls and has not been reported previously. |
| T5 |
705-813 |
Sentence |
denotes |
Our findings expand the number of reported cases and add a novel entry to the repertoire of MLYCD mutations. |