PubMed:21553387 JSONTXT

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    LitCoin-PubTator-for-Tuning

    {"project":"LitCoin-PubTator-for-Tuning","denotations":[{"id":"1","span":{"begin":9,"end":21},"obj":"DiseaseOrPhenotypicFeature"},{"id":"19","span":{"begin":212,"end":224},"obj":"DiseaseOrPhenotypicFeature"},{"id":"20","span":{"begin":370,"end":378},"obj":"OrganismTaxon"},{"id":"21","span":{"begin":384,"end":404},"obj":"DiseaseOrPhenotypicFeature"},{"id":"22","span":{"begin":590,"end":598},"obj":"OrganismTaxon"},{"id":"23","span":{"begin":625,"end":645},"obj":"DiseaseOrPhenotypicFeature"},{"id":"24","span":{"begin":659,"end":677},"obj":"DiseaseOrPhenotypicFeature"},{"id":"25","span":{"begin":682,"end":690},"obj":"OrganismTaxon"},{"id":"26","span":{"begin":709,"end":727},"obj":"DiseaseOrPhenotypicFeature"},{"id":"27","span":{"begin":734,"end":751},"obj":"DiseaseOrPhenotypicFeature"},{"id":"28","span":{"begin":768,"end":808},"obj":"DiseaseOrPhenotypicFeature"},{"id":"29","span":{"begin":822,"end":835},"obj":"DiseaseOrPhenotypicFeature"},{"id":"30","span":{"begin":839,"end":847},"obj":"OrganismTaxon"},{"id":"31","span":{"begin":931,"end":952},"obj":"DiseaseOrPhenotypicFeature"},{"id":"32","span":{"begin":963,"end":971},"obj":"OrganismTaxon"},{"id":"33","span":{"begin":990,"end":1002},"obj":"DiseaseOrPhenotypicFeature"},{"id":"34","span":{"begin":1018,"end":1041},"obj":"DiseaseOrPhenotypicFeature"},{"id":"35","span":{"begin":1056,"end":1076},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A28","pred":"tao:has_database_id","subj":"28","obj":"MESH:D014855"},{"id":"A20","pred":"tao:has_database_id","subj":"20","obj":"Tax:9606"},{"id":"A1","pred":"tao:has_database_id","subj":"1","obj":"MESH:D014549"},{"id":"A35","pred":"tao:has_database_id","subj":"35","obj":"MESH:D014549"},{"id":"A24","pred":"tao:has_database_id","subj":"24","obj":"MESH:D053201"},{"id":"A27","pred":"tao:has_database_id","subj":"27","obj":"MESH:D009122"},{"id":"A25","pred":"tao:has_database_id","subj":"25","obj":"Tax:9606"},{"id":"A23","pred":"tao:has_database_id","subj":"23","obj":"MESH:D014549"},{"id":"A19","pred":"tao:has_database_id","subj":"19","obj":"MESH:D014549"},{"id":"A30","pred":"tao:has_database_id","subj":"30","obj":"Tax:9606"},{"id":"A32","pred":"tao:has_database_id","subj":"32","obj":"Tax:9606"},{"id":"A26","pred":"tao:has_database_id","subj":"26","obj":"MESH:D053201"},{"id":"A34","pred":"tao:has_database_id","subj":"34","obj":"MESH:D019636"},{"id":"A31","pred":"tao:has_database_id","subj":"31","obj":"MESH:D009122"},{"id":"A29","pred":"tao:has_database_id","subj":"29","obj":"MESH:D003711"},{"id":"A22","pred":"tao:has_database_id","subj":"22","obj":"Tax:9606"},{"id":"A21","pred":"tao:has_database_id","subj":"21","obj":"MESH:D014549"},{"id":"A33","pred":"tao:has_database_id","subj":"33","obj":"MESH:D014549"}],"text":"[Urinary incontinence after radical prostatectomy. Neurophysiological and urodynamic diagnosis].\nL’incontinenza urinaria dopo prostatectomia radicale. Diagnosi urodinamica e neurofisiologica.\nOBJECTIVES: Urinary incontinence after radical prostatectomy presents different clinical features. We propose to clarify the etiology and the therapeutic approaches.\nMETHODS: 51 patients with urinary incontinence after radical prostatectomy underwent urodynamics, perineal EMG, perineal EMG frequency analysis and study of computerized sensory pudendal somatosensory evoked potentials.\nRESULTS: 18 patients (35%) were suffering from urinary incontinence secondary to overactive bladder, 14 patients (28%) had both an overactive bladder and a sphincter failure associated with Wallerian degenerative axonal neuropathy or segmental demyelination, 3 patients (6%) showed no significant urodynamics alterations but had signs of an incompetent sphincter neurapraxia, while 16 patients (31%) had urinary incontinence secondary to a degenerative neuropathy.\nCONCLUSIONS: Urinary incontinence after radical prostatectomy shows an etiologic diversification and therefore requires different therapeutic approaches"}

    LitCoin-Disease-Tuning-1

    {"project":"LitCoin-Disease-Tuning-1","denotations":[{"id":"T1","span":{"begin":1,"end":21},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T2","span":{"begin":204,"end":224},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T3","span":{"begin":384,"end":404},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T4","span":{"begin":625,"end":645},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T5","span":{"begin":659,"end":677},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T6","span":{"begin":709,"end":727},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T7","span":{"begin":822,"end":835},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T8","span":{"begin":982,"end":1002},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T9","span":{"begin":1056,"end":1076},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A9","pred":"ID:","subj":"T9","obj":"D014549"},{"id":"A7","pred":"ID:","subj":"T7","obj":"D003711"},{"id":"A5","pred":"ID:","subj":"T5","obj":"D053201"},{"id":"A3","pred":"ID:","subj":"T3","obj":"D014549"},{"id":"A2","pred":"ID:","subj":"T2","obj":"D014549"},{"id":"A4","pred":"ID:","subj":"T4","obj":"D014549"},{"id":"A8","pred":"ID:","subj":"T8","obj":"D014549"},{"id":"A1","pred":"ID:","subj":"T1","obj":"D014549"},{"id":"A6","pred":"ID:","subj":"T6","obj":"D053201"}],"text":"[Urinary incontinence after radical prostatectomy. Neurophysiological and urodynamic diagnosis].\nL’incontinenza urinaria dopo prostatectomia radicale. Diagnosi urodinamica e neurofisiologica.\nOBJECTIVES: Urinary incontinence after radical prostatectomy presents different clinical features. We propose to clarify the etiology and the therapeutic approaches.\nMETHODS: 51 patients with urinary incontinence after radical prostatectomy underwent urodynamics, perineal EMG, perineal EMG frequency analysis and study of computerized sensory pudendal somatosensory evoked potentials.\nRESULTS: 18 patients (35%) were suffering from urinary incontinence secondary to overactive bladder, 14 patients (28%) had both an overactive bladder and a sphincter failure associated with Wallerian degenerative axonal neuropathy or segmental demyelination, 3 patients (6%) showed no significant urodynamics alterations but had signs of an incompetent sphincter neurapraxia, while 16 patients (31%) had urinary incontinence secondary to a degenerative neuropathy.\nCONCLUSIONS: Urinary incontinence after radical prostatectomy shows an etiologic diversification and therefore requires different therapeutic approaches"}

    LitEisuke

    {"project":"LitEisuke","denotations":[{"id":"T1","span":{"begin":1,"end":21},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T2","span":{"begin":204,"end":224},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T3","span":{"begin":384,"end":404},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T4","span":{"begin":625,"end":645},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T5","span":{"begin":659,"end":677},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T6","span":{"begin":709,"end":727},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T7","span":{"begin":822,"end":835},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T8","span":{"begin":982,"end":1002},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T9","span":{"begin":1056,"end":1076},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A3","pred":"#label","subj":"T3","obj":"D014549"},{"id":"A4","pred":"#label","subj":"T4","obj":"D014549"},{"id":"A9","pred":"#label","subj":"T9","obj":"D014549"},{"id":"A5","pred":"#label","subj":"T5","obj":"D053201"},{"id":"A8","pred":"#label","subj":"T8","obj":"D014549"},{"id":"A2","pred":"#label","subj":"T2","obj":"D014549"},{"id":"A7","pred":"#label","subj":"T7","obj":"D003711"},{"id":"A1","pred":"#label","subj":"T1","obj":"D014549"},{"id":"A6","pred":"#label","subj":"T6","obj":"D053201"}],"text":"[Urinary incontinence after radical prostatectomy. Neurophysiological and urodynamic diagnosis].\nL’incontinenza urinaria dopo prostatectomia radicale. Diagnosi urodinamica e neurofisiologica.\nOBJECTIVES: Urinary incontinence after radical prostatectomy presents different clinical features. We propose to clarify the etiology and the therapeutic approaches.\nMETHODS: 51 patients with urinary incontinence after radical prostatectomy underwent urodynamics, perineal EMG, perineal EMG frequency analysis and study of computerized sensory pudendal somatosensory evoked potentials.\nRESULTS: 18 patients (35%) were suffering from urinary incontinence secondary to overactive bladder, 14 patients (28%) had both an overactive bladder and a sphincter failure associated with Wallerian degenerative axonal neuropathy or segmental demyelination, 3 patients (6%) showed no significant urodynamics alterations but had signs of an incompetent sphincter neurapraxia, while 16 patients (31%) had urinary incontinence secondary to a degenerative neuropathy.\nCONCLUSIONS: Urinary incontinence after radical prostatectomy shows an etiologic diversification and therefore requires different therapeutic approaches"}

    PubmedHPO

    {"project":"PubmedHPO","denotations":[{"id":"T1","span":{"begin":204,"end":224},"obj":"HP_0000020"}],"text":"[Urinary incontinence after radical prostatectomy. Neurophysiological and urodynamic diagnosis].\nL’incontinenza urinaria dopo prostatectomia radicale. Diagnosi urodinamica e neurofisiologica.\nOBJECTIVES: Urinary incontinence after radical prostatectomy presents different clinical features. We propose to clarify the etiology and the therapeutic approaches.\nMETHODS: 51 patients with urinary incontinence after radical prostatectomy underwent urodynamics, perineal EMG, perineal EMG frequency analysis and study of computerized sensory pudendal somatosensory evoked potentials.\nRESULTS: 18 patients (35%) were suffering from urinary incontinence secondary to overactive bladder, 14 patients (28%) had both an overactive bladder and a sphincter failure associated with Wallerian degenerative axonal neuropathy or segmental demyelination, 3 patients (6%) showed no significant urodynamics alterations but had signs of an incompetent sphincter neurapraxia, while 16 patients (31%) had urinary incontinence secondary to a degenerative neuropathy.\nCONCLUSIONS: Urinary incontinence after radical prostatectomy shows an etiologic diversification and therefore requires different therapeutic approaches"}