PubMed:21129193
Annnotations
PubMed_Structured_Abstracts
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 139-564 | BACKGROUND | denotes | A novel series of methylene-substituted DIMs (C-DIMs), namely 1,1-bis(3'-indolyl)-1-(p-substituted phenyl)methanes containing t-butyl (DIM-C-pPhtBu) and phenyl (DIM-C-pPhC6H5) groups inhibit proliferation of invasive estrogen receptor-negative MDA-MB-231 and MDA-MB-453 human breast cancer cell lines with IC50 values between 1-5 uM. The main purpose of this study was to investigate the pathways of C-DIM-induced cell death. |
| T2 | 574-841 | METHODS | denotes | The effects of the C-DIMs on apoptotic, necrotic and autophagic cell death were determined using caspase inhibitors, measurement of lactate dehydrogenase release, and several markers of autophagy including Beclin and light chain associated protein 3 expression (LC3). |
| T3 | 851-1559 | RESULTS | denotes | The C-DIM compounds did not induce apoptosis and only DIM-C-pPhCF3 exhibited necrotic effects. However, treatment of MDA-MB-231 and MDA-MB-453 cells with C-DIMs resulted in accumulation of LC3-II compared to LC3-I protein, a characteristic marker of autophagy, and transient transfection of green fluorescent protein-LC3 also revealed that treatment with C-DIMs induced a redistribution of LC3 to autophagosomes after C-DIM treatment. In addition, the autofluorescent drug monodansylcadaverine (MDC), a specific autophagolysosome marker, accumulated in vacuoles after C-DIM treatment, and western blot analysis of lysates from cells treated with C-DIMs showed that the Beclin 1/Bcl-2 protein ratio increased. |
| T4 | 1572-1740 | CONCLUSIONS | denotes | The results suggest that C-DIM compounds may represent a new mechanism-based agent for treating drug-resistant ER-negative breast tumors through induction of autophagy. |
sentences
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| TextSentencer_T1 | 0-126 | Sentence | denotes | 1,1-Bis(3'-indolyl)-1-(p-substituted phenyl)methanes induce autophagic cell death in estrogen receptor negative breast cancer. |
| TextSentencer_T2 | 127-138 | Sentence | denotes | BACKGROUND: |
| TextSentencer_T3 | 139-472 | Sentence | denotes | A novel series of methylene-substituted DIMs (C-DIMs), namely 1,1-bis(3'-indolyl)-1-(p-substituted phenyl)methanes containing t-butyl (DIM-C-pPhtBu) and phenyl (DIM-C-pPhC6H5) groups inhibit proliferation of invasive estrogen receptor-negative MDA-MB-231 and MDA-MB-453 human breast cancer cell lines with IC50 values between 1-5 uM. |
| TextSentencer_T4 | 473-564 | Sentence | denotes | The main purpose of this study was to investigate the pathways of C-DIM-induced cell death. |
| TextSentencer_T5 | 565-573 | Sentence | denotes | METHODS: |
| TextSentencer_T6 | 574-841 | Sentence | denotes | The effects of the C-DIMs on apoptotic, necrotic and autophagic cell death were determined using caspase inhibitors, measurement of lactate dehydrogenase release, and several markers of autophagy including Beclin and light chain associated protein 3 expression (LC3). |
| TextSentencer_T7 | 842-850 | Sentence | denotes | RESULTS: |
| TextSentencer_T8 | 851-945 | Sentence | denotes | The C-DIM compounds did not induce apoptosis and only DIM-C-pPhCF3 exhibited necrotic effects. |
| TextSentencer_T9 | 946-1285 | Sentence | denotes | However, treatment of MDA-MB-231 and MDA-MB-453 cells with C-DIMs resulted in accumulation of LC3-II compared to LC3-I protein, a characteristic marker of autophagy, and transient transfection of green fluorescent protein-LC3 also revealed that treatment with C-DIMs induced a redistribution of LC3 to autophagosomes after C-DIM treatment. |
| TextSentencer_T10 | 1286-1559 | Sentence | denotes | In addition, the autofluorescent drug monodansylcadaverine (MDC), a specific autophagolysosome marker, accumulated in vacuoles after C-DIM treatment, and western blot analysis of lysates from cells treated with C-DIMs showed that the Beclin 1/Bcl-2 protein ratio increased. |
| TextSentencer_T11 | 1560-1571 | Sentence | denotes | CONCLUSION: |
| TextSentencer_T12 | 1572-1740 | Sentence | denotes | The results suggest that C-DIM compounds may represent a new mechanism-based agent for treating drug-resistant ER-negative breast tumors through induction of autophagy. |
| T1 | 0-126 | Sentence | denotes | 1,1-Bis(3'-indolyl)-1-(p-substituted phenyl)methanes induce autophagic cell death in estrogen receptor negative breast cancer. |
| T2 | 127-138 | Sentence | denotes | BACKGROUND: |
| T3 | 139-472 | Sentence | denotes | A novel series of methylene-substituted DIMs (C-DIMs), namely 1,1-bis(3'-indolyl)-1-(p-substituted phenyl)methanes containing t-butyl (DIM-C-pPhtBu) and phenyl (DIM-C-pPhC6H5) groups inhibit proliferation of invasive estrogen receptor-negative MDA-MB-231 and MDA-MB-453 human breast cancer cell lines with IC50 values between 1-5 uM. |
| T4 | 473-564 | Sentence | denotes | The main purpose of this study was to investigate the pathways of C-DIM-induced cell death. |
| T5 | 565-573 | Sentence | denotes | METHODS: |
| T6 | 574-841 | Sentence | denotes | The effects of the C-DIMs on apoptotic, necrotic and autophagic cell death were determined using caspase inhibitors, measurement of lactate dehydrogenase release, and several markers of autophagy including Beclin and light chain associated protein 3 expression (LC3). |
| T7 | 842-850 | Sentence | denotes | RESULTS: |
| T8 | 851-945 | Sentence | denotes | The C-DIM compounds did not induce apoptosis and only DIM-C-pPhCF3 exhibited necrotic effects. |
| T9 | 946-1285 | Sentence | denotes | However, treatment of MDA-MB-231 and MDA-MB-453 cells with C-DIMs resulted in accumulation of LC3-II compared to LC3-I protein, a characteristic marker of autophagy, and transient transfection of green fluorescent protein-LC3 also revealed that treatment with C-DIMs induced a redistribution of LC3 to autophagosomes after C-DIM treatment. |
| T10 | 1286-1559 | Sentence | denotes | In addition, the autofluorescent drug monodansylcadaverine (MDC), a specific autophagolysosome marker, accumulated in vacuoles after C-DIM treatment, and western blot analysis of lysates from cells treated with C-DIMs showed that the Beclin 1/Bcl-2 protein ratio increased. |
| T11 | 1560-1571 | Sentence | denotes | CONCLUSION: |
| T12 | 1572-1740 | Sentence | denotes | The results suggest that C-DIM compounds may represent a new mechanism-based agent for treating drug-resistant ER-negative breast tumors through induction of autophagy. |
DisGeNET
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T0 | 85-102 | gene:2099 | denotes | estrogen receptor |
| T1 | 112-125 | disease:C0678222 | denotes | breast cancer |
| T2 | 85-102 | gene:2099 | denotes | estrogen receptor |
| T3 | 112-125 | disease:C0006142 | denotes | breast cancer |
| T4 | 356-373 | gene:2099 | denotes | estrogen receptor |
| T5 | 415-428 | disease:C0678222 | denotes | breast cancer |
| T6 | 356-373 | gene:2099 | denotes | estrogen receptor |
| T7 | 415-428 | disease:C0006142 | denotes | breast cancer |
| R1 | T0 | T1 | associated_with | estrogen receptor,breast cancer |
| R2 | T2 | T3 | associated_with | estrogen receptor,breast cancer |
| R3 | T4 | T5 | associated_with | estrogen receptor,breast cancer |
| R4 | T6 | T7 | associated_with | estrogen receptor,breast cancer |
PubmedHPO
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 415-428 | HP_0003002 | denotes | breast cancer |
| T2 | 415-428 | HP_0100013 | denotes | breast cancer |
| T3 | 422-428 | HP_0002664 | denotes | cancer |
Allie
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| SS1_21129193_5_0 | 791-834 | expanded | denotes | light chain associated protein 3 expression |
| SS2_21129193_5_0 | 836-839 | abbr | denotes | LC3 |
| SS1_21129193_9_0 | 1324-1344 | expanded | denotes | monodansylcadaverine |
| SS2_21129193_9_0 | 1346-1349 | abbr | denotes | MDC |
| AE1_21129193_5_0 | SS1_21129193_5_0 | SS2_21129193_5_0 | abbreviatedTo | light chain associated protein 3 expression,LC3 |
| AE1_21129193_9_0 | SS1_21129193_9_0 | SS2_21129193_9_0 | abbreviatedTo | monodansylcadaverine,MDC |
DisGeNET5_gene_disease
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| 21129193-0#85#102#gene2099 | 85-102 | gene2099 | denotes | estrogen receptor |
| 21129193-0#112#125#diseaseC0006142 | 112-125 | diseaseC0006142 | denotes | breast cancer |
| 21129193-0#112#125#diseaseC0678222 | 112-125 | diseaseC0678222 | denotes | breast cancer |
| 85#102#gene2099112#125#diseaseC0006142 | 21129193-0#85#102#gene2099 | 21129193-0#112#125#diseaseC0006142 | associated_with | estrogen receptor,breast cancer |
| 85#102#gene2099112#125#diseaseC0678222 | 21129193-0#85#102#gene2099 | 21129193-0#112#125#diseaseC0678222 | associated_with | estrogen receptor,breast cancer |
DisGeNet-2017-sample
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T346 | 85-102 | gene:2099 | denotes | estrogen receptor |
| T347 | 112-125 | disease:C0006142 | denotes | breast cancer |
| R1 | T346 | T347 | associated_with | estrogen receptor,breast cancer |
| R2 | T346 | T347 | associated_with | estrogen receptor,breast cancer |
UBERON-AE
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| PD-UBERON-AE-B_T1 | 112-118 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |
| PD-UBERON-AE-B_T2 | 415-421 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |
| PD-UBERON-AE-B_T3 | 1695-1701 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |
performance-test
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| PD-UBERON-AE-B_T1 | 112-118 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |
| PD-UBERON-AE-B_T2 | 415-421 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |
| PD-UBERON-AE-B_T3 | 1695-1701 | http://purl.obolibrary.org/obo/UBERON_0000310 | denotes | breast |