PubMed:20829364 / 1448-1689 JSONTXT

{"project":"GlyCosmos6-Glycan-Motif-Image","denotations":[{"id":"T13","span":{"begin":143,"end":151},"obj":"Glycan_Motif"}],"attributes":[{"id":"A13","pred":"image","subj":"T13","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G89565QL"},{"id":"A14","pred":"image","subj":"T13","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G49108TO"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"Glycosmos6-GlycoEpitope","denotations":[{"id":"T7","span":{"begin":143,"end":151},"obj":"GlycoEpitope"}],"attributes":[{"id":"A7","pred":"glyco_epitope_db_id","subj":"T7","obj":"http://www.glycoepitope.jp/epitopes/EP0004"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"GlyCosmos6-Glycan-Motif-Structure","denotations":[{"id":"T13","span":{"begin":143,"end":151},"obj":"https://glytoucan.org/Structures/Glycans/G49108TO"},{"id":"T14","span":{"begin":143,"end":151},"obj":"https://glytoucan.org/Structures/Glycans/G89565QL"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"sentences","denotations":[{"id":"T10","span":{"begin":0,"end":241},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"DisGeNET5_gene_disease","denotations":[{"id":"20829364-9#168#176#gene5829","span":{"begin":168,"end":176},"obj":"gene5829"},{"id":"20829364-9#208#218#diseaseC0332996","span":{"begin":208,"end":218},"obj":"diseaseC0332996"}],"relations":[{"id":"168#176#gene5829208#218#diseaseC0332996","pred":"associated_with","subj":"20829364-9#168#176#gene5829","obj":"20829364-9#208#218#diseaseC0332996"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"NGLY1-deficiency","denotations":[{"id":"PD-NGLY1-deficiency-B_T7","span":{"begin":145,"end":151},"obj":"chem:24139"}],"namespaces":[{"prefix":"hgnc","uri":"https://www.genenames.org/data/gene-symbol-report/#!/hgnc_id/HGNC:"},{"prefix":"omim","uri":"https://www.omim.org/entry/"},{"prefix":"chem","uri":"https://pubchem.ncbi.nlm.nih.gov/compound/"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"Glycan-GlyCosmos","denotations":[{"id":"T7","span":{"begin":143,"end":151},"obj":"Glycan"}],"attributes":[{"id":"A7","pred":"glycosmos_id","subj":"T7","obj":"https://glycosmos.org/glycans/show/G49108TO"},{"id":"A14","pred":"image","subj":"T7","obj":"https://api.glycosmos.org/wurcs2image/latest/png/binary/G49108TO"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"GlyCosmos-GlycoEpitope","denotations":[{"id":"T7","span":{"begin":143,"end":151},"obj":"http://purl.jp/bio/12/glyco/glycan#Glycan_epitope"}],"attributes":[{"id":"A7","pred":"glycoepitope_id","subj":"T7","obj":"http://www.glycoepitope.jp/epitopes/EP0004"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"sentences","denotations":[{"id":"T10","span":{"begin":0,"end":241},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"GlyCosmos15-Sentences","blocks":[{"id":"T10","span":{"begin":0,"end":241},"obj":"Sentence"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"GlyCosmos15-Glycan","denotations":[{"id":"T7","span":{"begin":143,"end":151},"obj":"Glycan"}],"attributes":[{"id":"A7","pred":"glycosmos_id","subj":"T7","obj":"https://glycosmos.org/glycans/show/G49108TO"},{"id":"A14","pred":"image","subj":"T7","obj":"https://api.glycosmos.org/wurcs2image/latest/png/binary/G49108TO"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}

{"project":"GlyCosmos15-GlycoEpitope","denotations":[{"id":"T7","span":{"begin":143,"end":151},"obj":"http://purl.jp/bio/12/glyco/glycan#Glycan_epitope"}],"attributes":[{"id":"A7","pred":"glycoepitope_id","subj":"T7","obj":"http://www.glycoepitope.jp/epitopes/EP0004"}],"text":"Together these observations suggest that murine pancreatic β cells may have restricted actin organization upon GlcN treatment by virtue of the O-GlcNAc modification of paxillin, which can be antagonized by a persistent cell adhesion process."}