> top > docs > PubMed:20716292 > annotations

PubMed:20716292 JSONTXT

Annnotations TAB JSON ListView MergeView

123123123

Id Subject Object Predicate Lexical cue
T1 580-590 B-Negative_Function denotes background
T2 591-593 B-Regulation denotes is
T3 758-772 B-Cell_Physiological_Activity denotes responsiveness
T4 918-932 B-Cell_Physiological_Activity denotes responsiveness
T5 1142-1149 B-Variation denotes insight
T6 1215-1229 B-Negative_Function denotes susceptibility

Wangshuguang

Id Subject Object Predicate Lexical cue
T1 580-590 B-Negative_Function denotes background
T2 591-593 B-Regulation denotes is
T3 758-772 B-Cell_Physiological_Activity denotes responsiveness
T4 918-932 B-Cell_Physiological_Activity denotes responsiveness
T5 1142-1149 B-Variation denotes insight
T6 1215-1229 B-Negative_Function denotes susceptibility

Inflammaging

Id Subject Object Predicate Lexical cue
T1 0-67 Sentence denotes Genetic mechanisms of susceptibility to ozone-induced lung disease.
T2 68-174 Sentence denotes Environmental oxidants remain a major public health concern in industrialized cities throughout the world.
T3 175-373 Sentence denotes Population and epidemiological studies have associated oxidant air pollutants with morbidity and mortality outcomes, and underscore the important detrimental effects of these pollutants on the lung.
T4 374-629 Sentence denotes Interindividual variation in pulmonary responses to air pollutants suggests that some subpopulations are at increased risk to detrimental effects of pollutant exposure, and it has become clear that genetic background is an important susceptibility factor.
T5 630-809 Sentence denotes A number of genetics and genomics tools have recently emerged to enable identification of genes that contribute to differential responsiveness to oxidants, including ozone (O(3)).
T6 810-1018 Sentence denotes Integrative omics approaches have been applied in inbred mice to identify genes that determine differential responsiveness to O(3)-induced injury and inflammation, including Tnf, Tlr4, and MHC Class II genes.
T7 1019-1242 Sentence denotes Combined investigations across cell models, inbred mice, and humans have provided, and will continue to provide, important insight to understanding genetic factors that contribute to differential susceptibility to oxidants.
T1 0-67 Sentence denotes Genetic mechanisms of susceptibility to ozone-induced lung disease.
T2 68-174 Sentence denotes Environmental oxidants remain a major public health concern in industrialized cities throughout the world.
T3 175-373 Sentence denotes Population and epidemiological studies have associated oxidant air pollutants with morbidity and mortality outcomes, and underscore the important detrimental effects of these pollutants on the lung.
T4 374-629 Sentence denotes Interindividual variation in pulmonary responses to air pollutants suggests that some subpopulations are at increased risk to detrimental effects of pollutant exposure, and it has become clear that genetic background is an important susceptibility factor.
T5 630-809 Sentence denotes A number of genetics and genomics tools have recently emerged to enable identification of genes that contribute to differential responsiveness to oxidants, including ozone (O(3)).
T6 810-1018 Sentence denotes Integrative omics approaches have been applied in inbred mice to identify genes that determine differential responsiveness to O(3)-induced injury and inflammation, including Tnf, Tlr4, and MHC Class II genes.
T7 1019-1242 Sentence denotes Combined investigations across cell models, inbred mice, and humans have provided, and will continue to provide, important insight to understanding genetic factors that contribute to differential susceptibility to oxidants.