PubMed:19318518 / 818-1207 JSONTXT

{"project":"PMID_GLOBAL","denotations":[{"id":"T4","span":{"begin":200,"end":202},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A4","pred":"mondo_id","subj":"T4","obj":"0002013"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"sentences","denotations":[{"id":"TextSentencer_T5","span":{"begin":0,"end":389},"obj":"Sentence"},{"id":"T5","span":{"begin":0,"end":389},"obj":"Sentence"},{"id":"T5","span":{"begin":0,"end":389},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"GlycoBiology-FMA","denotations":[{"id":"_T19","span":{"begin":271,"end":275},"obj":"FMAID:198663"}],"namespaces":[{"prefix":"FMAID","uri":"http://purl.org/sig/ont/fma/fma"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"GlycoBiology-NCBITAXON","denotations":[{"id":"T47","span":{"begin":253,"end":260},"obj":"http://purl.bioontology.org/ontology/STY/T033"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"GO-CC","denotations":[{"id":"T4","span":{"begin":99,"end":103},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T7","span":{"begin":99,"end":108},"obj":"http://purl.obolibrary.org/obo/GO_0005618"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"Lectin-Jamboree-Sentence","blocks":[{"id":"T5","span":{"begin":0,"end":389},"obj":"Sentence"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}

{"project":"Anatomy-UBERON","denotations":[{"id":"T5","span":{"begin":104,"end":108},"obj":"Body_part"}],"attributes":[{"id":"A5","pred":"uberon_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/UBERON_0000060"}],"text":"We demonstrate that the inactivation of pimB in M.tb does not abolish the production of any of its cell wall mannosylated lipoglycans; however, it results in a quantitative decrease in the ManLAM and LM content without affecting higher order PIMs. This finding indicates gene redundancy or the possibility of an alternative biosynthetic pathway that may compensate for the PimB deficiency."}