PubMed:18412970 / 0-284 JSONTXT

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    c_corpus

    {"project":"c_corpus","denotations":[{"id":"T1","span":{"begin":33,"end":43},"obj":"P42858"},{"id":"T2","span":{"begin":33,"end":43},"obj":"P42859"},{"id":"T3","span":{"begin":33,"end":43},"obj":"P51111"},{"id":"T4","span":{"begin":33,"end":43},"obj":"PR:000008840"},{"id":"T5","span":{"begin":33,"end":43},"obj":"P51112"},{"id":"T6","span":{"begin":85,"end":95},"obj":"P42858"},{"id":"T7","span":{"begin":85,"end":95},"obj":"P42859"},{"id":"T8","span":{"begin":85,"end":95},"obj":"P51111"},{"id":"T9","span":{"begin":85,"end":95},"obj":"PR:000008840"},{"id":"T10","span":{"begin":85,"end":95},"obj":"P51112"},{"id":"T11","span":{"begin":101,"end":106},"obj":"10090"},{"id":"T12","span":{"begin":101,"end":106},"obj":"D051379"},{"id":"T13","span":{"begin":107,"end":116},"obj":"P02301"},{"id":"T14","span":{"begin":107,"end":116},"obj":"PR:000029532"},{"id":"T15","span":{"begin":146,"end":159},"obj":"C097188"},{"id":"T16","span":{"begin":146,"end":159},"obj":"C097188"},{"id":"T17","span":{"begin":173,"end":183},"obj":"P42858"},{"id":"T18","span":{"begin":173,"end":183},"obj":"P42859"},{"id":"T19","span":{"begin":173,"end":183},"obj":"P51111"},{"id":"T20","span":{"begin":173,"end":183},"obj":"PR:000008840"},{"id":"T21","span":{"begin":173,"end":183},"obj":"P51112"},{"id":"T22","span":{"begin":185,"end":188},"obj":"PR:P51111"},{"id":"T23","span":{"begin":185,"end":188},"obj":"PR:000015189"},{"id":"T24","span":{"begin":185,"end":188},"obj":"PR:Q76P24"},{"id":"T25","span":{"begin":185,"end":188},"obj":"PR:P31645"},{"id":"T26","span":{"begin":185,"end":188},"obj":"PR:P42859"},{"id":"T27","span":{"begin":185,"end":188},"obj":"PR:Q60857"},{"id":"T28","span":{"begin":185,"end":188},"obj":"PR:P42858"},{"id":"T29","span":{"begin":185,"end":188},"obj":"PR:000008840"},{"id":"T34","span":{"begin":190,"end":197},"obj":"SO:0000104"},{"id":"T31","span":{"begin":190,"end":197},"obj":"CHEBI:36080"},{"id":"T32","span":{"begin":190,"end":197},"obj":"CHEBI:11122"},{"id":"T33","span":{"begin":190,"end":197},"obj":"PR:000000001"},{"id":"T30","span":{"begin":190,"end":197},"obj":"GO:0003675"},{"id":"T39","span":{"begin":212,"end":232},"obj":"D006816"},{"id":"T40","span":{"begin":212,"end":232},"obj":"D006816"},{"id":"T43","span":{"begin":239,"end":242},"obj":"PR:P51111"},{"id":"T44","span":{"begin":239,"end":242},"obj":"PR:000015189"},{"id":"T45","span":{"begin":239,"end":242},"obj":"PR:Q76P24"},{"id":"T46","span":{"begin":239,"end":242},"obj":"PR:P31645"},{"id":"T47","span":{"begin":239,"end":242},"obj":"PR:P42859"},{"id":"T48","span":{"begin":239,"end":242},"obj":"PR:Q60857"},{"id":"T49","span":{"begin":239,"end":242},"obj":"PR:P42858"},{"id":"T50","span":{"begin":239,"end":242},"obj":"PR:000008840"},{"id":"T51","span":{"begin":259,"end":263},"obj":"SO:0000704"},{"id":"T52","span":{"begin":267,"end":275},"obj":"SO:0000159"}],"text":"Elucidating a normal function of huntingtin by functional and microarray analysis of huntingtin-null mouse embryonic fibroblasts.\nBACKGROUND: The polyglutamine expansion in huntingtin (Htt) protein is a cause of Huntington's disease (HD). Htt is an essential gene as deletion of the m"}

    PMID_GLOBAL

    {"project":"PMID_GLOBAL","denotations":[{"id":"T1","span":{"begin":212,"end":232},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T2","span":{"begin":234,"end":236},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A1","pred":"mondo_id","subj":"T1","obj":"0007739"},{"id":"A2","pred":"mondo_id","subj":"T2","obj":"0007739"}],"text":"Elucidating a normal function of huntingtin by functional and microarray analysis of huntingtin-null mouse embryonic fibroblasts.\nBACKGROUND: The polyglutamine expansion in huntingtin (Htt) protein is a cause of Huntington's disease (HD). Htt is an essential gene as deletion of the m"}

    UseCases_ArguminSci_Discourse

    {"project":"UseCases_ArguminSci_Discourse","denotations":[{"id":"T1","span":{"begin":0,"end":129},"obj":"DRI_Background"},{"id":"T2","span":{"begin":142,"end":238},"obj":"DRI_Challenge"}],"text":"Elucidating a normal function of huntingtin by functional and microarray analysis of huntingtin-null mouse embryonic fibroblasts.\nBACKGROUND: The polyglutamine expansion in huntingtin (Htt) protein is a cause of Huntington's disease (HD). Htt is an essential gene as deletion of the m"}