| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-93 |
Sentence |
denotes |
Identification of the functional role of peroxiredoxin 6 in the progression of breast cancer. |
| TextSentencer_T2 |
94-107 |
Sentence |
denotes |
INTRODUCTION: |
| TextSentencer_T3 |
108-199 |
Sentence |
denotes |
The molecular mechanisms involved in breast cancer metastasis still remain unclear to date. |
| TextSentencer_T4 |
200-375 |
Sentence |
denotes |
In our previous study, differential expression of peroxiredoxin 6 was found between the highly metastatic MDA-MB-435HM cells and their parental counterparts, MDA-MB-435 cells. |
| TextSentencer_T5 |
376-543 |
Sentence |
denotes |
In this study, we investigated the effects of peroxiredoxin 6 on the proliferation and metastatic potential of human breast cancer cells and their potential mechanism. |
| TextSentencer_T6 |
544-552 |
Sentence |
denotes |
METHODS: |
| TextSentencer_T7 |
553-686 |
Sentence |
denotes |
Expression of peroxiredoxin 6 in the highly metastatic MDA-MB-231HM cells was investigated by RT-PCR, real-time PCR and western blot. |
| TextSentencer_T8 |
687-823 |
Sentence |
denotes |
A recombinant expression plasmid of the human peroxiredoxin 6 gene was constructed and transfected into MDA-MB-231 and MDA-MB-435 cells. |
| TextSentencer_T9 |
824-1126 |
Sentence |
denotes |
The effects of peroxiredoxin 6 on the proliferation and invasion of MDA-MB-231 and MDA-MB-435 cells were investigated by the Cell Counting Kit-8 method, colony-formation assay, adhesion assay, flow cytometry and invasion assay in vitro. miRNA was used to downregulate the expression of peroxiredoxin 6. |
| TextSentencer_T10 |
1127-1240 |
Sentence |
denotes |
Genes related to the invasion and metastasis of cancer were determined by RT-PCR, real-time PCR and western blot. |
| TextSentencer_T11 |
1241-1403 |
Sentence |
denotes |
The tumorigenicity and spontaneously metastatic capability regulated by peroxiredoxin 6 were determined using an orthotopic xenograft tumor model in athymic mice. |
| TextSentencer_T12 |
1404-1412 |
Sentence |
denotes |
RESULTS: |
| TextSentencer_T13 |
1413-1525 |
Sentence |
denotes |
Overexpression of peroxiredoxin 6 in MDA-MB-231HM cells compared with their parental counterparts was confirmed. |
| TextSentencer_T14 |
1526-1630 |
Sentence |
denotes |
Upregulation of peroxiredoxin 6 enhanced the in vitro proliferation and invasion of breast cancer cells. |
| TextSentencer_T15 |
1631-1953 |
Sentence |
denotes |
The enhancement was associated with decreasing levels of tissue inhibitor of matrix metalloproteinase (TIMP)-2 and increasing levels of the urokinase-type plasminogen activator receptor (uPAR), Ets-1 (E26 transformation-specific-1), matrix metalloproteinase (MMP)-9 and RhoC (ras homolog gene family, member C) expression. |
| TextSentencer_T16 |
1954-2033 |
Sentence |
denotes |
The results were further demonstrated by RNA interference experiments in vitro. |
| TextSentencer_T17 |
2034-2199 |
Sentence |
denotes |
In an in vivo study, we also demonstrated that peroxiredoxin 6-transfected breast cancer cells grew much faster and had more pulmonary metastases than control cells. |
| TextSentencer_T18 |
2200-2311 |
Sentence |
denotes |
By contrast, peroxiredoxin 6 knockdown breast cancer cells grew more slowly and had fewer pulmonary metastases. |
| TextSentencer_T19 |
2312-2471 |
Sentence |
denotes |
Effects similar to those of peroxiredoxin 6 on the uPAR, Ets-1, MMP-9, RhoC and TIMP-2 expression observed in in vitro studies were found in the in vivo study. |
| TextSentencer_T20 |
2472-2483 |
Sentence |
denotes |
CONCLUSION: |
| TextSentencer_T21 |
2484-2703 |
Sentence |
denotes |
Overexpression of peroxiredoxin 6 leads to a more invasive phenotype and metastatic potential in human breast cancer, at least in part, through regulation of the levels of uPAR, Ets-1, MMP-9, RhoC and TIMP-2 expression. |
| T1 |
0-93 |
Sentence |
denotes |
Identification of the functional role of peroxiredoxin 6 in the progression of breast cancer. |
| T2 |
94-107 |
Sentence |
denotes |
INTRODUCTION: |
| T3 |
108-199 |
Sentence |
denotes |
The molecular mechanisms involved in breast cancer metastasis still remain unclear to date. |
| T4 |
200-375 |
Sentence |
denotes |
In our previous study, differential expression of peroxiredoxin 6 was found between the highly metastatic MDA-MB-435HM cells and their parental counterparts, MDA-MB-435 cells. |
| T5 |
376-543 |
Sentence |
denotes |
In this study, we investigated the effects of peroxiredoxin 6 on the proliferation and metastatic potential of human breast cancer cells and their potential mechanism. |
| T6 |
544-552 |
Sentence |
denotes |
METHODS: |
| T7 |
553-686 |
Sentence |
denotes |
Expression of peroxiredoxin 6 in the highly metastatic MDA-MB-231HM cells was investigated by RT-PCR, real-time PCR and western blot. |
| T8 |
687-823 |
Sentence |
denotes |
A recombinant expression plasmid of the human peroxiredoxin 6 gene was constructed and transfected into MDA-MB-231 and MDA-MB-435 cells. |
| T9 |
824-1126 |
Sentence |
denotes |
The effects of peroxiredoxin 6 on the proliferation and invasion of MDA-MB-231 and MDA-MB-435 cells were investigated by the Cell Counting Kit-8 method, colony-formation assay, adhesion assay, flow cytometry and invasion assay in vitro. miRNA was used to downregulate the expression of peroxiredoxin 6. |
| T10 |
1127-1240 |
Sentence |
denotes |
Genes related to the invasion and metastasis of cancer were determined by RT-PCR, real-time PCR and western blot. |
| T11 |
1241-1403 |
Sentence |
denotes |
The tumorigenicity and spontaneously metastatic capability regulated by peroxiredoxin 6 were determined using an orthotopic xenograft tumor model in athymic mice. |
| T12 |
1404-1412 |
Sentence |
denotes |
RESULTS: |
| T13 |
1413-1525 |
Sentence |
denotes |
Overexpression of peroxiredoxin 6 in MDA-MB-231HM cells compared with their parental counterparts was confirmed. |
| T14 |
1526-1630 |
Sentence |
denotes |
Upregulation of peroxiredoxin 6 enhanced the in vitro proliferation and invasion of breast cancer cells. |
| T15 |
1631-1953 |
Sentence |
denotes |
The enhancement was associated with decreasing levels of tissue inhibitor of matrix metalloproteinase (TIMP)-2 and increasing levels of the urokinase-type plasminogen activator receptor (uPAR), Ets-1 (E26 transformation-specific-1), matrix metalloproteinase (MMP)-9 and RhoC (ras homolog gene family, member C) expression. |
| T16 |
1954-2033 |
Sentence |
denotes |
The results were further demonstrated by RNA interference experiments in vitro. |
| T17 |
2034-2199 |
Sentence |
denotes |
In an in vivo study, we also demonstrated that peroxiredoxin 6-transfected breast cancer cells grew much faster and had more pulmonary metastases than control cells. |
| T18 |
2200-2311 |
Sentence |
denotes |
By contrast, peroxiredoxin 6 knockdown breast cancer cells grew more slowly and had fewer pulmonary metastases. |
| T19 |
2312-2471 |
Sentence |
denotes |
Effects similar to those of peroxiredoxin 6 on the uPAR, Ets-1, MMP-9, RhoC and TIMP-2 expression observed in in vitro studies were found in the in vivo study. |
| T20 |
2472-2483 |
Sentence |
denotes |
CONCLUSION: |
| T21 |
2484-2703 |
Sentence |
denotes |
Overexpression of peroxiredoxin 6 leads to a more invasive phenotype and metastatic potential in human breast cancer, at least in part, through regulation of the levels of uPAR, Ets-1, MMP-9, RhoC and TIMP-2 expression. |