PubMed:17277312
Annnotations
Inflammaging
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 0-76 | Sentence | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. |
| T2 | 77-241 | Sentence | denotes | Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. |
| T3 | 242-311 | Sentence | denotes | However, how cytokine signals mediate THi differentiation is unclear. |
| T4 | 312-428 | Sentence | denotes | We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. |
| T5 | 429-511 | Sentence | denotes | STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. |
| T6 | 512-651 | Sentence | denotes | A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. |
| T7 | 652-952 | Sentence | denotes | Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). |
| T8 | 953-1142 | Sentence | denotes | Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression. |
| T1 | 0-76 | Sentence | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. |
| T2 | 77-241 | Sentence | denotes | Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. |
| T3 | 242-311 | Sentence | denotes | However, how cytokine signals mediate THi differentiation is unclear. |
| T4 | 312-428 | Sentence | denotes | We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. |
| T5 | 429-511 | Sentence | denotes | STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. |
| T6 | 512-651 | Sentence | denotes | A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. |
| T7 | 652-952 | Sentence | denotes | Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). |
| T8 | 953-1142 | Sentence | denotes | Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression. |
sentences
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 0-76 | Sentence | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. |
| T2 | 77-241 | Sentence | denotes | Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. |
| T3 | 242-311 | Sentence | denotes | However, how cytokine signals mediate THi differentiation is unclear. |
| T4 | 312-428 | Sentence | denotes | We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. |
| T5 | 429-511 | Sentence | denotes | STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. |
| T6 | 512-651 | Sentence | denotes | A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. |
| T7 | 652-952 | Sentence | denotes | Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). |
| T8 | 953-1142 | Sentence | denotes | Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression. |
PubmedHPO
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T1 | 514-525 | HP_0000752 | denotes | hyperactive |
2015-BEL-Sample-2
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| BEL:20062042 | 807-951 | p(MGI:Stat3) decreases r(MGI:Foxp3) | denotes | STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3) |
| BEL:20071554 | 0-1141 | tscript(p(MGI:Stat3)) decreases r(MGI:Gata3) | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. However, how cytokine signals mediate THi differentiation is unclear. We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression |
| BEL:20071576 | 0-1141 | tscript(p(MGI:Stat3)) increases r(MGI:Rorc) | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. However, how cytokine signals mediate THi differentiation is unclear. We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression |
| BEL:20071582 | 0-1141 | tscript(p(MGI:Stat3)) decreases r(MGI:Tbx21) | denotes | STAT3 regulates cytokine-mediated generation of inflammatory helper T cells. Interleukin-17 (IL-17)-producing helper T (TH) cells, named as TH(IL-17), TH17, or inflammatory TH (THi), have been recently identified as a novel effector lineage. However, how cytokine signals mediate THi differentiation is unclear. We found that IL-6 functioned to up-regulate IL-23R and that IL-23 synergized with IL-6 in promoting THi generation. STAT3, activated by both IL-6 and IL-23, plays a critical role in THi development. A hyperactive form of STAT3 promoted THi development, whereas this differentiation process was greatly impaired in STAT3-deficient T cells. Moreover, STAT3 regulated the expression of retinoic acid receptor-related orphan receptor gamma-T (RORgamma t), a THi-specific transcriptional regulator; STAT3 deficiency impaired RORgamma t expression and led to elevated expression of T-box expressed in T cells (T-bet) and Forkhead box P3 (Foxp3). Our data thus demonstrate a pathway whereby cytokines regulate THi differentiation through a selective STAT transcription factor that functions to regulate lineage-specific gene expression |
mondo_disease
| Id | Subject | Object | Predicate | Lexical cue | mondo_id |
|---|---|---|---|---|---|
| T1 | 807-823 | Disease | denotes | STAT3 deficiency | http://purl.obolibrary.org/obo/MONDO_0007818 |
CL-cell
| Id | Subject | Object | Predicate | Lexical cue | cl_id |
|---|---|---|---|---|---|
| T1 | 61-75 | Cell | denotes | helper T cells | http://purl.obolibrary.org/obo/CL:0000912 |
| T2 | 643-650 | Cell | denotes | T cells | http://purl.obolibrary.org/obo/CL:0000084 |
| T3 | 908-915 | Cell | denotes | T cells | http://purl.obolibrary.org/obo/CL:0000084 |