PubMed:16410744 / 871-1128 JSONTXT

{"project":"LitCoin-sentences","denotations":[{"id":"T8","span":{"begin":0,"end":257},"obj":"Sentence"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-entities","denotations":[{"id":"2718","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"},{"id":"2719","span":{"begin":93,"end":97},"obj":"GeneOrGeneProduct"},{"id":"2720","span":{"begin":116,"end":119},"obj":"GeneOrGeneProduct"},{"id":"2721","span":{"begin":189,"end":193},"obj":"GeneOrGeneProduct"},{"id":"2722","span":{"begin":208,"end":211},"obj":"GeneOrGeneProduct"},{"id":"2723","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A14","pred":"db_id","subj":"2718","obj":"MESH:D005909"},{"id":"A15","pred":"db_id","subj":"2719","obj":"NCBIGene:1956"},{"id":"A16","pred":"db_id","subj":"2720","obj":"NCBIGene:1029"},{"id":"A17","pred":"db_id","subj":"2721","obj":"NCBIGene:7157"},{"id":"A18","pred":"db_id","subj":"2722","obj":"NCBIGene:1029"},{"id":"A19","pred":"db_id","subj":"2723","obj":"MESH:D005909"}],"namespaces":[{"prefix":"_base","uri":"https://w3id.org/biolink/vocab/"},{"prefix":"MESH","uri":"http://id.nlm.nih.gov/mesh/"},{"prefix":"NCBITaxon","uri":"https://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id="},{"prefix":"NCBIGene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"OMIM","uri":"https://www.omim.org/entry/"},{"prefix":"DBSNP","uri":"https://www.ncbi.nlm.nih.gov/snp/"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin_Mondo","denotations":[{"id":"T7","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T8","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A7","pred":"mondo_id","subj":"T7","obj":"0018177"},{"id":"A8","pred":"mondo_id","subj":"T8","obj":"0018177"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-GeneOrGeneProduct-v2","denotations":[{"id":"T7","span":{"begin":93,"end":97},"obj":"GeneOrGeneProduct"},{"id":"T8","span":{"begin":189,"end":193},"obj":"GeneOrGeneProduct"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-GeneOrGeneProduct-v0","denotations":[{"id":"T21","span":{"begin":93,"end":97},"obj":"GeneOrGeneProduct"},{"id":"T22","span":{"begin":189,"end":193},"obj":"GeneOrGeneProduct"},{"id":"T23","span":{"begin":194,"end":203},"obj":"GeneOrGeneProduct"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-Disease-MeSH","denotations":[{"id":"T8","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T9","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A8","pred":"originalLabel","subj":"T8","obj":"D005909"},{"id":"A9","pred":"originalLabel","subj":"T9","obj":"D005909"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-GeneOrGeneProduct-v3","denotations":[{"id":"T4","span":{"begin":93,"end":97},"obj":"GeneOrGeneProduct"},{"id":"T5","span":{"begin":189,"end":193},"obj":"GeneOrGeneProduct"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-MeSH-Disease-2","denotations":[{"id":"T9","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T10","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A9","pred":"ID:","subj":"T9","obj":"D005909"},{"id":"A10","pred":"ID:","subj":"T10","obj":"D005909"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-MONDO_bioort2019","denotations":[{"id":"T8","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T9","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A8","pred":"#label","subj":"T8","obj":"D005909"},{"id":"A9","pred":"#label","subj":"T9","obj":"D005909"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"LitCoin-training-merged","denotations":[{"id":"T5","span":{"begin":189,"end":193},"obj":"GeneOrGeneProduct"},{"id":"T4","span":{"begin":93,"end":97},"obj":"GeneOrGeneProduct"},{"id":"T82858","span":{"begin":234,"end":247},"obj":"DiseaseOrPhenotypicFeature"},{"id":"T38489","span":{"begin":27,"end":40},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A9","pred":"#label","subj":"T82858","obj":"D005909"},{"id":"A8","pred":"#label","subj":"T38489","obj":"D005909"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"PubmedHPO","denotations":[{"id":"T7","span":{"begin":27,"end":40},"obj":"HP_0100843"},{"id":"T8","span":{"begin":234,"end":247},"obj":"HP_0100843"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}

{"project":"DisGeNET5_gene_disease","denotations":[{"id":"16410744-7#116#119#gene1029","span":{"begin":116,"end":119},"obj":"gene1029"},{"id":"16410744-7#189#193#gene7157","span":{"begin":189,"end":193},"obj":"gene7157"},{"id":"16410744-7#208#211#gene1029","span":{"begin":208,"end":211},"obj":"gene1029"},{"id":"16410744-7#27#40#diseaseC0017636","span":{"begin":27,"end":40},"obj":"diseaseC0017636"},{"id":"16410744-7#234#247#diseaseC0017636","span":{"begin":234,"end":247},"obj":"diseaseC0017636"},{"id":"16410744-7#27#40#diseaseC0017636","span":{"begin":27,"end":40},"obj":"diseaseC0017636"},{"id":"16410744-7#234#247#diseaseC0017636","span":{"begin":234,"end":247},"obj":"diseaseC0017636"},{"id":"16410744-7#27#40#diseaseC0017636","span":{"begin":27,"end":40},"obj":"diseaseC0017636"},{"id":"16410744-7#234#247#diseaseC0017636","span":{"begin":234,"end":247},"obj":"diseaseC0017636"}],"relations":[{"id":"116#119#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"116#119#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"116#119#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"116#119#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"116#119#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"116#119#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#116#119#gene1029","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"189#193#gene715727#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"189#193#gene7157234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"189#193#gene715727#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"189#193#gene7157234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"189#193#gene715727#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"189#193#gene7157234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#189#193#gene7157","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"208#211#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"208#211#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"208#211#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"208#211#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#234#247#diseaseC0017636"},{"id":"208#211#gene102927#40#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#27#40#diseaseC0017636"},{"id":"208#211#gene1029234#247#diseaseC0017636","pred":"associated_with","subj":"16410744-7#208#211#gene1029","obj":"16410744-7#234#247#diseaseC0017636"}],"text":"As previously observed for glioblastomas in Europe, there was a positive association between EGFR amplification and p16 deletion (p=0.009), whereas there was an inverse association between TP53 mutations and p16 deletion (p=0.049) in glioblastomas in Japan."}