PubMed:16096344 JSONTXT

{"project":"Glycan-Motif","denotations":[{"id":"T1","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T2","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T3","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T4","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T5","span":{"begin":218,"end":239},"obj":"https://glytoucan.org/Structures/Glycans/G27025MB"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlyCosmos600-Glycan-Motif-Structure","denotations":[{"id":"T1","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T2","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T3","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T4","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T5","span":{"begin":218,"end":239},"obj":"https://glytoucan.org/Structures/Glycans/G27025MB"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlyCosmos600-CLO","denotations":[{"id":"T1","span":{"begin":54,"end":59},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T2","span":{"begin":142,"end":147},"obj":"http://purl.obolibrary.org/obo/CLO_0007836"},{"id":"T3","span":{"begin":318,"end":333},"obj":"http://purl.obolibrary.org/obo/CL_0000451"},{"id":"T4","span":{"begin":582,"end":587},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T5","span":{"begin":793,"end":798},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T6","span":{"begin":814,"end":819},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T7","span":{"begin":835,"end":840},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T8","span":{"begin":915,"end":920},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T9","span":{"begin":968,"end":973},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T10","span":{"begin":1015,"end":1020},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T11","span":{"begin":1115,"end":1120},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T12","span":{"begin":1199,"end":1204},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T13","span":{"begin":1235,"end":1240},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T14","span":{"begin":1279,"end":1284},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T15","span":{"begin":1430,"end":1435},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlyCosmos6-Glycan-Motif-Image","denotations":[{"id":"T1","span":{"begin":101,"end":110},"obj":"Glycan_Motif"},{"id":"T3","span":{"begin":204,"end":213},"obj":"Glycan_Motif"},{"id":"T5","span":{"begin":218,"end":239},"obj":"Glycan_Motif"}],"attributes":[{"id":"A1","pred":"image","subj":"T1","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G68158BT"},{"id":"A2","pred":"image","subj":"T1","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G65889KE"},{"id":"A3","pred":"image","subj":"T3","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G68158BT"},{"id":"A4","pred":"image","subj":"T3","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G65889KE"},{"id":"A5","pred":"image","subj":"T5","obj":"https://api.glycosmos.org/wurcs2image/0.10.0/png/binary/G27025MB"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"Glycosmos6-MAT","denotations":[{"id":"T1","span":{"begin":74,"end":86},"obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"T2","span":{"begin":148,"end":155},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T3","span":{"begin":453,"end":458},"obj":"http://purl.obolibrary.org/obo/MAT_0000029"},{"id":"T4","span":{"begin":468,"end":475},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T5","span":{"begin":635,"end":642},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T6","span":{"begin":892,"end":899},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T7","span":{"begin":942,"end":953},"obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"T8","span":{"begin":1147,"end":1154},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T9","span":{"begin":1307,"end":1314},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T10","span":{"begin":1331,"end":1338},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"T11","span":{"begin":1527,"end":1534},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlyCosmos6-Glycan-Motif-Structure","denotations":[{"id":"T1","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T2","span":{"begin":101,"end":110},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T3","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G65889KE"},{"id":"T4","span":{"begin":204,"end":213},"obj":"https://glytoucan.org/Structures/Glycans/G68158BT"},{"id":"T5","span":{"begin":218,"end":239},"obj":"https://glytoucan.org/Structures/Glycans/G27025MB"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"uniprot-mouse","denotations":[{"id":"T1","span":{"begin":392,"end":396},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T2","span":{"begin":425,"end":429},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T3","span":{"begin":436,"end":440},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T4","span":{"begin":692,"end":696},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T5","span":{"begin":748,"end":752},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T6","span":{"begin":884,"end":888},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T7","span":{"begin":1127,"end":1131},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T8","span":{"begin":1139,"end":1143},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T9","span":{"begin":1299,"end":1303},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T10","span":{"begin":1323,"end":1327},"obj":"http://www.uniprot.org/uniprot/P49300"},{"id":"T11","span":{"begin":414,"end":419},"obj":"http://www.uniprot.org/uniprot/Q6VSG4"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlycoBiology-NCBITAXON","denotations":[{"id":"T1","span":{"begin":54,"end":59},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T2","span":{"begin":328,"end":333},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T3","span":{"begin":582,"end":587},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T4","span":{"begin":651,"end":655},"obj":"http://purl.bioontology.org/ontology/NCBITAXON/82391"},{"id":"T5","span":{"begin":793,"end":798},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T6","span":{"begin":814,"end":819},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T7","span":{"begin":835,"end":840},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T8","span":{"begin":915,"end":920},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T9","span":{"begin":963,"end":967},"obj":"http://purl.bioontology.org/ontology/NCBITAXON/9973"},{"id":"T10","span":{"begin":968,"end":973},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T11","span":{"begin":1015,"end":1020},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T12","span":{"begin":1115,"end":1120},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T13","span":{"begin":1199,"end":1204},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T14","span":{"begin":1235,"end":1240},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T15","span":{"begin":1279,"end":1284},"obj":"http://purl.bioontology.org/ontology/STY/T025"},{"id":"T16","span":{"begin":1430,"end":1435},"obj":"http://purl.bioontology.org/ontology/STY/T025"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GO-BP","denotations":[{"id":"T1","span":{"begin":54,"end":73},"obj":"http://purl.obolibrary.org/obo/GO_0048468"},{"id":"T2","span":{"begin":54,"end":80},"obj":"http://purl.obolibrary.org/obo/GO_0014032"},{"id":"T3","span":{"begin":63,"end":73},"obj":"http://purl.obolibrary.org/obo/GO_0032502"},{"id":"T4","span":{"begin":624,"end":634},"obj":"http://purl.obolibrary.org/obo/GO_0032502"},{"id":"T5","span":{"begin":1495,"end":1506},"obj":"http://purl.obolibrary.org/obo/GO_0032502"},{"id":"T6","span":{"begin":63,"end":86},"obj":"http://purl.obolibrary.org/obo/GO_0021915"},{"id":"T7","span":{"begin":414,"end":419},"obj":"http://purl.obolibrary.org/obo/GO_0000747"},{"id":"T8","span":{"begin":414,"end":419},"obj":"http://purl.obolibrary.org/obo/GO_0007618"},{"id":"T9","span":{"begin":414,"end":419},"obj":"http://purl.obolibrary.org/obo/GO_0009291"},{"id":"T10","span":{"begin":414,"end":419},"obj":"http://purl.obolibrary.org/obo/GO_1990277"},{"id":"T11","span":{"begin":559,"end":587},"obj":"http://purl.obolibrary.org/obo/GO_0043277"},{"id":"T12","span":{"begin":1407,"end":1435},"obj":"http://purl.obolibrary.org/obo/GO_0043277"},{"id":"T13","span":{"begin":559,"end":587},"obj":"http://purl.obolibrary.org/obo/GO_2000425"},{"id":"T14","span":{"begin":1407,"end":1435},"obj":"http://purl.obolibrary.org/obo/GO_2000425"},{"id":"T15","span":{"begin":624,"end":642},"obj":"http://purl.obolibrary.org/obo/GO_0009790"},{"id":"T16","span":{"begin":982,"end":991},"obj":"http://purl.obolibrary.org/obo/GO_0006915"},{"id":"T17","span":{"begin":982,"end":991},"obj":"http://purl.obolibrary.org/obo/GO_0097194"},{"id":"T18","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0001783"},{"id":"T19","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0006927"},{"id":"T20","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0010657"},{"id":"T21","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0033024"},{"id":"T22","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0045476"},{"id":"T23","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0097152"},{"id":"T24","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_1904516"},{"id":"T25","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_1904606"},{"id":"T26","span":{"begin":1420,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0033028"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GO-CC","denotations":[{"id":"T1","span":{"begin":54,"end":59},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T2","span":{"begin":328,"end":333},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T3","span":{"begin":582,"end":587},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T4","span":{"begin":793,"end":798},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T5","span":{"begin":814,"end":819},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T6","span":{"begin":835,"end":840},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T7","span":{"begin":915,"end":920},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T8","span":{"begin":968,"end":973},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T9","span":{"begin":1015,"end":1020},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T10","span":{"begin":1115,"end":1120},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T11","span":{"begin":1199,"end":1204},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T12","span":{"begin":1235,"end":1240},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T13","span":{"begin":1279,"end":1284},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T14","span":{"begin":1430,"end":1435},"obj":"http://purl.obolibrary.org/obo/GO_0005623"},{"id":"T15","span":{"begin":318,"end":327},"obj":"http://purl.obolibrary.org/obo/GO_0030425"},{"id":"T16","span":{"begin":1430,"end":1449},"obj":"http://purl.obolibrary.org/obo/GO_0042995"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"UBERON-AE","denotations":[{"id":"T1","span":{"begin":74,"end":86},"obj":"http://purl.obolibrary.org/obo/UBERON_0001049"},{"id":"T2","span":{"begin":942,"end":953},"obj":"http://purl.obolibrary.org/obo/UBERON_0001049"},{"id":"T3","span":{"begin":81,"end":86},"obj":"http://purl.obolibrary.org/obo/UBERON_0000025"},{"id":"T4","span":{"begin":949,"end":953},"obj":"http://purl.obolibrary.org/obo/UBERON_0000025"},{"id":"T5","span":{"begin":148,"end":155},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T6","span":{"begin":468,"end":475},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T7","span":{"begin":635,"end":642},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T8","span":{"begin":892,"end":899},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T9","span":{"begin":1147,"end":1154},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T10","span":{"begin":1307,"end":1314},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T11","span":{"begin":1331,"end":1338},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"T12","span":{"begin":1527,"end":1534},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

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of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

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of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

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{"project":"glycosmos-test-structure-v1","denotations":[{"id":"PD-GlycanStructures-B_T1","span":{"begin":101,"end":110},"obj":"http://rdf.glyconavi.org/CarTNa/CarTNa207/trivialname"},{"id":"PD-GlycanStructures-B_T2","span":{"begin":204,"end":213},"obj":"http://rdf.glyconavi.org/CarTNa/CarTNa207/trivialname"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"glycosmos-test-glycan-structure","denotations":[{"id":"PD-GlycanStructures-B_T1","span":{"begin":101,"end":110},"obj":"http://rdf.glyconavi.org/CarTNa/CarTNa207/trivialname"},{"id":"PD-GlycanStructures-B_T2","span":{"begin":204,"end":213},"obj":"http://rdf.glyconavi.org/CarTNa/CarTNa207/trivialname"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"GlyCosmos600-MAT","denotations":[{"id":"PD-MAT-B_T1","span":{"begin":74,"end":86},"obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"PD-MAT-B_T2","span":{"begin":942,"end":953},"obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"PD-MAT-B_T3","span":{"begin":148,"end":155},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T4","span":{"begin":468,"end":475},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T5","span":{"begin":635,"end":642},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T6","span":{"begin":892,"end":899},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T7","span":{"begin":1147,"end":1154},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T8","span":{"begin":1307,"end":1314},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T9","span":{"begin":1331,"end":1338},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T10","span":{"begin":1527,"end":1534},"obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"PD-MAT-B_T11","span":{"begin":453,"end":458},"obj":"http://purl.obolibrary.org/obo/MAT_0000029"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"performance-test","denotations":[{"id":"PD-UBERON-AE-B_T1","span":{"begin":74,"end":86},"obj":"http://purl.obolibrary.org/obo/UBERON_0001049"},{"id":"PD-UBERON-AE-B_T2","span":{"begin":942,"end":953},"obj":"http://purl.obolibrary.org/obo/UBERON_0001049"},{"id":"PD-UBERON-AE-B_T3","span":{"begin":81,"end":86},"obj":"http://purl.obolibrary.org/obo/UBERON_0000025"},{"id":"PD-UBERON-AE-B_T4","span":{"begin":949,"end":953},"obj":"http://purl.obolibrary.org/obo/UBERON_0000025"},{"id":"PD-UBERON-AE-B_T5","span":{"begin":148,"end":155},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T6","span":{"begin":468,"end":475},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T7","span":{"begin":635,"end":642},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T8","span":{"begin":892,"end":899},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T9","span":{"begin":1147,"end":1154},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T10","span":{"begin":1307,"end":1314},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T11","span":{"begin":1331,"end":1338},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"},{"id":"PD-UBERON-AE-B_T12","span":{"begin":1527,"end":1534},"obj":"http://purl.obolibrary.org/obo/UBERON_0000922"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"Anatomy-MAT","denotations":[{"id":"T1","span":{"begin":74,"end":86},"obj":"Body_part"},{"id":"T2","span":{"begin":148,"end":155},"obj":"Body_part"},{"id":"T3","span":{"begin":453,"end":458},"obj":"Body_part"},{"id":"T4","span":{"begin":468,"end":475},"obj":"Body_part"},{"id":"T5","span":{"begin":635,"end":642},"obj":"Body_part"},{"id":"T6","span":{"begin":892,"end":899},"obj":"Body_part"},{"id":"T7","span":{"begin":942,"end":953},"obj":"Body_part"},{"id":"T8","span":{"begin":1147,"end":1154},"obj":"Body_part"},{"id":"T9","span":{"begin":1307,"end":1314},"obj":"Body_part"},{"id":"T10","span":{"begin":1331,"end":1338},"obj":"Body_part"},{"id":"T11","span":{"begin":1527,"end":1534},"obj":"Body_part"}],"attributes":[{"id":"A1","pred":"mat_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"A2","pred":"mat_id","subj":"T2","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A3","pred":"mat_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/MAT_0000029"},{"id":"A4","pred":"mat_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A5","pred":"mat_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A6","pred":"mat_id","subj":"T6","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A7","pred":"mat_id","subj":"T7","obj":"http://purl.obolibrary.org/obo/MAT_0000069"},{"id":"A8","pred":"mat_id","subj":"T8","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A9","pred":"mat_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A10","pred":"mat_id","subj":"T10","obj":"http://purl.obolibrary.org/obo/MAT_0000226"},{"id":"A11","pred":"mat_id","subj":"T11","obj":"http://purl.obolibrary.org/obo/MAT_0000226"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"Lectin-Jamboree","denotations":[{"id":"T1","span":{"begin":123,"end":129},"obj":"lectin"},{"id":"T2","span":{"begin":181,"end":187},"obj":"lectin"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"Lectin-Jamboree-Sentence","blocks":[{"id":"T1","span":{"begin":0,"end":156},"obj":"Sentence"},{"id":"T2","span":{"begin":157,"end":357},"obj":"Sentence"},{"id":"T3","span":{"begin":358,"end":588},"obj":"Sentence"},{"id":"T4","span":{"begin":589,"end":761},"obj":"Sentence"},{"id":"T5","span":{"begin":762,"end":900},"obj":"Sentence"},{"id":"T6","span":{"begin":901,"end":1039},"obj":"Sentence"},{"id":"T7","span":{"begin":1040,"end":1121},"obj":"Sentence"},{"id":"T8","span":{"begin":1122,"end":1339},"obj":"Sentence"},{"id":"T9","span":{"begin":1340,"end":1436},"obj":"Sentence"},{"id":"T10","span":{"begin":1437,"end":1535},"obj":"Sentence"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"NCBITAXON","denotations":[{"id":"T1","span":{"begin":142,"end":147},"obj":"OrganismTaxon"},{"id":"T3","span":{"begin":352,"end":356},"obj":"OrganismTaxon"},{"id":"T4","span":{"begin":382,"end":386},"obj":"OrganismTaxon"}],"attributes":[{"id":"A1","pred":"db_id","subj":"T1","obj":"10088"},{"id":"A2","pred":"db_id","subj":"T1","obj":"10090"},{"id":"A3","pred":"db_id","subj":"T3","obj":"10088"},{"id":"A4","pred":"db_id","subj":"T4","obj":"10088"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"Anatomy-UBERON","denotations":[{"id":"T1","span":{"begin":74,"end":86},"obj":"Body_part"},{"id":"T2","span":{"begin":90,"end":100},"obj":"Body_part"},{"id":"T3","span":{"begin":302,"end":313},"obj":"Body_part"},{"id":"T4","span":{"begin":318,"end":333},"obj":"Body_part"},{"id":"T5","span":{"begin":942,"end":953},"obj":"Body_part"}],"attributes":[{"id":"A1","pred":"uberon_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/UBERON_0001049"},{"id":"A2","pred":"uberon_id","subj":"T2","obj":"http://purl.obolibrary.org/obo/CL_0000235"},{"id":"A3","pred":"uberon_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/CL_0000235"},{"id":"A4","pred":"uberon_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/CL_0000451"},{"id":"A5","pred":"uberon_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/UBERON_0001049"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}

{"project":"CL-cell","denotations":[{"id":"T1","span":{"begin":90,"end":100},"obj":"Cell"},{"id":"T3","span":{"begin":302,"end":313},"obj":"Cell"},{"id":"T5","span":{"begin":318,"end":333},"obj":"Cell"}],"attributes":[{"id":"A1","pred":"cl_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/CL:0000235"},{"id":"A2","pred":"cl_id","subj":"T1","obj":"http://purl.obolibrary.org/obo/CL:0000394"},{"id":"A3","pred":"cl_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/CL:0000235"},{"id":"A4","pred":"cl_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/CL:0000394"},{"id":"A5","pred":"cl_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/CL:0000451"},{"id":"A6","pred":"cl_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/CL:0001056"}],"text":"Retardation of removal of radiation-induced apoptotic cells in developing neural tubes in macrophage galactose-type C-type lectin-1-deficient mouse embryos.\nMGL1/CD301a is a C-type lectin that recognizes galactose and N-acetylgalactosamine as monosaccharides and is expressed on limited populations of macrophages and dendritic cells at least in adult mice. In this study, pregnant mice with Mgl1+/- genotype were mated with Mgl1+/- or Mgl1-/- genotype males, and the embryos were used to assess a hypothesis that this molecule plays an important role in the clearance of apoptotic cells. After X-ray irradiation at 1 Gy of developing embryos at 10.5 days post coitus (d.p.c.), the number of Mgl1-/- pups was significantly reduced as compared with Mgl1+/+ pups. Distributions of MGL1-positive cells, MGL2-positive cells, and apoptotic cells were histologically examined in irradiated Mgl1+/+ embryos. MGL1-positive cells were detected in the neural tube in which many cells undergo apoptosis, whereas MGL2-positive cells were not observed. Biotinylated recombinant MGL1 bound a significant portion of the apoptotic cells. When Mgl1+/+ and Mgl1-/- embryos were examined for the presence of apoptotic cells, similar numbers of apoptotic cells gave rise, but the clearance of these cells was slower in Mgl1-/- embryos than in Mgl1+/+ embryos. These results strongly suggest that MGL1/CD301a is involved in the clearance of apoptotic cells. This process should be essential in the repair and normal development of X-ray-irradiated embryos."}