PubMed:16037488 / 448-677 JSONTXT

{"project":"GlycoBiology-GDGDB","denotations":[{"id":"_T33","span":{"begin":222,"end":227},"obj":"http://acgg.asia/db/diseases/gdgdb?con_ui=CON00351"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"uniprot-human","denotations":[{"id":"T2","span":{"begin":106,"end":117},"obj":"http://www.uniprot.org/uniprot/P02787"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"uniprot-mouse","denotations":[{"id":"T2","span":{"begin":106,"end":117},"obj":"http://www.uniprot.org/uniprot/Q63915"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"GO-BP","denotations":[{"id":"T2","span":{"begin":144,"end":156},"obj":"http://purl.obolibrary.org/obo/GO_0036065"},{"id":"T4","span":{"begin":190,"end":203},"obj":"http://purl.obolibrary.org/obo/GO_0070085"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"UBERON-AE","denotations":[{"id":"T2","span":{"begin":100,"end":105},"obj":"http://purl.obolibrary.org/obo/UBERON_0001977"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"sentences","denotations":[{"id":"TextSentencer_T4","span":{"begin":0,"end":229},"obj":"Sentence"},{"id":"T4","span":{"begin":0,"end":229},"obj":"Sentence"},{"id":"T4","span":{"begin":0,"end":229},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"performance-test","denotations":[{"id":"PD-UBERON-AE-B_T2","span":{"begin":100,"end":105},"obj":"http://purl.obolibrary.org/obo/UBERON_0001977"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"HP-phenotype","denotations":[{"id":"T3","span":{"begin":31,"end":43},"obj":"Phenotype"}],"attributes":[{"id":"A3","pred":"hp_id","subj":"T3","obj":"HP:0012024"}],"namespaces":[{"prefix":"HP","uri":"http://purl.obolibrary.org/obo/HP_"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}

{"project":"mondo_disease","denotations":[{"id":"T9","span":{"begin":31,"end":43},"obj":"Disease"},{"id":"T11","span":{"begin":222,"end":225},"obj":"Disease"}],"attributes":[{"id":"A9","pred":"mondo_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/MONDO_0009258"},{"id":"A10","pred":"mondo_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/MONDO_0018116"},{"id":"A11","pred":"mondo_id","subj":"T11","obj":"http://purl.obolibrary.org/obo/MONDO_0015286"}],"text":"Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I)."}