| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-67 |
Sentence |
denotes |
Are variants in the CAPN10 gene related to risk of type 2 diabetes? |
| TextSentencer_T2 |
68-145 |
Sentence |
denotes |
A quantitative assessment of population and family-based association studies. |
| TextSentencer_T3 |
146-313 |
Sentence |
denotes |
The calpain-10 gene (CAPN10) on chromosome 2q37.3 was the first candidate gene for type 2 diabetes (T2D) identified through a genomewide screen and positional cloning. |
| TextSentencer_T4 |
314-505 |
Sentence |
denotes |
One polymorphism (UCSNP-43: G-->A) and a specific haplotype combination defined by three polymorphisms (UCSNP-43, -19, and -63) were linked to an increased risk of T2D in several populations. |
| TextSentencer_T5 |
506-688 |
Sentence |
denotes |
To quantitatively assess the collective evidence for the effects of CAPN10 on risk of T2D, we conducted a meta-analysis of both population-based and family-based association studies. |
| TextSentencer_T6 |
689-863 |
Sentence |
denotes |
We retrieved data from the MEDLINE, PubMed, and Online Mendelian Inheritance in Man databases, as well as from other relevant reports and abstracts published up to July 2003. |
| TextSentencer_T7 |
864-938 |
Sentence |
denotes |
From a total of 26 studies with primary data (21 population-based studies: |
| TextSentencer_T8 |
939-994 |
Sentence |
denotes |
5,013 cases and 5,876 controls; 5 family-based studies: |
| TextSentencer_T9 |
995-1228 |
Sentence |
denotes |
487 parent-offspring trios), we developed a summary database that contains variables of study design, study population/ethnicity, specific polymorphisms and haplotype combinations in CAPN10, and diabetes-related metabolic phenotypes. |
| TextSentencer_T10 |
1229-1447 |
Sentence |
denotes |
For population-based studies, we used both fixed-effects and random-effects models to calculate the pooled odds ratio (OR) and 95% confidence interval (CI) for the associations of CAPN10 genotypes with the risk of T2D. |
| TextSentencer_T11 |
1448-1570 |
Sentence |
denotes |
We also calculated weighted mean differences for the associations between CAPN10 and diabetes-related quantitative traits. |
| TextSentencer_T12 |
1571-1731 |
Sentence |
denotes |
Under either an additive or a dominant effect model, we found no statistically significant relation between CAPN10 genotypes in the UCSNP-43 locus and T2D risk. |
| TextSentencer_T13 |
1732-1926 |
Sentence |
denotes |
However, under a recessive model, individuals homozygous for the common G allele had a statistically significant 19% higher risk of T2D than carriers of the A allele (OR 1.19; 95% CI 1.07-1.33). |
| TextSentencer_T14 |
1927-2111 |
Sentence |
denotes |
The association between the 112/121 haplotype combination and T2D risk appeared to be overestimated by several initial small studies with positive findings (OR 1.38; 95% CI 1.04-1.84). |
| TextSentencer_T15 |
2112-2219 |
Sentence |
denotes |
After we removed these initial studies, this association became nonsignificant (OR 1.11; 95% CI 0.91-1.35). |
| TextSentencer_T16 |
2220-2369 |
Sentence |
denotes |
Moreover, we found no evidence for the associations between the UCSNP-43 G/G genotype and the 112/121 haplotype combination and metabolic phenotypes. |
| TextSentencer_T17 |
2370-2540 |
Sentence |
denotes |
Our meta-analysis of family-based studies showed only an overtransmission of the rare allele C in UCSNP-44 from heterozygous parents to their affected offspring with T2D. |
| TextSentencer_T18 |
2541-2881 |
Sentence |
denotes |
Our analysis indicates that inadequate statistical power, racial/ethnic differences in frequencies of alleles, haplotypes and haplotype combinations, potential gene-gene or gene-environment interactions, publication bias, and multiple hypothesis testing may contribute to the significant heterogeneity in previous studies of CAPN10 and T2D. |
| TextSentencer_T19 |
2882-3116 |
Sentence |
denotes |
Our findings also suggest that both large-scale, well-designed association studies and functional studies are warranted to either reliably confirm or conclusively refute the initial hypothesis regarding the role of CAPN10 in T2D risk. |
| T1 |
0-67 |
Sentence |
denotes |
Are variants in the CAPN10 gene related to risk of type 2 diabetes? |
| T2 |
68-145 |
Sentence |
denotes |
A quantitative assessment of population and family-based association studies. |
| T3 |
146-313 |
Sentence |
denotes |
The calpain-10 gene (CAPN10) on chromosome 2q37.3 was the first candidate gene for type 2 diabetes (T2D) identified through a genomewide screen and positional cloning. |
| T4 |
314-505 |
Sentence |
denotes |
One polymorphism (UCSNP-43: G-->A) and a specific haplotype combination defined by three polymorphisms (UCSNP-43, -19, and -63) were linked to an increased risk of T2D in several populations. |
| T5 |
506-688 |
Sentence |
denotes |
To quantitatively assess the collective evidence for the effects of CAPN10 on risk of T2D, we conducted a meta-analysis of both population-based and family-based association studies. |
| T6 |
689-863 |
Sentence |
denotes |
We retrieved data from the MEDLINE, PubMed, and Online Mendelian Inheritance in Man databases, as well as from other relevant reports and abstracts published up to July 2003. |
| T7 |
864-938 |
Sentence |
denotes |
From a total of 26 studies with primary data (21 population-based studies: |
| T8 |
939-994 |
Sentence |
denotes |
5,013 cases and 5,876 controls; 5 family-based studies: |
| T9 |
995-1228 |
Sentence |
denotes |
487 parent-offspring trios), we developed a summary database that contains variables of study design, study population/ethnicity, specific polymorphisms and haplotype combinations in CAPN10, and diabetes-related metabolic phenotypes. |
| T10 |
1229-1447 |
Sentence |
denotes |
For population-based studies, we used both fixed-effects and random-effects models to calculate the pooled odds ratio (OR) and 95% confidence interval (CI) for the associations of CAPN10 genotypes with the risk of T2D. |
| T11 |
1448-1570 |
Sentence |
denotes |
We also calculated weighted mean differences for the associations between CAPN10 and diabetes-related quantitative traits. |
| T12 |
1571-1731 |
Sentence |
denotes |
Under either an additive or a dominant effect model, we found no statistically significant relation between CAPN10 genotypes in the UCSNP-43 locus and T2D risk. |
| T13 |
1732-1926 |
Sentence |
denotes |
However, under a recessive model, individuals homozygous for the common G allele had a statistically significant 19% higher risk of T2D than carriers of the A allele (OR 1.19; 95% CI 1.07-1.33). |
| T14 |
1927-2111 |
Sentence |
denotes |
The association between the 112/121 haplotype combination and T2D risk appeared to be overestimated by several initial small studies with positive findings (OR 1.38; 95% CI 1.04-1.84). |
| T15 |
2112-2219 |
Sentence |
denotes |
After we removed these initial studies, this association became nonsignificant (OR 1.11; 95% CI 0.91-1.35). |
| T16 |
2220-2369 |
Sentence |
denotes |
Moreover, we found no evidence for the associations between the UCSNP-43 G/G genotype and the 112/121 haplotype combination and metabolic phenotypes. |
| T17 |
2370-2540 |
Sentence |
denotes |
Our meta-analysis of family-based studies showed only an overtransmission of the rare allele C in UCSNP-44 from heterozygous parents to their affected offspring with T2D. |
| T18 |
2541-2881 |
Sentence |
denotes |
Our analysis indicates that inadequate statistical power, racial/ethnic differences in frequencies of alleles, haplotypes and haplotype combinations, potential gene-gene or gene-environment interactions, publication bias, and multiple hypothesis testing may contribute to the significant heterogeneity in previous studies of CAPN10 and T2D. |
| T19 |
2882-3116 |
Sentence |
denotes |
Our findings also suggest that both large-scale, well-designed association studies and functional studies are warranted to either reliably confirm or conclusively refute the initial hypothesis regarding the role of CAPN10 in T2D risk. |