| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-72 |
Sentence |
denotes |
Experimental allergic encephalomyelitis: the effect of 6-mercaptopurine. |
| T2 |
73-75 |
Sentence |
denotes |
1. |
| T3 |
76-228 |
Sentence |
denotes |
6-Mercaptopurine (6-MP) prevents experimental allergic encephalomyelitis (EAE) during the period of drug administration in both rabbits and guinea pigs. |
| T4 |
229-380 |
Sentence |
denotes |
The disease is suppressed even when treatment is started as late as the 5th day after antigenic stimulation in guinea pigs and the 12th day in rabbits. |
| T5 |
381-383 |
Sentence |
denotes |
2. |
| T6 |
384-478 |
Sentence |
denotes |
After discontinuation of 6-MP treatment, there is a latent period before the disease is noted. |
| T7 |
479-562 |
Sentence |
denotes |
The length of this latent period is not modified by the duration of 6-MP treatment. |
| T8 |
563-565 |
Sentence |
denotes |
3. |
| T9 |
566-742 |
Sentence |
denotes |
The effect of 6-MP on EAE is not the result of leukopenia, non-specific toxicity and debilitation, anti-inflammatory activity, or mere masking of clinical signs of the disease. |
| T10 |
743-814 |
Sentence |
denotes |
It is, rather, the result of 6-MP's specific anti-immunologic activity. |
| T11 |
815-817 |
Sentence |
denotes |
4. |
| T12 |
818-909 |
Sentence |
denotes |
The effects of 6-MP on antibody production, delayed hypersensitivity, and EAE are compared. |
| T13 |
910-1012 |
Sentence |
denotes |
This provides indirect evidence for the importance of circulating antibody in the pathogenesis of EAE. |
| T14 |
1013-1015 |
Sentence |
denotes |
5. |
| T15 |
1016-1157 |
Sentence |
denotes |
The important considerations in the use of 6-MP are discussed and the possible usefulness of 6-MP in human neurologic diseases is considered. |
| T1 |
0-72 |
Sentence |
denotes |
Experimental allergic encephalomyelitis: the effect of 6-mercaptopurine. |
| T2 |
73-75 |
Sentence |
denotes |
1. |
| T3 |
76-228 |
Sentence |
denotes |
6-Mercaptopurine (6-MP) prevents experimental allergic encephalomyelitis (EAE) during the period of drug administration in both rabbits and guinea pigs. |
| T4 |
229-380 |
Sentence |
denotes |
The disease is suppressed even when treatment is started as late as the 5th day after antigenic stimulation in guinea pigs and the 12th day in rabbits. |
| T5 |
381-383 |
Sentence |
denotes |
2. |
| T6 |
384-478 |
Sentence |
denotes |
After discontinuation of 6-MP treatment, there is a latent period before the disease is noted. |
| T7 |
479-562 |
Sentence |
denotes |
The length of this latent period is not modified by the duration of 6-MP treatment. |
| T8 |
563-565 |
Sentence |
denotes |
3. |
| T9 |
566-742 |
Sentence |
denotes |
The effect of 6-MP on EAE is not the result of leukopenia, non-specific toxicity and debilitation, anti-inflammatory activity, or mere masking of clinical signs of the disease. |
| T10 |
743-814 |
Sentence |
denotes |
It is, rather, the result of 6-MP's specific anti-immunologic activity. |
| T11 |
815-817 |
Sentence |
denotes |
4. |
| T12 |
818-909 |
Sentence |
denotes |
The effects of 6-MP on antibody production, delayed hypersensitivity, and EAE are compared. |
| T13 |
910-1012 |
Sentence |
denotes |
This provides indirect evidence for the importance of circulating antibody in the pathogenesis of EAE. |
| T14 |
1013-1015 |
Sentence |
denotes |
5. |
| T15 |
1016-1157 |
Sentence |
denotes |
The important considerations in the use of 6-MP are discussed and the possible usefulness of 6-MP in human neurologic diseases is considered. |