Id |
Subject |
Object |
Predicate |
Lexical cue |
T1 |
0-123 |
Sentence |
denotes |
Interleukin-6 antagonizes tumor necrosis factor-mediated mycobacteriostatic and mycobactericidal activities in macrophages. |
T2 |
124-267 |
Sentence |
denotes |
Interleukin-6 (IL-6) is a cytokine produced by a number of cells, including macrophages, and is directly involved in the inflammatory response. |
T3 |
268-367 |
Sentence |
denotes |
The production of IL-6 can be stimulated by monokines such as IL-1 and tumor necrosis factor (TNF). |
T4 |
368-466 |
Sentence |
denotes |
Mycobacterium avium complex organisms frequently cause disseminated disease in patients with AIDS. |
T5 |
467-543 |
Sentence |
denotes |
M. avium is an intracellular bacterium that that mainly infects macrophages. |
T6 |
544-836 |
Sentence |
denotes |
Treatment of M. avium-infected macrophage monolayers with recombinant IL-6 decreased the ability of TNF to activate cultured macrophages to inhibit growth of or kill intracellular M. avium (68% +/- 14% decrease in intracellular killing compared with that in monolayers not treated with IL-6). |
T7 |
837-1076 |
Sentence |
denotes |
To further evaluate whether this effect was dependent on the down regulation of membrane receptors to TNF, we examined 125I-TNF binding to macrophages previously exposed to IL-6: the expression of TNF receptors was decreased by 78% +/- 9%. |
T8 |
1077-1155 |
Sentence |
denotes |
The effect of IL-6 on TNF receptors was observed after 4 h and was reversible. |
T9 |
1156-1389 |
Sentence |
denotes |
Infection of macrophages with different M. avium serovars was associated with release of IL-6, and IL-6 production peaked at 48 h after infection in concentrations ranging from 328 +/- 87 ng/10(5) cells to 907 +/- 224 ng/10(5) cells. |
T10 |
1390-1508 |
Sentence |
denotes |
IL-6 did not have any influence on the rate of growth of the tested strains of M. avium within or outside macrophages. |
T11 |
1509-1657 |
Sentence |
denotes |
These results suggest that release of IL-6 by M. avium-infected macrophages may influence the host's immune response and the outcome of the disease. |
T1 |
0-123 |
Sentence |
denotes |
Interleukin-6 antagonizes tumor necrosis factor-mediated mycobacteriostatic and mycobactericidal activities in macrophages. |
T2 |
124-267 |
Sentence |
denotes |
Interleukin-6 (IL-6) is a cytokine produced by a number of cells, including macrophages, and is directly involved in the inflammatory response. |
T3 |
268-367 |
Sentence |
denotes |
The production of IL-6 can be stimulated by monokines such as IL-1 and tumor necrosis factor (TNF). |
T4 |
368-466 |
Sentence |
denotes |
Mycobacterium avium complex organisms frequently cause disseminated disease in patients with AIDS. |
T5 |
467-543 |
Sentence |
denotes |
M. avium is an intracellular bacterium that that mainly infects macrophages. |
T6 |
544-836 |
Sentence |
denotes |
Treatment of M. avium-infected macrophage monolayers with recombinant IL-6 decreased the ability of TNF to activate cultured macrophages to inhibit growth of or kill intracellular M. avium (68% +/- 14% decrease in intracellular killing compared with that in monolayers not treated with IL-6). |
T7 |
837-1076 |
Sentence |
denotes |
To further evaluate whether this effect was dependent on the down regulation of membrane receptors to TNF, we examined 125I-TNF binding to macrophages previously exposed to IL-6: the expression of TNF receptors was decreased by 78% +/- 9%. |
T8 |
1077-1155 |
Sentence |
denotes |
The effect of IL-6 on TNF receptors was observed after 4 h and was reversible. |
T9 |
1156-1389 |
Sentence |
denotes |
Infection of macrophages with different M. avium serovars was associated with release of IL-6, and IL-6 production peaked at 48 h after infection in concentrations ranging from 328 +/- 87 ng/10(5) cells to 907 +/- 224 ng/10(5) cells. |
T10 |
1390-1508 |
Sentence |
denotes |
IL-6 did not have any influence on the rate of growth of the tested strains of M. avium within or outside macrophages. |
T11 |
1509-1657 |
Sentence |
denotes |
These results suggest that release of IL-6 by M. avium-infected macrophages may influence the host's immune response and the outcome of the disease. |