> top > docs > PubMed:11235952 > annotations

PubMed:11235952 JSONTXT

Annnotations TAB JSON ListView MergeView

DisGeNET

Id Subject Object Predicate Lexical cue
T0 54-94 gene:7077 denotes tissue inhibitor of metalloproteinases 2
T1 134-150 disease:C0555198 denotes malignant glioma
T2 9-49 gene:4323 denotes membrane type 1 matrix metalloproteinase
T3 134-150 disease:C0555198 denotes malignant glioma
T4 1038-1045 gene:4323 denotes MT1-MMP
T5 1076-1088 disease:C0017636 denotes glioblastoma
T6 1491-1497 gene:7077 denotes TIMP-2
T7 1542-1554 disease:C0017636 denotes glioblastoma
R1 T0 T1 associated_with tissue inhibitor of metalloproteinases 2,malignant glioma
R2 T2 T3 associated_with membrane type 1 matrix metalloproteinase,malignant glioma
R3 T4 T5 associated_with MT1-MMP,glioblastoma
R4 T6 T7 associated_with TIMP-2,glioblastoma

DisGeNET5_gene_disease

Id Subject Object Predicate Lexical cue
11235952-0#9#49#gene4323 9-49 gene4323 denotes membrane type 1 matrix metalloproteinase
11235952-0#54#94#gene7077 54-94 gene7077 denotes tissue inhibitor of metalloproteinases 2
11235952-0#134#150#diseaseC0555198 134-150 diseaseC0555198 denotes malignant glioma
11235952-12#28#35#gene4323 2170-2177 gene4323 denotes MT1-MMP
11235952-12#145#151#gene7077 2287-2293 gene7077 denotes TIMP-2
11235952-12#94#106#diseaseC0017636 2236-2248 diseaseC0017636 denotes glioblastoma
11235952-4#111#117#gene7076 842-848 gene7076 denotes TIMP-1
11235952-4#163#176#diseaseC0017636 894-907 diseaseC0017636 denotes glioblastomas
9#49#gene4323134#150#diseaseC0555198 11235952-0#9#49#gene4323 11235952-0#134#150#diseaseC0555198 associated_with membrane type 1 matrix metalloproteinase,malignant glioma
54#94#gene7077134#150#diseaseC0555198 11235952-0#54#94#gene7077 11235952-0#134#150#diseaseC0555198 associated_with tissue inhibitor of metalloproteinases 2,malignant glioma
28#35#gene432394#106#diseaseC0017636 11235952-12#28#35#gene4323 11235952-12#94#106#diseaseC0017636 associated_with MT1-MMP,glioblastoma
145#151#gene707794#106#diseaseC0017636 11235952-12#145#151#gene7077 11235952-12#94#106#diseaseC0017636 associated_with TIMP-2,glioblastoma
111#117#gene7076163#176#diseaseC0017636 11235952-4#111#117#gene7076 11235952-4#163#176#diseaseC0017636 associated_with TIMP-1,glioblastomas

PubMed_Structured_Abstracts

Id Subject Object Predicate Lexical cue
T1 160-721 OBJECTIVE denotes Acquisition of invasive and metastatic potentials through proteinase expression is an essential event in tumor progression. Among proteinases, matrix metalloproteinases (MMPs) are thought to play a key role in tumor progression through the degradation of the extracellular matrix. In the present study, the authors examined the role of MMP-2 (gelatinase A) and membrane type 1 MMP (MT1-MMP), an activator of the zymogen of MMP-2, proMMP-2, together with tissue inhibitors of metalloproteinases (TIMP-1 and TIMP-2) in the invasion of astrocytic tumors in humans.
T2 731-2128 METHODS denotes Analyses performed using sandwich enzyme immunoassays demonstrated that the production levels of pro-MMP-2 and TIMP-1, but not TIMP-2, are significantly higher in glioblastomas multiforme than in other grades of astrocytic tumors. Quantitative reverse transcription-polymerase chain reaction indicated that MT1-MMP is expressed predominantly in glioblastoma tissues, and its expression levels are significantly enhanced as tumor grade increases. In addition, the expression levels and proMMP-2 activation ratio were remarkably higher in glioblastomas associated with cerebrospinal fluid (CSF) dissemination than in those not associated with CSF dissemination. In contrast, an examination of TIMP-2 levels showed a reverse correlation. Like MT1-MMP, TIMP-1 and TIMP-2 were immunolocalized to neoplastic cells in glioblastoma samples. To study the roles of these molecules in the invasion of astrocytic tumors more fully, stable transfectants expressing the MT1-MMP gene were developed in a U251 human glioblastoma cell line. The MT1-MMP transfectants displayed prominent activation of proMMP-2 and invasive growth in three-dimensional collagen gel; however, mock transfectants and parental cells displayed noninvasive growth without the activation. The invasion and gelatinolytic activity of the transfectants were completely inhibited by addition of recombinant TIMP-2, but not recombinant TIMP-1.
T3 2142-2294 CONCLUSIONS denotes These results indicate that MT1-MMP may contribute to tumor invasion and CSF dissemination of glioblastoma cells on the basis of an imbalance of TIMP-2.