Id |
Subject |
Object |
Predicate |
Lexical cue |
TextSentencer_T1 |
0-116 |
Sentence |
denotes |
Interactions of perturbations in intrauterine growth and growth during childhood on the risk of adult-onset disease. |
TextSentencer_T2 |
117-293 |
Sentence |
denotes |
The 'fetal origins' hypothesis (Barker, 1995) would predict that the rising epidemic of diabetes and CHD in India would be due to poor intrauterine growth of the Indian babies. |
TextSentencer_T3 |
294-507 |
Sentence |
denotes |
While this explanation may be valid to an extent, the higher prevalence of these disorders in urban compared with rural India (where birth weights are lower) would suggest a significant role for postnatal factors. |
TextSentencer_T4 |
508-671 |
Sentence |
denotes |
In a cohort of 477 children born in the King Edward Memorial Hospital, Pune, we found that at 8 years of age current obesity strongly predicted insulin resistance. |
TextSentencer_T5 |
672-824 |
Sentence |
denotes |
When this effect was allowed for, low birth weight was significantly associated with insulin-resistance variables and other cardiovascular risk factors. |
TextSentencer_T6 |
825-974 |
Sentence |
denotes |
Children who were born small but had grown heavy (or tall) were the most insulin resistant and had the highest levels of cardiovascular risk factors. |
TextSentencer_T7 |
975-1054 |
Sentence |
denotes |
Accelerated growth in relation to mid parental height was similarly predictive. |
TextSentencer_T8 |
1055-1138 |
Sentence |
denotes |
Poor intrauterine growth also predicted higher central adiposity at 8 years of age. |
TextSentencer_T9 |
1139-1222 |
Sentence |
denotes |
We have also studied maternal nutrition and fetal growth in six villages near Pune. |
TextSentencer_T10 |
1223-1476 |
Sentence |
denotes |
A newborn Indian baby is small (2650 g, SD score (SDS) -1.6 compared with an average white Caucasian baby born in the UK) and 'thin' (ponderal index 2.45 kg/m3, SDS -1.2), but has preserved its subcutaneous fat (subscapular skinfold thickness SDS -0.6). |
TextSentencer_T11 |
1477-1561 |
Sentence |
denotes |
The thinness of the Indian babies is due to poor muscle and small abdominal viscera. |
TextSentencer_T12 |
1562-1663 |
Sentence |
denotes |
We have proposed this composition as the 'thrifty phenotype' (Hales & Barker, 1992) of Indian babies. |
TextSentencer_T13 |
1664-1781 |
Sentence |
denotes |
Maternal size and intake of certain food groups during pregnancy were important determinants of the baby's phenotype. |
TextSentencer_T14 |
1782-1871 |
Sentence |
denotes |
Thus, the small Indian babies are programmed to deposit fat from their intrauterine life. |
TextSentencer_T15 |
1872-1963 |
Sentence |
denotes |
Exaggeration of this tendency in later life is associated with insulin-resistance syndrome. |
TextSentencer_T16 |
1964-2102 |
Sentence |
denotes |
Control of the insulin-resistance epidemic in India might depend on improved intrauterine development and prevention of childhood obesity. |
T1 |
0-116 |
Sentence |
denotes |
Interactions of perturbations in intrauterine growth and growth during childhood on the risk of adult-onset disease. |
T2 |
117-293 |
Sentence |
denotes |
The 'fetal origins' hypothesis (Barker, 1995) would predict that the rising epidemic of diabetes and CHD in India would be due to poor intrauterine growth of the Indian babies. |
T3 |
294-507 |
Sentence |
denotes |
While this explanation may be valid to an extent, the higher prevalence of these disorders in urban compared with rural India (where birth weights are lower) would suggest a significant role for postnatal factors. |
T4 |
508-671 |
Sentence |
denotes |
In a cohort of 477 children born in the King Edward Memorial Hospital, Pune, we found that at 8 years of age current obesity strongly predicted insulin resistance. |
T5 |
672-824 |
Sentence |
denotes |
When this effect was allowed for, low birth weight was significantly associated with insulin-resistance variables and other cardiovascular risk factors. |
T6 |
825-974 |
Sentence |
denotes |
Children who were born small but had grown heavy (or tall) were the most insulin resistant and had the highest levels of cardiovascular risk factors. |
T7 |
975-1054 |
Sentence |
denotes |
Accelerated growth in relation to mid parental height was similarly predictive. |
T8 |
1055-1138 |
Sentence |
denotes |
Poor intrauterine growth also predicted higher central adiposity at 8 years of age. |
T9 |
1139-1222 |
Sentence |
denotes |
We have also studied maternal nutrition and fetal growth in six villages near Pune. |
T10 |
1223-1476 |
Sentence |
denotes |
A newborn Indian baby is small (2650 g, SD score (SDS) -1.6 compared with an average white Caucasian baby born in the UK) and 'thin' (ponderal index 2.45 kg/m3, SDS -1.2), but has preserved its subcutaneous fat (subscapular skinfold thickness SDS -0.6). |
T11 |
1477-1561 |
Sentence |
denotes |
The thinness of the Indian babies is due to poor muscle and small abdominal viscera. |
T12 |
1562-1663 |
Sentence |
denotes |
We have proposed this composition as the 'thrifty phenotype' (Hales & Barker, 1992) of Indian babies. |
T13 |
1664-1781 |
Sentence |
denotes |
Maternal size and intake of certain food groups during pregnancy were important determinants of the baby's phenotype. |
T14 |
1782-1871 |
Sentence |
denotes |
Thus, the small Indian babies are programmed to deposit fat from their intrauterine life. |
T15 |
1872-1963 |
Sentence |
denotes |
Exaggeration of this tendency in later life is associated with insulin-resistance syndrome. |
T16 |
1964-2102 |
Sentence |
denotes |
Control of the insulin-resistance epidemic in India might depend on improved intrauterine development and prevention of childhood obesity. |