PubMed:10766914
Annnotations
PMID_GLOBAL
Id | Subject | Object | Predicate | Lexical cue | mondo_id |
---|---|---|---|---|---|
T1 | 42-57 | DiseaseOrPhenotypicFeature | denotes | cystic fibrosis | 0009061 |
T2 | 198-213 | DiseaseOrPhenotypicFeature | denotes | cystic fibrosis | 0009061 |
2015-BEL-Sample
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
T1 | 423-812 | a(CHEBI:genistein) decreases p(MGI:Cftr) | denotes | TR. 2. Addition of genistein (100 microM) to the intracellular solution caused a small decrease in single-channel current amplitude (i), but a large reduction in open probability (Po). 3. Single-channel analysis of channel block suggested that genistein (100 microM) may inhibit CFTR by two mechanisms: first, it may slow the rate of channel opening and second, it may block open channels. |
2015-BEL-Sample-2
Id | Subject | Object | Predicate | Lexical cue |
---|---|---|---|---|
BEL:20000150 | 423-812 | a(CHEBI:genistein) decreases p(MGI:Cftr) | denotes | TR. 2. Addition of genistein (100 microM) to the intracellular solution caused a small decrease in single-channel current amplitude (i), but a large reduction in open probability (Po). 3. Single-channel analysis of channel block suggested that genistein (100 microM) may inhibit CFTR by two mechanisms: first, it may slow the rate of channel opening and second, it may block open channels. |